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Developmental Cell, ISSN 1534-5807, 01/2018, Volume 44, Issue 1, pp. 5 - 7
In a recent issue of , describe a signaling pathway that prevents transgenerational inheritance of cytoplasmic protein aggregates. Fertilizing sperm trigger... 
DEVELOPMENTAL BIOLOGY | DIFFERENTIATION | PROTEINS | ELEGANS | CELL BIOLOGY | Index Medicus
Journal Article
Cell, ISSN 0092-8674, 08/2013, Volume 154, Issue 3, pp. X481 - 483
The selective breeding of rats as physiological, behavioral, and disease models generated a wealth of variation relevant to the genetics of complex traits. In... 
Journal Article
by Chiu, Hua-Sheng and Somvanshi, Sonal and Patel, Ektaben and Patel, Tushar and Chen, Amy and Chen, Chu and Chen, Ting-Wen and Singh, Rosy and Singh, Vivek P and Singh, Bhuvanesh and Zorman, Barry and Patil, Sagar L and Pan, Yinghong and Chatterjee, Sujash S and Caesar-Johnson, Samantha J and Demchok, John A and Felau, Ina and Kasapi, Melpomeni and Ferguson, Martin L and Hutter, Carolyn M and Sofia, Heidi J and Tarnuzzer, Roy and Wang, Jing and Wang, Timothy and Wang, Min and Wang, Jioajiao and Wang, Zhining and Wang, Linghua and Yang, Ju Dong and Yang, Liming and Yang, Ian and Yang, Hannah and Zenklusen, Jean C and Zhang, Jiashan (Julia) and Zhang, Hailei and Zhang, Wei and Zhang, Jiexin and Zhang, Hongxin and Zhang, Lizhi and Zhang, Hongzheng and Chudamani, Sudha and Liu, Jia and Liu, Yuexin and Liu, Xiuping and Liu, Wenbin and Lolla, Laxmi and Naresh, Rashi and Pihl, Todd and Sun, Qiang and Sun, Yichao and Wan, Yunhu and Wu, Ye and Cho, Juok and DeFreitas, Timothy and Frazer, Scott and Gehlenborg, Nils and Getz, Gad and Heiman, David I and Kim, Jaegil and Lawrence, Michael S and Lin, Pei and Meier, Sam and Noble, Michael S and Saksena, Gordon and Voet, Doug and Bernard, Brady and Chambwe, Nyasha and Dhankani, Varsha and Knijnenburg, Theo and Kramer, Roger and Leinonen, Kalle and Miller, Christopher A and Miller, Michael and Miller, Judy and Reynolds, Sheila and Shmulevich, Ilya and Thorsson, Vesteinn and Akbani, Rehan and Broom, Bradley M and Hegde, Apurva M and Ju, Zhenlin and Kanchi, Rupa S and Korkut, Anil and Li, Jun and Li, Wei and Liang, Han and Ling, Shiyun and Lu, Yiling and Mills, Gordon B and Ng, Kwok-Shing and Rao, Arvind and Ryan, Michael and Weinstein, John N and Abeshouse, Adam and Armenia, Joshua and Chakravarty, Debyani and Chatila, Walid K and de Bruijn, Ino and Gao, Galen F and Gao, Jianjiong and ... and Canc Genome Atlas Res Network and Cancer Genome Atlas Research Network
Cell Reports, ISSN 2211-1247, 04/2018, Volume 23, Issue 1, pp. 297 - 312.e12
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 1049 - 1061
The TOR (target of rapamycin) kinase limits longevity by poorly understood mechanisms. Rapamycin suppresses the mammalian TORC1 complex, which regulates... 
