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Biopolymers, ISSN 0006-3525, 08/2016, Volume 105, Issue 8, pp. 594 - 607
Journal Article
Nature Reviews Cancer, ISSN 1474-175X, 01/2016, Volume 16, Issue 1, pp. 35 - 42
Journal Article
Journal Article
Journal Article
Nature Reviews Cancer, ISSN 1474-175X, 02/2015, Volume 15, Issue 3, pp. 166 - 180
Journal Article
PLoS Pathogens, ISSN 1553-7366, 10/2013, Volume 9, Issue 10
  The innate immune system is critical in the response to infection by pathogens and it is activated by pattern recognition receptors (PRRs) binding to... 
Medical research | Vaccines | Cytokines | Kinases | Immune system
Journal Article
Nucleic Acids Research, ISSN 1362-4962, 01/2011, Volume 39, Issue 1, pp. 313 - 324
TopBP1 is a scaffold protein that coordinates activation of the DNA-damage-checkpoint response by coupling binding of the 9-1-1 checkpoint clamp at sites of... 
Journal Article
Nature Structural and Molecular Biology, ISSN 1545-9993, 07/2016, Volume 23, Issue 7, pp. 647 - 655
The opposing activities of 53BP1 and BRCA1 influence pathway choice in DNA double-strand-break repair. How BRCA1 counteracts the inhibitory effect of 53BP1 on... 
STRAND BREAK REPAIR | RING-RING COMPLEX | TUMOR SUPPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | REPAIR PATHWAY CHOICE | BRCA1 | REMODELING ENZYME | CELL BIOLOGY | E3 LIGASE | BIOPHYSICS | END RESECTION | HOMOLOGOUS RECOMBINATION | FUN30 | Chromatin - metabolism | DNA, Neoplasm - metabolism | Humans | Gene Expression Regulation, Neoplastic | Ubiquitin - metabolism | DNA Breaks, Double-Stranded | Ubiquitination - drug effects | BRCA1 Protein - metabolism | Tumor Suppressor Proteins - genetics | Cloning, Molecular | Escherichia coli - metabolism | Binding Sites | Chromatin - drug effects | DNA Helicases - genetics | Chromatin - chemistry | Tumor Suppressor p53-Binding Protein 1 - metabolism | Recombinant Proteins - metabolism | Gene Expression | Recombinational DNA Repair | Tumor Suppressor Proteins - metabolism | Tumor Suppressor p53-Binding Protein 1 - genetics | Signal Transduction | Ubiquitin-Protein Ligases - metabolism | Models, Molecular | Recombinant Proteins - genetics | Ubiquitin - genetics | Piperazines - pharmacology | DNA Cleavage - drug effects | BRCA1 Protein - genetics | DNA Helicases - metabolism | Phthalazines - pharmacology | Histones - genetics | Escherichia coli - genetics | Protein Binding | DNA, Neoplasm - genetics | HeLa Cells | Histones - metabolism | Ubiquitin-Protein Ligases - genetics | Camptothecin - pharmacology | Breast cancer | BRCA mutations | DNA repair | Analysis | Risk factors | Enzymes | Protein expression | Chromatin | DNA damage
Journal Article
Current Cancer Drug Targets, ISSN 1568-0096, 10/2003, Volume 3, Issue 5, pp. 301 - 323
Understanding the mode of action of Hsp90 requires that molecular detail of its interactions with client proteins and co-chaperones are known. The structure... 
ONCOLOGY | Sequence Homology, Amino Acid | DNA Gyrase - metabolism | Animals | HSP90 Heat-Shock Proteins - physiology | Humans | Adenosine Triphosphatases - metabolism | HSP90 Heat-Shock Proteins - chemistry | Models, Molecular | Protein Conformation | Structure-Activity Relationship
Journal Article
Nature Structural and Molecular Biology, ISSN 1545-9993, 07/2012, Volume 19, Issue 7, pp. 685 - 692
Journal Article