2007, Medical intelligence unit (Unnumbered : 2003), ISBN 0387722033, 214
Attempts to dissect the complexity of the molecular mechanisms of insulin action with a special emphasis on those features of the system that are subject to...
Insulin | Pre-clinical Medicine: Basic Sciences | Physiological effect
Insulin | Pre-clinical Medicine: Basic Sciences | Physiological effect
Book
Frontiers in Endocrinology, ISSN 1664-2392, 2013, Volume 4, p. 71
For many years, adipose tissue was considered as an inert energy storage organ that accumulates and stores triacylglycerols during energy excess and releases...
Adipocyte | Inflammation | Adipokine | Macrophages | Metabolism | Insulin | macrophages and metabolism
Adipocyte | Inflammation | Adipokine | Macrophages | Metabolism | Insulin | macrophages and metabolism
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 01/2018, Volume 19, Issue 1, p. 201
One of the main causes of hyperglycemia is inefficient or impaired glucose utilization by skeletal muscle, which can be exacerbated by chronic high caloric...
Metabolomics | Mitochondrial bioenergetics | Transcriptomics | TCA cycle | Glycolysis | Glucose | Mangiferin | glucose | ENERGY-PRODUCTION | HIGH-FAT DIET | BIOCHEMISTRY & MOLECULAR BIOLOGY | glycolysis | mitochondrial bioenergetics | GAPDH ACTIVITY | mangiferin | CHEMISTRY, MULTIDISCIPLINARY | PYRUVATE-DEHYDROGENASE COMPLEX | RECEPTOR-ALPHA | SKELETAL-MUSCLE | SUCCINATE-DEHYDROGENASE | metabolomics | GLUCOSE-METABOLISM | INSULIN-RESISTANCE | GLYCERALDEHYDE-3-PHOSPHATE DEHYDROGENASE | transcriptomics | Cell Line | DNA, Mitochondrial - metabolism | Mice, Inbred C57BL | Citric Acid Cycle - drug effects | Glyceraldehyde-3-Phosphate Dehydrogenases - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Xanthones - pharmacology | Glycolysis - drug effects | Succinate Dehydrogenase - genetics | Animals | Metabolome - drug effects | Mitochondria - genetics | Diet, High-Fat | Succinate Dehydrogenase - metabolism | Mice | Glyceraldehyde-3-Phosphate Dehydrogenases - metabolism | Energy Metabolism - drug effects | Tricarboxylic acid cycle | Transcription factors | Succinate dehydrogenase | Transcription | Muscles | Metabolism | Insulin | Skeletal muscle | High fat diet | Electron transport chain | Glucose metabolism | Musculoskeletal system | Hyperglycemia | Mitochondria | Molecular modelling | Metabolites | Bioenergetics | Electron transport
Metabolomics | Mitochondrial bioenergetics | Transcriptomics | TCA cycle | Glycolysis | Glucose | Mangiferin | glucose | ENERGY-PRODUCTION | HIGH-FAT DIET | BIOCHEMISTRY & MOLECULAR BIOLOGY | glycolysis | mitochondrial bioenergetics | GAPDH ACTIVITY | mangiferin | CHEMISTRY, MULTIDISCIPLINARY | PYRUVATE-DEHYDROGENASE COMPLEX | RECEPTOR-ALPHA | SKELETAL-MUSCLE | SUCCINATE-DEHYDROGENASE | metabolomics | GLUCOSE-METABOLISM | INSULIN-RESISTANCE | GLYCERALDEHYDE-3-PHOSPHATE DEHYDROGENASE | transcriptomics | Cell Line | DNA, Mitochondrial - metabolism | Mice, Inbred C57BL | Citric Acid Cycle - drug effects | Glyceraldehyde-3-Phosphate Dehydrogenases - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Xanthones - pharmacology | Glycolysis - drug effects | Succinate Dehydrogenase - genetics | Animals | Metabolome - drug effects | Mitochondria - genetics | Diet, High-Fat | Succinate Dehydrogenase - metabolism | Mice | Glyceraldehyde-3-Phosphate Dehydrogenases - metabolism | Energy Metabolism - drug effects | Tricarboxylic acid cycle | Transcription factors | Succinate dehydrogenase | Transcription | Muscles | Metabolism | Insulin | Skeletal muscle | High fat diet | Electron transport chain | Glucose metabolism | Musculoskeletal system | Hyperglycemia | Mitochondria | Molecular modelling | Metabolites | Bioenergetics | Electron transport
Journal Article
Physiology & Behavior, ISSN 0031-9384, 09/2019, Volume 208, p. 112581
FAM19A2/TAFA-2, a member of the chemokine CC family, shares 31% sequence identity with MIP-1α, which is known to elevate body temperature and reduce food...
FAM19A2/TAFA-2 | Food intake | Metabolic cage | Body temperature | BEHAVIORAL SCIENCES | PSYCHOLOGY, BIOLOGICAL
FAM19A2/TAFA-2 | Food intake | Metabolic cage | Body temperature | BEHAVIORAL SCIENCES | PSYCHOLOGY, BIOLOGICAL
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 20, pp. 7578 - 7591
The systems integration of whole-body metabolism and immune signaling are central homeostatic mechanisms necessary for maintenance of normal physiology, and...
