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1. Full Text Anti–neurofascin-155 IgG4 antibodies prevent paranodal complex formation in vivo
by Manso, Constance and Manso, Constance and Querol, Luis and Querol, Luis and Lleixà, Cinta and Lleixà, Cinta and Poncelet, Mallory and Poncelet, Mallory and Mekaouche, Mourad and Mekaouche, Mourad and Vallat, Jean-Michel and Vallat, Jean-Michel and Illa, Isabel and Illa, Isabel and Devaux, Jerome J and Devaux, Jérôme J
The Journal of clinical investigation, ISSN 0021-9738, 06/2019, Volume 129, Issue 6, pp. 2222 - 2236
Autoimmunity | Neuromuscular disease | Neuroscience | Autoimmune diseases | Neurological disorders | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Humans | Cell Adhesion Molecules - antagonists & inhibitors | Rats, Inbred Lew | Male | Polyneuropathies - drug therapy | Cell Adhesion Molecules - immunology | Schwann Cells - immunology | Motor Neurons - pathology | Polyradiculoneuropathy - pathology | Immunoglobulin G - immunology | HEK293 Cells | Immunoglobulin G - pharmacology | Female | Motor Neurons - immunology | Rats | Schwann Cells - pathology | Nerve Growth Factors - immunology | Polyneuropathies - immunology | Animals | Polyneuropathies - pathology | Polyradiculoneuropathy - immunology | Axons - pathology | Nerve Growth Factors - antagonists & inhibitors | Axons - immunology | Chronic Disease | Polyradiculoneuropathy - drug therapy | Immunoglobulin G | Cerebellum | Neonates | Intravenous administration | Pathogenesis | Insertion | Nervous system | Polyneuropathy | Neuronal-glial interactions | Cell adhesion molecules | Cell adhesion & migration | Proteins | Nerve conduction | Specialization | Clonal deletion | Demyelination | Contactin | Nerves | Physiology | Immunoglobulins | Tremor | Inflammation | Patients | Biopsy | Internalization | Morphology | Axon guidance | Index Medicus | Abridged Index Medicus | Life Sciences | Immunology | Neurons and Cognition | Neurobiology | Cellular Biology | Adaptive immunology | Cell Behavior
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2. Full Text CTCF modulates allele-specific sub-TAD organization and imprinted gene activity at the mouse Dlk1-Dio3 and Igf2-H19 domains
by Lleres, David and Moindrot, Benoit and Pathak, Rakesh and Piras, Vincent and Matelot, Melody and Pignard, Benoit and Marchand, Alice and Poncelet, Mallory and Perrin, Aurelien and Tellier, Virgile and Feil, Robert and Noordermeer, Daan
Genome biology, ISSN 1474-760X, 12/2019, Volume 20, Issue 1, pp. 272 - 272
Genetics & Heredity | Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Science & Technology | Genomic Imprinting | Animals | Insulin-Like Growth Factor II - genetics | CCCTC-Binding Factor - metabolism | Mice | RNA, Long Noncoding - genetics | Iodide Peroxidase - genetics | Calcium-Binding Proteins - genetics | Chromatin | Transcription activation | Genes | Alleles | Stem cells | DNA methylation | Insulin-like growth factor II | Preadipocyte factor 1 | Chromosomes | Deoxyribonucleic acid--DNA | Binding sites | Genomic imprinting | Index Medicus | Life Sciences | Meg3 | Igf2 | CTCF | Dlk1 | 3D genome organization
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3. Full Text Biallelic mutations in neurofascin cause neurodevelopmental impairment and peripheral demyelination
by Efthymiou, Stephanie and Salpietro, Vincenzo and Malintan, Nancy and Poncelet, Mallory and Kriouile, Yamna and Fortuna, Sara and De Zorzi, Rita and Payne, Katelyn and Henderson, Lindsay and Cortese, Andrea
Brain (London, England : 1878), ISSN 0006-8950, 10/2019, Volume 142, Issue 10, pp. 2948 - 2964
Life Sciences | Immunology | Neurons and Cognition | Neurobiology | Cellular Biology | Adaptive immunology | Cell Behavior
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