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FASEB journal : official publication of the Federation of American Societies for Experimental Biology, ISSN 0892-6638, 11/2019, Volume 33, Issue 11, pp. 12888 - 12899
Endothelial up-regulation of VCAM-1 at susceptible sites in arteries modulates the recruitment efficiency of inflammatory monocytes that initiates... 
Journal Article
American Journal of Physiology, ISSN 0363-6135, 06/2012, Volume 302, Issue 12, p. H2498
Atherosclerosis occurs preferentially at sites of disturbed blood flow despite the influence of risk factors contributing to systemic inflammation. The... 
Hogs | Atherosclerosis | Physiology | Shear stresses | Diabetes | Gene expression
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e78322
Circulating triglyceride-rich lipoproteins (TGRL) from hypertriglyceridemic subjects exacerbate endothelial inflammation and promote monocyte infiltration into... 
OBESITY | HOMEOSTASIS | UNFOLDED PROTEIN RESPONSE | HOMOLOGOUS PROTEIN | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DISEASE | ATHEROSCLEROSIS | NECROSIS-FACTOR-ALPHA | INDUCED ER STRESS | FATTY-ACIDS | Tumor Necrosis Factor-alpha - metabolism | Endoplasmic Reticulum - genetics | Unfolded Protein Response - genetics | Endothelial Cells - metabolism | Gene Expression Regulation - genetics | Lipoproteins - genetics | Atherosclerosis - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | Cells, Cultured | Endoplasmic Reticulum - metabolism | Male | Monocytes - metabolism | Up-Regulation - genetics | Endoplasmic Reticulum Stress - genetics | Atherosclerosis - metabolism | Down-Regulation - genetics | Triglycerides - metabolism | Endothelium, Vascular - metabolism | Lipoproteins - metabolism | Female | Vascular Cell Adhesion Molecule-1 - genetics | Triglycerides - genetics | Vascular Cell Adhesion Molecule-1 - metabolism | Type 2 diabetes | Tumor necrosis factor | Triglycerides | Interferon | Inflammation | Blood lipids | Protein binding | Endothelium | Regulations | Vascular cell adhesion molecule 1 | Transcription factors | Interferon regulatory factor 1 | Metastases | Proteins | Cell adhesion | Aorta | Sensors | Stress response | Stresses | Obesity | Inositol | Tumor necrosis factor-α | Metabolism | Fatty acids | Internalization | Low density lipoprotein receptors | Infiltration | Cardiovascular diseases | Endoplasmic reticulum | Endocrinology | Phenylbutyric acid | Interferon regulatory factor | Correlation | Gene regulation | Homeostasis | Homology | Lipids | Stimulation | CCAAT/enhancer-binding protein | Kinases | Cell adhesion & migration | Cell adhesion molecules | Engineering | Sterols | Protein folding | Atherosclerosis | Tumor necrosis factor-TNF | Biomedical engineering | Nutrition | Health risks | siRNA | Adhesion | Cholesterol | Stress | Endothelial cells | Lipoproteins | Monocytes | Insulin resistance | Apoptosis
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 02/2019, Volume 316, Issue 2, pp. H408 - H420
Increased expression of vascular cell adhesion molecule 1 (VCAM-1) on the aortic endothelium is an early marker of atherogenesis, promoted in part by elevated... 
atherosclerosis | GATA6 | endothelium | vascular cell adhesion molecule 1 | MAMMALIAN TARGET | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MOLECULE-1 EXPRESSION | INDUCTION | RAPTOR | INACTIVATION | TUMOR-NECROSIS-FACTOR | ABLATION | PERIPHERAL VASCULAR DISEASE | RICTOR | Endothelium, Vascular - cytology | Protein Kinase C-alpha - metabolism | Signal Transduction | TOR Serine-Threonine Kinases - metabolism | Endothelial Cells - metabolism | Down-Regulation | Humans | Cells, Cultured | Aorta - metabolism | Cell Adhesion | Atherosclerosis - metabolism | GATA6 Transcription Factor - metabolism | Tumor Necrosis Factor-alpha - pharmacology | TOR Serine-Threonine Kinases - genetics | Endothelium, Vascular - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Aorta - cytology | GATA6 Transcription Factor - genetics | Monocytes - physiology | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - physiology | Endothelial Cells - drug effects | TOR protein | Post-transcription | Flow cytometry | Atherogenesis | Chromatin | Protein kinase C | Vascular cell adhesion molecule 1 | Immunoprecipitation | Kinases | Cell adhesion molecules | Cell adhesion & migration | Transcription activation | Atherosclerosis | Cell adhesion | Aorta | Inhibition | Cytokines | Rapamycin | Inflammation | siRNA | Tumor necrosis factor-α | Adhesion | Endothelial cells | Endothelium | Organic chemistry | Signaling | Monocytes | Depletion | Arteriosclerosis
Journal Article
Journal Article
JOURNAL OF LIPID RESEARCH, ISSN 0022-2275, 12/2019, Volume 60, Issue 12, pp. 1996 - 2005
Epoxyeicosatrienoic acids (EETs) are formed from the metabolism of arachidonic acid by cytochrome P450s. EETs promote angiogenesis linked to tumor growth in... 
arachidonic acid | endothelial cells | METABOLITES | COX-2 | angiogenesis | BIOCHEMISTRY & MOLECULAR BIOLOGY | soluble epoxide hydrolase | 5,6-EPOXYEICOSATRIENOIC ACID | CYCLOOXYGENASE | HYDROLASE | 8,9-EPOXYEICOSATRIENOIC ACID | INHIBITION | ENDOTHELIAL-CELLS | mass spectrometry | cyclooxygenase 2 | cancer | metabolism | GROWTH-FACTOR | TUMOR-GROWTH
Journal Article
Circulation Research, ISSN 0009-7330, 09/2012, Volume 111, Issue 8, pp. 1054 - 1064
Journal Article
American Journal of Physiology, ISSN 0363-6135, 10/2015, Volume 309, Issue 7, p. H1130
  Mechanisms of restenosis in type 2 diabetes mellitus (T2DM) are incompletely elucidated, but advanced glycation end-product (AGE)-induced vascular remodeling... 