ACTIVATION | UNFOLDED PROTEIN RESPONSE | TP53-INDUCED GLYCOLYSIS | IMMUNE-SYSTEM | GLUCOSE-METABOLISM | INSULIN-RESISTANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | IKK-BETA | KAPPA-B | APOPTOSIS REGULATOR TIGAR | CELL-DEATH
ACTIVATION | UNFOLDED PROTEIN RESPONSE | TP53-INDUCED GLYCOLYSIS | IMMUNE-SYSTEM | GLUCOSE-METABOLISM | INSULIN-RESISTANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | IKK-BETA | KAPPA-B | APOPTOSIS REGULATOR TIGAR | CELL-DEATH
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, p. 7019
Pancreatic beta-cell loss through apoptosis is an important disease mechanism in type 2 diabetes. Apoptosis Repressor with CARD (ARC) is a cell death inhibitor...
SURVIVAL | APOPTOSIS REPRESSOR | ER STRESS | MULTIDISCIPLINARY SCIENCES | DEATH | MECHANISMS | ENDOPLASMIC-RETICULUM STRESS | INHIBITOR | PROTEINS | FAILURE | CASPASE RECRUITMENT DOMAIN | Insulin-Secreting Cells - physiology | Nerve Tissue Proteins - metabolism | Animals | Islets of Langerhans - pathology | Cell Survival | Cytoskeletal Proteins - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Transcription Factor CHOP - metabolism | Insulin Secretion | Disease Models, Animal | Mice, Knockout | Animal models | Cell survival | Secretion | Diabetes mellitus | CCAAT/enhancer-binding protein | Gene deletion | Insulin | Intolerance | Hyperglycemia | Clonal deletion | Cell death | Diabetes | Islets of Langerhans | Pancreas | Apoptosis | Structure-function relationships
SURVIVAL | APOPTOSIS REPRESSOR | ER STRESS | MULTIDISCIPLINARY SCIENCES | DEATH | MECHANISMS | ENDOPLASMIC-RETICULUM STRESS | INHIBITOR | PROTEINS | FAILURE | CASPASE RECRUITMENT DOMAIN | Insulin-Secreting Cells - physiology | Nerve Tissue Proteins - metabolism | Animals | Islets of Langerhans - pathology | Cell Survival | Cytoskeletal Proteins - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Transcription Factor CHOP - metabolism | Insulin Secretion | Disease Models, Animal | Mice, Knockout | Animal models | Cell survival | Secretion | Diabetes mellitus | CCAAT/enhancer-binding protein | Gene deletion | Insulin | Intolerance | Hyperglycemia | Clonal deletion | Cell death | Diabetes | Islets of Langerhans | Pancreas | Apoptosis | Structure-function relationships
Journal Article
7.
Full Text
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Autophagy, ISSN 1554-8627, 01/2016, Volume 12, Issue 1, pp. 1 - 222
stress | chaperone-mediated autophagy | vacuole | autolysosome | macroautophagy | flux | autophagosome | LC3 | lysosome | phagophore | Phagophore | Lysosome | Autolysosome | Flux | Autophagosome | Vacuole | Chaperonemediated autophagy | Macroautophagy | Stress | ACTIVATED PROTEIN-KINASE | ENDOPLASMIC-RETICULUM STRESS | STARVATION-INDUCED AUTOPHAGY | GLUCAGON-INDUCED AUTOPHAGY | CELL BIOLOGY | PROGRAMMED CELL-DEATH | LIFE-SPAN EXTENSION | BETAINE HOMOCYSTEINE METHYLTRANSFERASE | VACUOLAR MEMBRANE DYNAMICS | NF-KAPPA-B | Autophagy - physiology | Biological Assay - methods | Animals | Biological Assay - standards | Computer Simulation | Humans | Life Sciences | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 12/2018, Volume 315, Issue 6, pp. F1658 - F1669
Muscle dysfunction is an important cause of morbidity among patients with chronic kidney disease (CKD). Although muscle fibrosis is present in a CKD rodent...
UNITED-STATES | chronic kidney disease | PHYSIOLOGY | MECHANICAL-PROPERTIES | FORCE TRANSMISSION | DIALYSIS | inflammation | UROLOGY & NEPHROLOGY | HEMODIALYSIS | fibrosis | EXTRACELLULAR-MATRIX | PHYSICAL-ACTIVITY | SATELLITE CELL | skeletal muscle | STAGE RENAL-DISEASE | CONNECTIVE-TISSUE
UNITED-STATES | chronic kidney disease | PHYSIOLOGY | MECHANICAL-PROPERTIES | FORCE TRANSMISSION | DIALYSIS | inflammation | UROLOGY & NEPHROLOGY | HEMODIALYSIS | fibrosis | EXTRACELLULAR-MATRIX | PHYSICAL-ACTIVITY | SATELLITE CELL | skeletal muscle | STAGE RENAL-DISEASE | CONNECTIVE-TISSUE
Journal Article