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Cell Death and Disease, ISSN 2041-4889, 10/2018, Volume 9, Issue 10, pp. 1002 - 11
Infection with Influenza A virus (IAV) causes significant cell death within the upper and lower respiratory tract and lung parenchyma. In severe infections,... 
NS1 PROTEIN | SECONDARY NECROSIS | IN-VITRO | DENDRITIC CELLS | EPITHELIAL-CELLS | EXTRACELLULAR VESICLES | INDUCED APOPTOSIS | ALVEOLAR MACROPHAGES | INFECTED-CELLS | NF-KAPPA-B | CELL BIOLOGY | Epithelial cells | Parenchyma | Infections | T cell receptors | Leukocytes (neutrophilic) | Macrophages | Respiratory tract | Monocytes | Lungs | Cell death | Influenza | Alveoli | Apoptosis
Journal Article
Blood, ISSN 0006-4971, 03/2010, Volume 115, Issue 12, pp. 2473 - 2482
Under normal physiologic conditions, necrotic cells resulting from tissue injury are rapidly removed from the circulation and tissues by phagocytes, thus... 
C-REACTIVE PROTEIN | SYSTEMIC-LUPUS-ERYTHEMATOSUS | TERMINAL DOMAIN | MANNOSE-BINDING LECTIN | CROSS-LINKING | SERUM-AMYLOID-P | HEMATOLOGY | HUMAN MONOCYTES | LATE APOPTOTIC CELLS | INSOLUBLE IMMUNE-COMPLEXES | ANTIPHOSPHOLIPID SYNDROME
Journal Article
Trends in Cell Biology, ISSN 0962-8924, 2016, Volume 27, Issue 2, pp. 151 - 162
The disassembly of an apoptotic cell into subcellular fragments, termed apoptotic bodies (ApoBDs), is a hallmark of apoptosis. Although the generation of... 
Pathology | apoptosis | cell clearance | apoptotic bodies | HORIZONTAL TRANSFER | IMPAIRED UPTAKE | PHAGOCYTOSIS | APOPTOTIC CELL CLEARANCE | EXTRACELLULAR VESICLES | MEMBRANE | CASPASE-MEDIATED ACTIVATION | BODIES | FIND-ME SIGNAL | T-CELLS | CELL BIOLOGY | Cell Communication | Animals | Models, Biological | Humans | Apoptosis | Cell death
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 06/2018, Volume 9, p. 1486
Journal Article
PLoS ONE, ISSN 1932-6203, 2014, Volume 9, Issue 10, p. e109596
Heparanase is a beta-D-endoglucuronidase that cleaves heparan sulfate (HS), facilitating degradation of the extracellular matrix (ECM) and the release of... 
ADHESION | TOLL-LIKE RECEPTOR-4 | ROLES | CELL-MIGRATION | MULTIDISCIPLINARY SCIENCES | LIPOPOLYSACCHARIDE | MAMMALIAN HEPARANASE | LUNG INJURY | DEGRADING ENZYME | EXPRESSION | T-CELLS | Leukocytes, Mononuclear - drug effects | Cytokines - metabolism | Heparitin Sulfate - pharmacology | Leukocytes, Mononuclear - secretion | Humans | Cytokines - secretion | Extracellular Space - drug effects | Glucuronidase - metabolism | Solubility | Heparitin Sulfate - metabolism | Toll-Like Receptor 4 - metabolism | Gene Expression Regulation - drug effects | Extracellular Space - metabolism | Animals | Signal Transduction - drug effects | Heparitin Sulfate - chemistry | Leukocytes, Mononuclear - cytology | Mice | Myeloid Differentiation Factor 88 - metabolism | Leukocyte migration | Leukocytes (mononuclear) | Cardiovascular disease | Arthritis | Biochemistry | Metastasis | Sulfates | Cell surface | Fragmentation | Metastases | Degradation | Interleukin 6 | Proteins | Angiogenesis | Splenocytes | Rodents | Atherosclerosis | Toll-like receptors | Peripheral blood mononuclear cells | Extracellular matrix | Biomolecules | Interleukin 8 | Immune system | Spleen | Heparan sulfate | NF-κB protein | Wound healing | Cytokines | Fragments | Inflammation | TLR4 protein | White blood cells | Tumor necrosis factor | Interleukin 10 | MyD88 protein | Sepsis | Diabetes | Sulfate | Cell migration | Chemokines | Monocyte chemoattractant protein 1 | Tumors
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 507, Issue 7492, pp. 329 - 334
Journal Article