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Motriz: Revista de Educação Física, ISSN 1980-6574, 05/2017, Volume 23, Issue spe
Abstract The validity and relevance of research with animals for the development of knowledge in Exercise Science have for long been discussed. Given the... 
SPORT SCIENCES
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, p. e0146795
A series of studies have demonstrated that activation of the sympathetic nervous system (SNS) causes osteopenia via β2-adrenoceptor (β2-AR) signaling. However,... 
Receptors, Adrenergic, alpha-2 - genetics | Gene Expression | Thyrotoxicosis - blood | Signal Transduction | Femur - metabolism | Thyrotoxicosis - complications | Bone Diseases, Metabolic - etiology | Receptors, Adrenergic, alpha-2 - metabolism | Spine - metabolism | Spine - physiopathology | Thyroxine - blood | Femur - physiopathology | Mice, Knockout | Triiodothyronine - blood | Biomechanical Phenomena | Phenotype | Animals | Thyrotoxicosis - genetics | Bone Diseases, Metabolic - blood | Female | Tibia - metabolism | Bone Remodeling | Tibia - physiopathology | Femur | Spine | Nervous system | Thyroid gland | Hormones | Bone (trabecular) | Inactivation | Osteoblasts | Bend strength | Genotype & phenotype | Osteoporosis | Computed tomography | Tibia | Acid resistance | Biocompatibility | Bone strength | Osteopenia | Thyroid | Vertebrae | Young adults | Hyperthyroidism | Bone turnover | Metabolism | Gene expression | Studies | Bone mass | Ligands | Acid phosphatase (tartrate-resistant) | Mice | Hyperactivity | Genes | Vertebra | Mouse devices | Rodents | Bones | Skeleton | Thyroid hormones | Bone loss | Puberty | Osteocalcin | Deactivation | Bone remodelling | Mechanical properties | Sympathetic nervous system | Augmented reality | Biomechanics | Signaling | Collagen | Osteoclasts | Norepinephrine | Adrenergic receptors | Bone | Acid phosphatase | Receptors (physiology)
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, p. e33175
Myocardial remodeling and heart failure (HF) are common sequelae of many forms of cardiovascular disease and a leading cause of mortality worldwide.... 
HYPERTROPHY | KINASE-C-BETA | UBIQUITIN-PROTEASOME SYSTEM | IN-VIVO | BIOLOGY | CARDIAC DYSFUNCTION | DEATH | DILATED CARDIOMYOPATHY | BORTEZOMIB | HYPERTENSIVE-RATS | EPSILON | Protein Kinase C beta | Phosphorylation | Rats, Wistar | Humans | Middle Aged | Immunoblotting | Male | Heart Failure - prevention & control | Protein Kinase C - metabolism | Adult | Female | Rats, Inbred Dahl | Heart Failure - etiology | Animals, Newborn | Cell Survival - drug effects | Oligopeptides - chemical synthesis | Myocytes, Cardiac - cytology | Cells, Cultured | Rats | Heart Failure - metabolism | Protein Kinase C - antagonists & inhibitors | Rats, Sprague-Dawley | Myocardial Infarction - complications | Animals | Myocytes, Cardiac - drug effects | Proteins - metabolism | Hypertension - complications | Myocytes, Cardiac - metabolism | Aged | Protein Kinase Inhibitors - pharmacology | Proteasome Endopeptidase Complex - metabolism | Oligopeptides - pharmacology | Ventricular Remodeling - drug effects | Heart | Neonates | Animal models | Protein kinase C | Heart attacks | Kinases | Accumulation | Proteins | Protein folding | Rodents | Surgery | Inhibition | Damage accumulation | Physical education | Heart diseases | Heart failure | Hypertension | Hematology | Complications | Cardiomyocytes | Inhibitors | Quality control | Proteasomes | Hypoxia | Disruption | Cardiovascular diseases | Cancer | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e98012
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e45699
Journal Article
Acta Scientiarum. Biological Sciences, ISSN 1679-9283, 08/2013, Volume 35, Issue 3, pp. 437 - 443
Activation of β2 adrenergic receptors by catecholamine or catecholamine-mimetic substances may enhance insulin secretion. We herein investigated KCl- and... 
pancreatic beta cells | adrenergic receptors | glucose, leucine
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2011, Volume 6, Issue 12, p. e29004
Tubule-interstitial nephritis (TIN) results in decreased renal function and interstitial inflammation, which ultimately leads to fibrosis. Excessive adenine... 