Transplants & implants | Rodents | Collagen | Protein expression | Diabetes | Stents
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 06/2012, Volume 302, Issue 12, pp. 2498 - 2508
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 03/2011, Volume 300, Issue 3, pp. 784 - 791
A rise in postprandial serum triglycerides (PP-sTG) can potentiate inflammatory responses in vascular endothelial cells (ECs) and thus serves as an independent... 
Tumor necrosis factor-a | Triglyceride-rich lipoprotein | Endothelial cells | Atherosclerosis | Dyslipidemia | Vascular cell adhesion molecule-1 | atherosclerosis | HYPERTRIGLYCERIDEMIC SUBJECTS | endothelial cells | METABOLIC SYNDROME | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | triglyceride-rich lipoprotein | NECROSIS-FACTOR-ALPHA | POSTPRANDIAL STATE | vascular cell adhesion molecule-1 | APOLIPOPROTEIN-E | tumor necrosis factor-alpha | CELL-ADHESION MOLECULE-1 | RICH LIPOPROTEINS | NONFASTING TRIGLYCERIDES | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | dyslipidemia | Dietary Fats - metabolism | Endothelial Cells - metabolism | Humans | Obesity, Abdominal - metabolism | Cells, Cultured | E-Selectin - metabolism | Male | Monocytes - metabolism | Aorta - metabolism | Lipoproteins - blood | Triglycerides - metabolism | Young Adult | Inflammation - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Waist Circumference | Lipoproteins - metabolism | Postprandial Period | Triglycerides - blood | Tumor Necrosis Factor-alpha - administration & dosage | Female | Vascular Cell Adhesion Molecule-1 - metabolism | Obesity | Vascular endothelium | Inflammation | Triglycerides | Research | Cardiovascular diseases | Health aspects | Risk factors | Vascular Biology and Microcirculation | tumor necrosis factor-α
Journal Article
Circulation Research, ISSN 0009-7330, 02/2007, Volume 100, Issue 3, pp. 381 - 390
High levels of triglyceride-rich lipoproteins (TGRLs) in blood are linked to development of atherosclerosis, yet the mechanisms by which these particles... 
Vascular adhesion molecule-1 | Monocyte | NFκB | Triglyceride-rich lipoprotein | Atherosclerosis | Endothelium | atherosclerosis | ATHEROSCLEROTIC LESIONS | endothelium | CHYLOMICRON REMNANTS | CARDIAC & CARDIOVASCULAR SYSTEMS | INTERCELLULAR-ADHESION MOLECULE-1 | RECEPTOR-RELATED PROTEIN | vascular adhesion molecule-1 | triglyceride-rich lipoprotein | SIGNAL-TRANSDUCTION | FAMILY-MEMBER | METABOLISM | E-SELECTIN | monocyte | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | NF-KAPPA-B | EXPRESSION | NF kappa B | Endothelium, Vascular - cytology | LDL-Receptor Related Proteins - metabolism | Oxidative Stress | Humans | Apolipoprotein C-III - metabolism | NF-kappa B - metabolism | Endocytosis | E-Selectin - genetics | Dietary Fats - administration & dosage | E-Selectin - biosynthesis | Aorta | p38 Mitogen-Activated Protein Kinases - metabolism | Lipoproteins, VLDL - toxicity | Arteriosclerosis - etiology | Cells, Cultured - drug effects | Endothelial Cells - metabolism | Models, Cardiovascular | Receptors, LDL - metabolism | Cell Adhesion - drug effects | Cell Adhesion Molecules - metabolism | Triglycerides - toxicity | Monocytes - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Lipoproteins, LDL - toxicity | Lipoproteins, VLDL - blood | Signal Transduction - drug effects | Intercellular Adhesion Molecule-1 - genetics | Lipopolysaccharides - pharmacology | Lipoproteins, HDL - toxicity | Tumor Necrosis Factor-alpha - physiology | Leukocytes - drug effects | Vascular Cell Adhesion Molecule-1 - biosynthesis | Cells, Cultured - metabolism | Lipoproteins, HDL - blood | Monocytes - cytology | LDL-Receptor Related Proteins - drug effects | Membrane Transport Proteins - drug effects | Low Density Lipoprotein Receptor-Related Protein-1 - drug effects | LDL-Receptor Related Protein-Associated Protein - pharmacology | Intercellular Adhesion Molecule-1 - biosynthesis | Chylomicrons - blood | Membrane Transport Proteins - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Arteritis - etiology | Leukocytes - cytology | Dietary Fats - adverse effects | Rheology | Low Density Lipoprotein Receptor-Related Protein-1 - metabolism | Hypoglycemia | Hypertriglyceridemia - blood | Gene Expression Regulation - drug effects | Hypertriglyceridemia - complications | Triglycerides - blood | Fat Emulsions, Intravenous - pharmacology | Receptors, LDL - drug effects | Apolipoprotein C-III - pharmacology | Aortic Diseases - etiology | Lipoproteins, LDL - blood | Endothelial Cells - drug effects
Journal Article
Journal Article
PLoS ONE, 10/2013, Volume 8, Issue 10
Circulating triglyceride-rich lipoproteins (TGRL) from hypertriglyceridemic subjects exacerbate endothelial inflammation and promote monocyte infiltration into... 
Adhesion
Journal Article