Australian Veterinary Journal, ISSN 0005-0423, 05/2017, Volume 95, Issue 5, p. 173
Journal Article
2017, Fifth edition., ISBN 1626374511, xi, 457 pages
Book
1998, Current problems in cardiology, Volume 23, no. 11., 643-717
Book
Pathology, ISSN 0031-3025, 02/2017, Volume 49, pp. S33 - S34
Journal Article
1967, 72
Book
1987, Field note / Forest Engineering Research Institute of Canada, Volume Trucking-10., [2] leaves
Book
7.
Full Text
PIK3CA mutations in breast cancer: Reconciling findings from preclinical and clinical data
Breast Cancer Research, ISSN 1465-5411, 01/2014, Volume 16, Issue 1, pp. 201 - 201
PIK3CA mutations represent one of the most common genetic aberrations in breast cancer. They have been reported to be present in over one-third of cases, with...
ACTIVATION | P110-ALPHA | PHOSPHATIDYLINOSITOL 3-KINASES | TRASTUZUMAB | GENE | ONCOLOGY | PI3K PATHWAY | GROWTH | PTEN | RESISTANCE | GAIN | Humans | Treatment Outcome | Signal Transduction - genetics | Proto-Oncogene Proteins c-akt - genetics | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | TOR Serine-Threonine Kinases - genetics | Female | Mice | Receptor, ErbB-2 - antagonists & inhibitors | Mutation | Disease Models, Animal | Health aspects | Epidermal growth factor | Review
ACTIVATION | P110-ALPHA | PHOSPHATIDYLINOSITOL 3-KINASES | TRASTUZUMAB | GENE | ONCOLOGY | PI3K PATHWAY | GROWTH | PTEN | RESISTANCE | GAIN | Humans | Treatment Outcome | Signal Transduction - genetics | Proto-Oncogene Proteins c-akt - genetics | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | TOR Serine-Threonine Kinases - genetics | Female | Mice | Receptor, ErbB-2 - antagonists & inhibitors | Mutation | Disease Models, Animal | Health aspects | Epidermal growth factor | Review
Journal Article
2009, 4th ed., ISBN 1588266389, xiv, 433
Book
Nature, ISSN 0028-0836, 09/2015, Volume 525, Issue 7567, pp. 119 - 123
Breast cancer is the most frequent cancer in women and consists of heterogeneous types of tumours that are classified into different histological and molecular...
STEM-CELLS | ACTIVATION | ETV6-NTRK3 | MAMMARY-GLAND DEVELOPMENT | PACKAGE | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | MUTATIONS | IDENTIFICATION | FATE | CANCER | Receptors, Estrogen - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Genes, p53 - genetics | Mutation - genetics | Breast Neoplasms - metabolism | Receptors, Progesterone - metabolism | Phosphatidylinositol 3-Kinases - genetics | Cell Differentiation - genetics | Cell Lineage | Mammary Neoplasms, Animal - pathology | Phenotype | Animals | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Breast Neoplasms - pathology | Cell Transformation, Neoplastic | Cell Division | Mammary Neoplasms, Animal - metabolism | Female | Mice | Neoplasm Invasiveness - genetics | Mammary Neoplasms, Animal - genetics | Breast tumors | Genetic aspects | Gene mutations | Health aspects | Oncogenes | Rodents | Medical prognosis | Mortality | Principal components analysis | Breast cancer | Mutation | Gene expression | Tumors | Life Sciences | Cellular Biology | Cancer
STEM-CELLS | ACTIVATION | ETV6-NTRK3 | MAMMARY-GLAND DEVELOPMENT | PACKAGE | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | MUTATIONS | IDENTIFICATION | FATE | CANCER | Receptors, Estrogen - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Genes, p53 - genetics | Mutation - genetics | Breast Neoplasms - metabolism | Receptors, Progesterone - metabolism | Phosphatidylinositol 3-Kinases - genetics | Cell Differentiation - genetics | Cell Lineage | Mammary Neoplasms, Animal - pathology | Phenotype | Animals | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Breast Neoplasms - pathology | Cell Transformation, Neoplastic | Cell Division | Mammary Neoplasms, Animal - metabolism | Female | Mice | Neoplasm Invasiveness - genetics | Mammary Neoplasms, Animal - genetics | Breast tumors | Genetic aspects | Gene mutations | Health aspects | Oncogenes | Rodents | Medical prognosis | Mortality | Principal components analysis | Breast cancer | Mutation | Gene expression | Tumors | Life Sciences | Cellular Biology | Cancer
Journal Article
Critical Reviews in Oncogenesis, ISSN 0893-9675, 2012, Volume 17, Issue 1, pp. 69 - 95
The phosphatidylinositol 3 kinase (PI3K) pathway is one of the major pathways modulating cell growth, proliferation, metabolism, survival, and angiogenesis....