EPITHELIAL-CELL LINE | GROWTH ARREST | DNA-DAMAGE | IN-VIVO | INFLAMMATORY CYTOKINE SECRETION | HUMAN FIBROBLASTS | TUMOR-CELLS | CANCER-THERAPY | REPLICATIVE SENESCENCE | P53 | CELL BIOLOGY | Up-Regulation | Fibroblasts - enzymology | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Gene Expression Regulation, Neoplastic | Interleukin-1alpha - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Prostatic Neoplasms - secretion | Prostatic Neoplasms - immunology | Dose-Response Relationship, Drug | TOR Serine-Threonine Kinases - antagonists & inhibitors | Prostatic Neoplasms - genetics | TOR Serine-Threonine Kinases - genetics | Transfection | RNA Interference | Time Factors | Cellular Senescence | Inflammation Mediators - metabolism | Transcription, Genetic | Antineoplastic Agents - pharmacology | Interleukin-1alpha - genetics | Interleukin-6 - metabolism | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Anti-Inflammatory Agents - pharmacology | Interleukin-1alpha - secretion | Sirolimus - metabolism | Tumor Burden | Mice, SCID | Sirolimus - pharmacology | Xenograft Model Antitumor Assays | Phenotype | Animals | Fibroblasts - drug effects | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Mitoxantrone - pharmacology | Interleukins | Aging | Genetic aspects | Properties | Phosphotransferases | Carcinogenesis | Health aspects | Cells | Index Medicus
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2009, Volume 11, Issue 8, pp. 973 - 979
Cellular senescence suppresses cancer by stably arresting the proliferation of damaged cells. Paradoxically, senescent cells also secrete factors that alter... 
TUMOR SUPPRESSION | HUMAN-CELLS | HUMAN FIBROBLASTS | GROWTH-FACTOR | ONCOGENE-INDUCED SENESCENCE | CELLULAR SENESCENCE | CANCER | TUMORIGENESIS | P53 | TELOMERES | CELL BIOLOGY | RNA, Small Interfering - genetics | Humans | Male | Green Fluorescent Proteins - genetics | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Telomerase - genetics | Transfection | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Telomerase - metabolism | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Telomere - genetics | Cell Line | Green Fluorescent Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Enzyme-Linked Immunosorbent Assay | Cell Cycle Proteins - metabolism | Cells, Cultured | Cytokines - secretion | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | Cellular Senescence - physiology | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Nuclear Proteins - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Interleukin-6 - secretion | Blotting, Western | Fibroblasts - radiation effects | Checkpoint Kinase 2 | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Signal Transduction - physiology | Fibroblasts - cytology | DNA Damage | Microscopy, Fluorescence | Physiological aspects | Cellular signal transduction | Genetic aspects | Research | Cytokines | DNA damage | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2011, Volume 286, Issue 42, pp. 36396 - 36403
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9188 - e9188
Cellular senescence irreversibly arrests cell proliferation in response to oncogenic stimuli. Human cells develop a senescence-associated secretory phenotype... 
HUMAN-FIBROBLASTS | EPITHELIAL-CELLS | DNA-DAMAGE | IN-VIVO | TRIGGERS SENESCENCE | BIOLOGY | GENE-EXPRESSION | GROWTH-FACTOR | CANCER PROGRESSION | CELLULAR SENESCENCE | REPLICATIVE SENESCENCE | Genomic Instability | Fibroblasts - secretion | Matrix Metalloproteinases - genetics | Proteome - genetics | Epithelial Cells - metabolism | Species Specificity | Fibroblasts - physiology | Humans | Insulin-Like Growth Factor Binding Protein 6 - genetics | Transplantation, Heterologous | Intracellular Signaling Peptides and Proteins - metabolism | Oxygen - metabolism | Neoplasms, Experimental - pathology | Epithelial Cells - physiology | Epithelial Cells - secretion | Neoplasms, Experimental - genetics | Insulin-Like Growth Factor Binding Protein 6 - metabolism | Proteomics - methods | Interleukin-6 - metabolism | Intracellular Signaling Peptides and Proteins - genetics | DNA-Binding Proteins | Fibroblasts - metabolism | Cellular Senescence - genetics | Interleukin-6 - genetics | Oxygen - physiology | Mice, Inbred C57BL | Cells, Cultured | Chromosomal Proteins, Non-Histone | Cellular Senescence - physiology | Tumor Burden | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Phenotype | Animals | Mice, Nude | Mice | DNA Damage | Matrix Metalloproteinases - metabolism | Neoplasms, Experimental - metabolism | Tumor Suppressor p53-Binding Protein 1 | Proteome - metabolism | Physiological aspects | Genetic aspects | Cytokines | Cells | Cell proliferation | Cell culture | Senescence | Matrix metalloproteinases | Laboratories | Epithelial cells | DNA damage | Radiation | Genomes | Genotype & phenotype | Cell growth | Cell cycle | Fibroblasts | Aging | Physiology | Life sciences | Tumorigenesis | Vascular endothelial growth factor | Telomerase | Age | Deoxyribonucleic acid--DNA | Phenotypes | Oxygen | Secretion | Breast cancer | Gene expression | Studies | Chemokines | Esophageal cancer | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article