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Molecular Therapy - Nucleic Acids, ISSN 2162-2531, 01/2014, Volume 3, Issue 9, pp. e195 - e195
The microRNA(miRNA)-34a is a key regulator of tumor suppression. It controls the expression of a plethora of target proteins involved in cell cycle,... 
delivery | multiple myeloma | cell cycle | miR-34a | DNA damage | nanotechnology | apoptosis | microRNA | cancer | p53 | MicroRNA | Cell cycle | Multiple myeloma | Delivery | Nanotechnology | Apoptosis | Cancer | MULTIPLE-MYELOMA | MEDICINE, RESEARCH & EXPERIMENTAL | PANCREATIC-CANCER | DOWN-REGULATION | CELL-PROLIFERATION | ANTITUMOR-ACTIVITY | FEEDBACK LOOP | COLON-CANCER | IN-VITRO | REGULATING MIR-34A | MICRORNA EXPRESSION | Review
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 07/2013, Volume 228, Issue 7, pp. 1506 - 1515
Journal Article
Journal of Hematology and Oncology, ISSN 1756-8722, 05/2018, Volume 11, Issue 1, pp. 63 - 19
The deeper understanding of non-coding RNAs has recently changed the dogma of molecular biology assuming protein-coding genes as unique functional biological... 
Epigenetics | Experimental therapeutics | MALAT1 | lncRNA | Non-coding RNA | Long non-coding RNA | MULTIPLE-MYELOMA | DOWN-REGULATION | CERVICAL-CANCER | CANCER CELL-PROLIFERATION | LUNG-CANCER | LNCRNA MALAT1 | MESSENGER-RNA | INTEGRATIVE ANNOTATION | ONCOLOGY | POOR-PROGNOSIS | HEMATOLOGY | UP-REGULATION | Physiological aspects | Antisense RNA | Care and treatment | Research | Methods | Cancer
Journal Article
Leukemia, ISSN 0887-6924, 09/2018, Volume 32, Issue 9, pp. 1948 - 1957
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e89659
Background & Aim: The miR-221/222 cluster is upregulated in malignant plasma cells from multiple myeloma (MM) patients harboring the t(4;14) translocation. We... 
MIR-221 | SURVIVAL | RENAL FIBROSIS | GLIOBLASTOMA | PROSTATE CARCINOMA | INHIBITION | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | ANTITUMOR-ACTIVITY | MICRORNA EXPRESSION | PROGRESSION | MicroRNAs - therapeutic use | MicroRNAs - antagonists & inhibitors | Humans | Gene Silencing | Male | Antineoplastic Agents - therapeutic use | Mice, SCID | Molecular Targeted Therapy | MicroRNAs - pharmacology | Xenograft Model Antitumor Assays | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Multiple Myeloma - drug therapy | Animals | Cell Line, Tumor | Mice, Inbred NOD | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Gene Expression Regulation, Neoplastic - drug effects | Multiple Myeloma - genetics | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Immunohistochemistry | RNA | Analysis | Multiple myeloma | Cell proliferation | Severe combined immunodeficiency | Toxicity | Leukemia | Oncology | Infections | Kinases | Anticancer properties | Antitumor agents | Xenografts | Biocompatibility | Phosphorothioate | Translocation | Medical research | Diabetes mellitus | Cyclin-dependent kinases | Immunodeficiency | Organs | Antisense oligonucleotides | Gene expression | Ribonucleic acid--RNA | College campuses | Medicine | Inhibitors | Acids | MicroRNAs | Cyclin-dependent kinase inhibitor p27 | Plasma cells | In vivo methods and tests | Clinical medicine | In vitro methods and tests | Cancer | Tumors | Ribonucleic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e90005
Journal Article
Nanomedicine: Nanotechnology, Biology, and Medicine, ISSN 1549-9634, 2011, Volume 7, Issue 6, pp. 955 - 964
Journal Article