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index medicus (155) 155
animals (131) 131
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immunology (70) 70
mice, knockout (60) 60
humans (59) 59
inflammation (54) 54
apoptosis (53) 53
cell biology (46) 46
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abridged index medicus (20) 20
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cytokines (18) 18
gene expression (18) 18
in-vivo (18) 18
phosphorylation (18) 18
tumor-necrosis-factor (18) 18
analysis (17) 17
cell-death (17) 17
disease models, animal (17) 17
gastroenterology & hepatology (17) 17
research article (17) 17
tumor necrosis factor (17) 17
genetic aspects (16) 16
i-kappa b kinase - genetics (16) 16
incontinentia pigmenti (16) 16
mouse model (16) 16
necrosis (16) 16
receptor-interacting protein serine-threonine kinases - metabolism (16) 16
health aspects (15) 15
pathogenesis (15) 15
transcription factors (15) 15
deficient mice (14) 14
hematology (14) 14
inflammation - pathology (14) 14
receptor-interacting protein serine-threonine kinases - genetics (14) 14
signaling (13) 13
tumor necrosis factor-alpha - genetics (13) 13
tumor necrosis factor-alpha - physiology (13) 13
deletion (12) 12
disease (12) 12
macrophages (12) 12
prevention (12) 12
skin (12) 12
epithelial cells - metabolism (11) 11
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i-kappa b kinase - deficiency (11) 11
inflammation - metabolism (11) 11
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Journal Article
Nature Reviews Immunology, ISSN 1474-1733, 11/2009, Volume 9, Issue 11, pp. 778 - 788
The nuclear factor-kappa B (NF-kappa B) signalling pathway regulates immune responses and is implicated in the pathogenesis of many inflammatory diseases.... 
TOLL-LIKE-RECEPTORS | CHRONIC INTESTINAL INFLAMMATION | ALPHA-INDUCED APOPTOSIS | BETA LINKS INFLAMMATION | SEVERE LIVER DEGENERATION | NECROSIS-FACTOR-ALPHA | TNF-INDUCED APOPTOSIS | IMMUNOLOGY | GAMMA-DEFICIENT MICE | INDUCED INSULIN-RESISTANCE | BINDING DOMAIN PEPTIDE
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e54128 - e54128
Rationale: COPD is an inflammatory lung disease largely associated with exposure to cigarette smoke (CS). The mechanism by which CS leads to the pathogenesis... 
CHRONIC-BRONCHITIS | OBSTRUCTIVE PULMONARY-DISEASE | OXIDATIVE STRESS | EPITHELIAL-CELLS | INDUCED LUNG INFLAMMATION | MULTIDISCIPLINARY SCIENCES | IKK-BETA | BETA-DEPENDENT GENES | INDUCED EMPHYSEMA | FLOW LIMITATION | MOUSE LUNG | Immunohistochemistry | Smoking - adverse effects | Epithelial Cells - metabolism | Respiratory Tract Diseases - metabolism | Humans | Male | NF-kappa B - metabolism | Pulmonary Disease, Chronic Obstructive - etiology | Respiratory Tract Diseases - genetics | Inflammation - metabolism | I-kappa B Kinase - metabolism | Time Factors | I-kappa B Kinase - antagonists & inhibitors | Pulmonary Disease, Chronic Obstructive - metabolism | Disease Models, Animal | Pulmonary Disease, Chronic Obstructive - genetics | Amides - pharmacology | Gene Expression | Lipopolysaccharides - toxicity | Signal Transduction | Smoke - adverse effects | Mice, Inbred C57BL | Cells, Cultured | Thiophenes - pharmacology | I-kappa B Kinase - genetics | Reverse Transcriptase Polymerase Chain Reaction | Inflammation - etiology | Mice, Knockout | Respiratory Tract Diseases - etiology | Animals | Transcription Factor RelA - metabolism | Inflammation - genetics | Tobacco - chemistry | Mice | Heart | Oxidative stress | Animal models | Laboratories | Pathogenesis | Lung | Inflammatory response | Activation | Smoke | Kinases | Assaying | Modulators | Respiratory tract | Signal transduction | Rodents | Respiratory tract diseases | Chronic obstructive pulmonary disease | Genetic modification | NF-κB protein | Myeloid cells | Tobacco smoke | Cytokines | Lung diseases | Pharmacology | Inflammation | Exposure | Gene expression | Cigarettes | Medicine | Signaling | Lungs | Conditioned stimulus | Cigarette smoke | Apoptosis | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2014, Volume 111, Issue 47, pp. 16836 - 16841
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7516, pp. 90 - 94
Necroptosis has emerged as an important pathway of programmed cell death in embryonic development, tissue homeostasis, immunity and inflammation(1-8). RIPK1 is... 
CHRONIC INTESTINAL INFLAMMATION | CELLS | KAPPA-B ACTIVATION | NECROSIS-FACTOR RECEPTOR | MULTIDISCIPLINARY SCIENCES | IN-VIVO | KINASE | ALPHA-DEPENDENT APOPTOSIS | MICE | SKIN INFLAMMATION | DELETION | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Inflammation - pathology | Skin - cytology | Epithelial Cells - metabolism | Skin - metabolism | Caspase 8 - metabolism | Homeostasis | Paneth Cells - pathology | Male | Intestines - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Necrosis | Receptors, Tumor Necrosis Factor, Type I - deficiency | Inflammation - metabolism | Female | Epithelial Cells - cytology | Skin - pathology | Paneth Cells - metabolism | Intestines - pathology | Cell Survival | Fas-Associated Death Domain Protein - metabolism | Fas-Associated Death Domain Protein - deficiency | Epithelial Cells - pathology | Mice, Knockout | Keratinocytes - pathology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Keratinocytes - metabolism | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Myeloid Differentiation Factor 88 - metabolism | Apoptosis | Intestines - cytology | Physiological aspects | Physiological research | Inflammation | Research | Protein kinases | Pathology | Antibiotics | Rodents | Skin | Kinases | Index Medicus
Journal Article
Journal Article
Blood, ISSN 0006-4971, 06/2016, Volume 127, Issue 22, pp. 2732 - 2741
Journal Article
Cell, ISSN 0092-8674, 2008, Volume 133, Issue 2, pp. 235 - 249
Journal Article