Anticancer drugs | Clinical trials | Phosphatidylinositol 3 kinase | Targeted therapeutics | Kinase inhibitors | Combinational therapy | TOR Serine-Threonine Kinases - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Receptor Cross-Talk - physiology | Proto-Oncogene Proteins c-akt - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | TOR Serine-Threonine Kinases - genetics | TOR Serine-Threonine Kinases - physiology | Proto-Oncogene Proteins c-akt - metabolism | Molecular Targeted Therapy - methods | Proto-Oncogene Proteins c-akt - physiology | Signal Transduction - genetics | Neoplasms - drug therapy | Phosphatidylinositol 3-Kinases - genetics | Protein Kinase Inhibitors - administration & dosage | Animals | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Models, Biological | Phosphatidylinositol 3-Kinases - physiology | Feedback, Physiological - drug effects | Feedback, Physiological - physiology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Receptor Cross-Talk - drug effects
Anticancer drugs | Clinical trials | Phosphatidylinositol 3 kinase | Targeted therapeutics | Kinase inhibitors | Combinational therapy | TOR Serine-Threonine Kinases - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Receptor Cross-Talk - physiology | Proto-Oncogene Proteins c-akt - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | TOR Serine-Threonine Kinases - genetics | TOR Serine-Threonine Kinases - physiology | Proto-Oncogene Proteins c-akt - metabolism | Molecular Targeted Therapy - methods | Proto-Oncogene Proteins c-akt - physiology | Signal Transduction - genetics | Neoplasms - drug therapy | Phosphatidylinositol 3-Kinases - genetics | Protein Kinase Inhibitors - administration & dosage | Animals | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Models, Biological | Phosphatidylinositol 3-Kinases - physiology | Feedback, Physiological - drug effects | Feedback, Physiological - physiology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Receptor Cross-Talk - drug effects
Journal Article
1978, Monographs of the High/Scope Educational Research Foundation, ISBN 0931114047, Volume no. 5., xii, 70
Book
1968, University of Maryland. Agricultural Experiment Station. Miscellaneous publication 623, viii, 102
Book
1990, Working papers / Population Council, Volume no. 17., 34
Book
1999, 2nd ed., ISBN 1555877036, xv, 411
Book
1992, ISBN 9781555872960, xx, 367
Book
AMERICAN JOURNAL OF NEURORADIOLOGY, ISSN 0195-6108, 08/2000, Volume 21, Issue 7, pp. 1363 - 1363
Journal Article
1994, 1st ed., Complete library of Christian worship, ISBN 1562330179, Volume 7, xliii, 495
Book
1994, Complete library of Christian worship, ISBN 1562330152, Volume 5, xlv, 497
Book
1994, Complete library of Christian worship, ISBN 1562330160, Volume 6, xliii, 366
Book
1993, 1st ed., Complete library of Christian worship, ISBN 9781562330132, Volume 3, xxxv, 435
Book
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