KIDNEY-DISEASE | FIBROSIS | DANGER | XANTHINE OXIDOREDUCTASE | OXIDASE | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | INJURY | URIC-ACID | DEHYDROGENASE | Xanthine Dehydrogenase - metabolism | Inflammation - pathology | Inflammasomes - metabolism | Xanthine Dehydrogenase - antagonists & inhibitors | Adenine - pharmacology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Disease Progression | Adenine - administration & dosage | Mice, Knockout | Allopurinol - therapeutic use | Animals | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Diet | Inflammasomes - drug effects | Signal Transduction - drug effects | Nephritis, Interstitial - prevention & control | Allopurinol - pharmacology | Nephritis, Interstitial - drug therapy | Mice | Kidney Tubules - pathology | Kidney Tubules - metabolism | Myeloid Differentiation Factor 88 - metabolism | Nephritis | Care and treatment | Analysis | Collagen | Genes | Gout | Inflammation | Uric acid | Oxidative stress | Nephrology | Renal function | Dehydrogenases | Laboratories | Xanthine dehydrogenase | Caspase-1 | Proteins | Signal transduction | Receptors | Immunology | Rodents | Toll-like receptors | Physical education | Deposition | Food | Immune system | Hypertension | Enzymes | NF-κB protein | Cytokines | Allopurinol | Adenine | Caspase | Xanthine | Metabolism | Gene expression | Precipitates | Medicine | Signaling | TLR2 protein | Fibrosis | MyD88 protein | Infiltration | Kidney diseases | Clinical medicine
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, pp. e108543 - e108543
Nephrotoxicity is substantial side effect for 30% of patients undergoing cancer therapy with cisplatin and may force them to change or even abandon the... 
CELLS | APOPTOSIS | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | HEME OXYGENASE-1 | INTERLEUKIN-6 | RENAL INJURY | CANCER | CHEMOTHERAPY | ADIPOSE-TISSUE | LYMPHOCYTES | Tumor Necrosis Factors - genetics | Heme Oxygenase-1 - metabolism | Apoptosis - drug effects | Male | Acute Kidney Injury - genetics | Cisplatin - administration & dosage | Heme Oxygenase-1 - genetics | Inflammation - metabolism | Cachexia - etiology | Acute Kidney Injury - chemically induced | Interleukin-6 - metabolism | Physical Conditioning, Animal | Disease Models, Animal | Interleukin-6 - genetics | Acute Kidney Injury - pathology | Gene Expression Regulation | Tumor Necrosis Factors - metabolism | Cisplatin - pharmacology | Acute Kidney Injury - physiopathology | Acute Kidney Injury - prevention & control | Cachexia - prevention & control | Animals | Cisplatin - adverse effects | Inflammation - genetics | Mice | Physical exercise | Inflammatory response | Biology | Interleukin 6 | Immunology | Rodents | Tumor necrosis factor-TNF | Gastrocnemius muscle | Physical education | Kidneys | Cytokines | Medical treatment | Muscles | Inflammation | Metabolism | Aerobics | Cisplatin | Urea | Musculoskeletal system | Physical fitness | Exercise physiology | Tumor necrosis factor | Cell death | Interleukin 10 | Regulation | Kidney diseases | Laboratory animals | Cancer | Tumors | Apoptosis | Index Medicus
Journal Article
Revista Brasileira de Educação Física e Esporte, ISSN 1807-5509, 06/2013, Volume 27, Issue 2, pp. 289 - 296
A metodologia dialética propõe um ensino de dupla mão (professor-aluno) que provoque a aprendizagem por meio de tarefas contínuas dos sujeitos. Para isso, o... 
Educação física | Ensino | Esporte
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 03/2018, Volume 22, Issue 3, pp. 1452 - 1463
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, p. e0127843
Background Besides its role as a fuel source in intermediary metabolism, lactate has been considered a signaling molecule modulating lactate-sensitive genes... 
LACTIC-ACID | SKELETAL-MUSCLE | METABOLISM | HUMAN ASTROCYTOMA-CELLS | PGC-1-ALPHA | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | MCT1 | SUPEROXIDE-DISMUTASE | MYOCARDIAL VOLUME OVERLOAD | DEHYDROGENASE | Up-Regulation | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Signal Transduction | Antioxidants - metabolism | Lactic Acid - metabolism | Gene Expression Regulation | NADPH Oxidases - metabolism | Ventricular Function | Rats | Male | Hydrogen Peroxide - metabolism | Catalase - metabolism | Animals | Energy Metabolism | Myocardium - metabolism | Heart Ventricles - metabolism | Hemodynamics | In Vitro Techniques | NAD - metabolism | Superoxide Dismutase - metabolism | Oxidases | RNA | Genes | Physiological aspects | Muscles | Oxidation-reduction reaction | Mitochondrial DNA | Heart | Oxidative stress | Regulations | Reactive oxygen species | Membranes | Hydrogen peroxide | Dehydrogenases | Genomics | Gene regulation | Oxidase | mRNA | NAD(P)H oxidase | Exercise | NADH | Catalase | Metabolites | Rodents | Physiology | Oxidation | Physical education | Fluorimetry | Heart diseases | Deoxyribonucleic acid--DNA | Heart failure | Cardiac muscle | Metabolism | Gene expression | NADH oxidase | Skeletal muscle | Polymerase chain reaction | Musculoskeletal system | Signaling | Nuclear fuels | Muscle fatigue | Nicotinamide adenine dinucleotide | Ventricle | Lactic acid | Spectrophotometry | Laboratory animals | Acetic acid | Deoxyribonucleic acid | DNA
Journal Article