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Nephrology Dialysis Transplantation, ISSN 0931-0509, 05/2018, Volume 33, Issue suppl_1, pp. i440 - i440
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2019, Volume 14, Issue 1, p. e0210859
Journal Article
Kidney International, ISSN 0085-2538, 02/2018, Volume 93, Issue 2, pp. 308 - 310
The incidence and prevalence of diabetic kidney disease is increasing. Observational and interventional studies suggest that the pathophysiology varies between... 
UROLOGY & NEPHROLOGY | Prognosis | Diabetic Nephropathies | Humans | Incidence | Nephrology | Kidneys | Diabetes mellitus | Gene expression | Metabolism | Patients | Studies | Genotype & phenotype | Nephropathy | Cocitation | Biopsy | Phenotyping | Biomarkers | Diabetes | Growth factors
Journal Article
Diabetologia, ISSN 0012-186X, 07/2019, Volume 62, Issue 7, pp. 1154 - 1166
Aims/hypothesis The sodium-glucose cotransporter 2 (SGLT2) inhibitor canagliflozin slows progression of kidney function decline in type 2 diabetes. The aim of... 
Type 2 diabetes | MORTALITY | Chronic kidney disease | RENAL-DISEASE | FUNCTION DECLINE | Canagliflozin | PATHWAY | Biomarkers | Sodium-glucose cotransporter 2 inhibitors | PROXIMAL TUBULE | CKD | TNF RECEPTORS 1 | EMPAGLIFLOZIN | PROGRESSION | Sodium–glucose cotransporter 2 inhibitors | ENDOCRINOLOGY & METABOLISM | Biomarkers - metabolism | Cell Line | Sodium-Glucose Transporter 2 Inhibitors - therapeutic use | Fibrosis - drug therapy | Humans | Kidney Diseases - drug therapy | Diabetes Mellitus, Type 2 - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Fibrosis - metabolism | Fibronectins - metabolism | Renal Insufficiency, Chronic - drug therapy | Renal Insufficiency, Chronic - metabolism | Inflammation - metabolism | Matrix Metalloproteinase 7 - metabolism | Canagliflozin - therapeutic use | Inflammation - drug therapy | Diabetes Mellitus, Type 2 - drug therapy | Interleukin-6 - metabolism | Kidney Diseases - metabolism | Fibronectins | Medical research | Chronic kidney failure | Analysis | Fibrosis | Medicine, Experimental | Development and progression | Inflammation | Glucose | Biological markers | Dextrose | Tumor necrosis factor receptors | Diabetes mellitus | Matrix metalloproteinase | Fibronectin | Interleukin 6 | Proteins | Sodium | Tumor necrosis factor | Extracellular matrix | Plasma levels | Metalloproteinase | Diabetes | Kidney diseases | Diabetes mellitus (non-insulin dependent) | Matrilysin
Journal Article
Human Genomics, ISSN 1473-9542, 2011, Volume 5, Issue 6, pp. 709 - 717
Since 1998, the bioinformatics, systems biology, genomics and medical communities have enjoyed a synergistic relationship with the GeneCards database of human... 
Human genes | Set Distiller | Genetic variations | GeneDecks | Genomics | Partner Hunter | Genecards | Omics | Database | Synthetic lethality | Databases, Genetic | Genes - genetics | Genome, Human | Humans | Computational Biology | database | omics | genetic variations | synthetic lethality | GeneCards | genomics | human genes
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, p. e0169518
Objective To evaluate tacrolimus as therapeutic option for diabetic nephropathy (DN) based on molecular profile and network-based molecular model comparisons.... 
ENDOTHELIN ANTAGONISTS | GROWTH-FACTOR-BETA | ACTIVATION | CYCLOSPORINE-A | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MARKERS | TNF-ALPHA | CHRONIC KIDNEY-DISEASE | CALCINEURIN INHIBITORS | PROGRESSION | Computational Biology - methods | Diabetic Nephropathies - metabolism | Humans | Transcriptome | Diabetic Nephropathies - drug therapy | Diabetic Nephropathies - genetics | Gene Expression Profiling | Mesangial Cells - metabolism | Mesangial Cells - drug effects | Models, Biological | Tacrolimus - pharmacology | Biomarkers | Immunosuppressive Agents - pharmacology | Dosage and administration | Research | Tacrolimus | Drug therapy | Genetic polymorphisms | Diabetic nephropathies | Cell culture | Nephrology | Renal function | Endothelin | Transplants & implants | Calcium | Interleukin | Glucose | Kinases | Experiments | Proteins | Calcium signalling | Signal transduction | Interleukin 4 | Annotations | Pathways | Rodents | Network analysis | Mesangial cells | Collagen (type IV) | Growth factors | Immune system | Damage assessment | Immune response | Transforming growth factor-b1 | Diabetes mellitus | MAP kinase | Adherens junctions | Inflammation | Endothelin 1 | Tumor necrosis factor-α | Gene expression | Studies | Molecular modelling | Nephropathy | Data sets | Computer applications | Fibrosis | Angiotensin | Interleukin 10 | Diagnostic systems | Kidney diseases | Diabetes | Kidney transplantation
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, p. e104164
Journal Article
Diabetes Care, ISSN 0149-5992, 09/2018, Volume 41, Issue 9, pp. 1947 - 1954
Journal Article
Oncotarget, ISSN 1949-2553, 11/2018, Volume 9, Issue 91, pp. 36379 - 36391
In recent years, the concept of synthetic lethality, describing a cellular state where loss of two genes leads to a non-viable phenotype while loss of one gene... 
Drug combination | Breast cancer | Treatment | Synthetic lethality | Cancer
Journal Article
Diabetes Care, ISSN 0149-5992, 03/2017, Volume 40, Issue 3, pp. 391 - 397
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 02/2008, Volume 294, Issue 2, pp. C543 - 554