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Positive IgG Western Blot for Borrelia burgdorferi in Colombia, 12/1999
In order to evaluate the presence of specific IgG antibodies to Borrelia burgdorferi in patients with clinical manifestations associated with Lyme borreliosis... 
Borrelia burgdorferi - Colombia - morphea (localized scleroderma) - Lyme borreliosis - spirochete
Journal
Neuroscience Letters, ISSN 0304-3940, 04/2018, Volume 671, pp. 38 - 42
Lyme neuroborreliosis, caused by the gram-negative bacterium may affect the central and/or peripheral nervous systems. In previous studies, we showed that... 
Innate immunity | Lyme neuroborreliosis | Toll-like receptor | Oligodendrocytes | PATHOGENESIS | APOPTOSIS | CELLS | RECOGNITION | RNA | INDUCED INFLAMMATION | RECEPTORS | NEUROSCIENCES | Immunohistochemistry | Tyrosine | Lyme disease | Medical research | Analysis | Medicine, Experimental | Bacteria | Inflammation | Index Medicus | oligodendrocytes | innate immunity
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 05/2017, Volume 14, Issue 1, pp. 110 - 110
Background: In previous studies, human oligodendrocytes were demonstrated to undergo apoptosis in the presence of Borrelia burgdorferi under an inflammatory... 
Human oligodendrocytes | Inflammation | Lyme neuroborreliosis | B. burgdorferi | Receptor tyrosine kinases | ACTIVATION | PROTEIN-COUPLED RECEPTORS | IMMUNOLOGY | GLIAL-CELLS | NEUROSCIENCES | PATHOGENESIS | NEUROINFLAMMATION | TRANSACTIVATION | GROWTH-FACTOR RECEPTOR-3 | EXPRESSION | PROGRESSION | NEUROBORRELIOSIS | RNA, Small Interfering - genetics | Cytokines - metabolism | Apoptosis - drug effects | Humans | Phosphatidylcholines - pharmacology | Enzyme Inhibitors - pharmacology | Epidermal Growth Factor - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Oligodendroglia - microbiology | Dose-Response Relationship, Drug | Cell Differentiation - genetics | Receptor Protein-Tyrosine Kinases - genetics | Signal Transduction - drug effects | Borrelia burgdorferi - pathogenicity | Cell Differentiation - drug effects | Inflammation Mediators - metabolism | Apoptosis - physiology | Cell Line, Transformed | RNA, Small Interfering - metabolism | Immunohistochemistry | Lyme disease | Fibroblast growth factor | Platelet-derived growth factor | Pathogenesis | Nervous system | Glucose | Kinases | Interleukin 6 | Proteins | Signal transduction | Epidermal growth factor | Oligodendrocytes | Toll-like receptors | Fibroblasts | Bacteria | Growth factors | Protein-tyrosine kinase | Tyrosine | TLR5 protein | Epidermal growth factor receptors | Extracellular signal-regulated kinase | siRNA | TLR4 protein | Gene expression | Cell death | TLR2 protein | Cancer | Apoptosis | Fibroblast growth factor receptors | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. e29914 - e29914
The persistence of symptoms in Lyme disease patients following antibiotic therapy, and their causes, continue to be a matter of intense controversy. The... 
VLSE | LYME-DISEASE SPIROCHETE | IMMUNE-RESPONSE | C-6 TEST | TERMINAL INVARIABLE DOMAIN | NONHUMAN PRIMATE MODEL | IXODES-DAMMINI | PERIPHERAL NERVOUS-SYSTEM | MULTIDISCIPLINARY SCIENCES | OUTER SURFACE PROTEIN | ANTIBODY-RESPONSE | Inflammation - pathology | Antibody Formation - immunology | Inflammation - microbiology | Peptides - immunology | Macaca mulatta - immunology | Treatment Outcome | Lyme Disease - pathology | Macaca mulatta - microbiology | Anti-Bacterial Agents - therapeutic use | Inflammation - complications | Lyme Disease - complications | Animals | DNA, Bacterial - genetics | Lyme Disease - drug therapy | Xenodiagnosis | Antibody Formation - drug effects | DNA, Bacterial - analysis | Anti-Bacterial Agents - pharmacology | Lyme Disease - microbiology | Borrelia burgdorferi - drug effects | Viral antibodies | Lyme disease | Drug resistance in microorganisms | Care and treatment | Antibiotics | RNA | Primates | Antibodies | Diagnosis | Health aspects | Spirochetes | Health sciences | Therapy | Laboratories | Ticks | Disseminated infection | Monkeys | Infections | Ceftriaxone | Veterinary medicine | Tissues | Experiments | Proteins | Immunology | Penicillin | Research centers | Deoxyribonucleic acid--DNA | Vector-borne diseases | Medical research | Pathogens | Antigens | Parasitology | Gene expression | Ribonucleic acid--RNA | Monkeys & apes | Bacteriology | Pathogenicity | Lipoproteins | Doxycycline | Diagnostic systems | Immunofluorescence | Index Medicus | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 02/2017, Volume 14, Issue 1, pp. 28 - 28
Journal Article