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Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 2/2015, Volume 35, Issue 2, pp. 329 - 337
Journal Article
Pharmaceutical Research, ISSN 0724-8741, 3/2019, Volume 36, Issue 3, pp. 1 - 14
Fast-acting insulin aspart (faster aspart) is a novel formulation of insulin aspart containing two additional excipients: niacinamide, to increase early... 
Biochemistry, general | Biomedicine | oligomerization | absorption | fast-acting insulin aspart | vasodilation | Pharmacy | niacinamide | Medical Law | Pharmacology/Toxicology | Biomedical Engineering and Bioengineering | absorption; fast-acting insulin aspart; niacinamide; oligomerization; vasodilation | SUBCUTANEOUS INJECTION | GLYCEMIC CONTROL | PROFILES | HYDROTROPIC SOLUBILIZATION | NICOTINAMIDE | BLOOD-FLOW | CHEMISTRY, MULTIDISCIPLINARY | DOUBLE-BLIND | PHARMACOLOGY & PHARMACY | ONSET | Hypoglycemic Agents - pharmacokinetics | Endothelial Cells - metabolism | Humans | Cells, Cultured | Insulin Aspart - pharmacokinetics | Scattering, Small Angle | Subcutaneous Absorption - drug effects | Diabetes Mellitus, Type 1 - drug therapy | Dose-Response Relationship, Drug | Insulin Aspart - administration & dosage | Animals | Regional Blood Flow - drug effects | Hypoglycemic Agents - administration & dosage | Models, Biological | X-Ray Diffraction | Diabetes Mellitus, Type 1 - blood | Injections, Subcutaneous | Subcutaneous Tissue - blood supply | Female | Niacinamide - pharmacology | Vasodilation - drug effects | Subcutaneous Tissue - metabolism | Sus scrofa | Subcutaneous Tissue - drug effects | Oligomers | Niacinamide | Arginine | Swine | Vasodilators | Permeability | Insulin | Endothelium | Pharmacology | Stability analysis | Small angle X ray scattering | Arteries | Monomers | Endothelial cells | Vasodilation | Blood flow | X-ray scattering | Absorption | Perfusion | Skin | X ray scattering | Transport | Research Paper
Journal Article
Journal Article
Journal of Molecular Neuroscience, ISSN 0895-8696, 8/2017, Volume 62, Issue 3, pp. 380 - 394
Subarachnoid hemorrhage (SAH) is a serious clinical condition where leakage of blood into the subarachnoid space causes an acute rise in intracranial pressure... 
Neurochemistry | Neurology | Animal model | Neurosciences | Biomedicine | MEK1/2 inhibition | Proteomics | SAH | Mass spectrometry | Cell Biology | VASOSPASM | 14-3-3 PROTEINS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | NEUROSCIENCES | ADHESION | 5-HT1B | UP-REGULATION | Proteome - genetics | Rats | Male | