X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (6) 6
mice (5) 5
cell line (4) 4
activation (3) 3
biochemistry & molecular biology (3) 3
crescentic glomerulonephritis (3) 3
health aspects (3) 3
index medicus (3) 3
nephrotic syndrome (3) 3
phosphorylation (3) 3
physiological aspects (3) 3
protein kinases (3) 3
protein-serine-threonine kinases - metabolism (3) 3
receptors, igg - physiology (3) 3
akt (2) 2
b-cells (2) 2
benzothiophene inhibitors (2) 2
cells, cultured (2) 2
disease models, animal (2) 2
gene expression (2) 2
heat-shock proteins - metabolism (2) 2
humans (2) 2
immunology (2) 2
inflammation (2) 2
intracellular signaling peptides and proteins - metabolism (2) 2
kidney diseases (2) 2
lipopolysaccharides - pharmacology (2) 2
lps (2) 2
macrophages (2) 2
mice, transgenic (2) 2
mitogens (2) 2
murine peritoneal-macrophages (2) 2
nf-kappa-b (2) 2
p38 mapk (2) 2
p38 mitogen-activated protein kinases - metabolism (2) 2
phosphatidylinositol-3,4,5-trisphosphate 5-phosphatases (2) 2
physiology (2) 2
podocyte injury (2) 2
protein-kinase (2) 2
protein-serine-threonine kinases - genetics (2) 2
proto-oncogene proteins c-akt (2) 2
renal injury (2) 2
research article (2) 2
signal transduction (2) 2
urology & nephrology (2) 2
5-phosphatase ship2 (1) 1
activated protein-kinase (1) 1
activated protein-kinase-2 mk-2 (1) 1
activation motif (1) 1
acute disease (1) 1
acute renal failure (1) 1
analysis (1) 1
animal models (1) 1
articles (1) 1
biochemistry (1) 1
biology (1) 1
biology, molecular (1) 1
bone marrow cells (1) 1
care and treatment (1) 1
cell differentiation (1) 1
cellular stress (1) 1
cellular stress responses (1) 1
cellular-survival (1) 1
cercopithecus aethiops (1) 1
children & youth (1) 1
comparative analysis (1) 1
cos cells (1) 1
cox-2 (1) 1
cytokine production (1) 1
cytokines (1) 1
developmental biology (1) 1
diabetes (1) 1
diabetic-nephropathy (1) 1
disease (1) 1
disease susceptibility (1) 1
down-regulation (1) 1
doxorubicin nephropathy (1) 1
embryonic stem-cells (1) 1
enzyme activation (1) 1
enzyme activation - physiology (1) 1
enzyme inhibitors - pharmacology (1) 1
enzyme-linked immunosorbent assay (1) 1
enzymes (1) 1
epidemiology (1) 1
fc-gamma-r (1) 1
fc-gamma-riib (1) 1
female (1) 1
focal segmental glomerulosclerosis (1) 1
gene deletion (1) 1
gene dosage (1) 1
gene expression regulation (1) 1
gene expression regulation, enzymologic - genetics (1) 1
gene expression regulation, enzymologic - immunology (1) 1
gene-expression (1) 1
glomerular disease (1) 1
glomerulonephritis (1) 1
glomerulonephritis - genetics (1) 1
glomerulonephritis - metabolism (1) 1
glomerulonephritis - physiopathology (1) 1
glycogen (1) 1
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 09/2011, Volume 301, Issue 3, pp. 509 - 519
While mitogen-activated protein kinase (MAPK) activation has been implicated in the pathogenesis of various glomerular diseases, including nephrotic syndrome... 
P38 MAPK | HSPB1 | Inhibitor | Nephrotic syndrome | Serum albumin | Cox-2 | CRESCENTIC GLOMERULONEPHRITIS | OXIDATIVE STRESS | PHYSIOLOGY | TGF-BETA | serum albumin | p38 MAPK | nephrotic syndrome | HEAT-SHOCK-PROTEIN | P38 MAP KINASE | ACTIVATED PROTEIN-KINASE-2 MK-2 | TUMOR-NECROSIS-FACTOR | inhibitor | EMBRYONIC STEM-CELLS | BENZOTHIOPHENE INHIBITORS | UROLOGY & NEPHROLOGY | RENAL INJURY | Intracellular Signaling Peptides and Proteins - drug effects | Nephrotic Syndrome - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Neoplasm Proteins - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | p38 Mitogen-Activated Protein Kinases - metabolism | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Cell Line | Puromycin Aminonucleoside - pharmacology | Podocytes - metabolism | Heat-Shock Proteins - metabolism | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Nephrotic Syndrome - pathology | Imidazoles - pharmacology | Podocytes - pathology | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Podocytes - drug effects | Serum Albumin - pharmacology | Signal Transduction - drug effects | Nephrotic Syndrome - physiopathology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Protein-Serine-Threonine Kinases - drug effects | Signal Transduction - physiology | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Physiological aspects | Care and treatment | Kidney diseases | Protein kinases | Health aspects
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 04/2018, Volume 314, Issue 4, pp. F602 - F613
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e54239
Elevated mitogen-activated protein kinase p38 (p38 MAPK) signaling has been implicated in various experimental and human glomerulopathies, and its inhibition... 
SMALL HSP PHOSPHORYLATION | CRESCENTIC GLOMERULONEPHRITIS | IN-VITRO | ACTIVATION | PODOCYTE INJURY | MAPKAP KINASES | MULTIDISCIPLINARY SCIENCES | P38 MAPK | NEPHROTIC SYNDROME | RENAL INJURY | CELLULAR STRESS | Acute Disease | Phosphorylation | Signal Transduction | Heat-Shock Proteins - metabolism | Humans | Gene Expression Regulation | Glomerulonephritis - genetics | Protein-Serine-Threonine Kinases - genetics | Stress, Physiological | p38 Mitogen-Activated Protein Kinases - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Glomerulonephritis - metabolism | Kidney Function Tests | Mice, Knockout | Heat-Shock Proteins - genetics | Animals | Gene Deletion | Female | Mice | p38 Mitogen-Activated Protein Kinases - metabolism | Glomerulonephritis - physiopathology | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Glomerulonephritis | Physiological aspects | Heat shock proteins | Disease susceptibility | Comparative analysis | Health aspects | Mitogens | Epidemiology | Protein kinases | Pediatrics | Animal models | Syngeneic grafts | Biochemistry | Phylogeny | Kinases | Proteins | Signal transduction | Rodents | Gene dosage | Enzymes | Cytokines | MAP kinase | Gene expression | Substrates | Stress | Children & youth | Signaling | Hospitals | Protein kinase | Diabetes | Kidney diseases | Stress proteins | Kidney transplantation
Journal Article
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2003, Volume 278, Issue 25, pp. 22657 - 22663
SHIP-2, a recently identified inositol 5'-phosphatase, shares high level homology with SHIP-1. Although the role of SHIP-1 has been extensively studied, the... 
B-CELLS | ACTIVATION MOTIF | INSULIN | PHAGOCYTOSIS | TYROSINE PHOSPHORYLATION | PHOSPHATIDYLINOSITOL 3,4,5-TRISPHOSPHATE | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | LIPOPOLYSACCHARIDE-INDUCED SUPPRESSION | TRANSDUCTION | 5-PHOSPHATASE SHIP2
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2004, Volume 279, Issue 52, p. 54416
Fcγ receptor (FcγR) clustering by immune complexes activates multiple signaling pathways leading to phagocytosis. We and others have previously reported that... 
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2004, Volume 279, Issue 52, pp. 54416 - 54425
Fcgamma receptor (FcgammaR) clustering by immune complexes activates multiple signaling pathways leading to phagocytosis. We and others have previously... 
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2004, Volume 173, Issue 1, pp. 360 - 366
LPS stimulates monocytes/macrophages through TLR4, resulting in the activation of a series of signaling events that potentiate the production of inflammatory... 
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2003, Volume 278, Issue 25, p. 22657
SHIP-2, a recently identified inositol 5′-phosphatase, shares high level homology with SHIP-1. Although the role of SHIP-1 has been extensively studied, the... 
Journal Article
Monocytes and macrophages express innate immune receptors such as FcgammaR and Toll-like receptor 4, which when engaged by their respective ligands; IgG immune... 
Inflammation | Akt | Macrophages | Inositol phosphatases | LPS
Dissertation
01/2005, ISBN 0542242206
Monocytes and macrophages express innate immune receptors such as FcγR and Toll-like receptor 4, which when engaged by their respective ligands; IgG immune... 
Molecular biology
Dissertation
Thesis (Ph. D.)--Ohio State University, 2005. Title from first page of PDF file. Document formatted into pages; contains xv, 152 p.; also includes graphics... 
Macrophages. Inflammation Inositol. Phosphatases. Protein kinases. Endotoxins
Dissertation
No results were found for your search.

Cannot display more than 1000 results, please narrow the terms of your search.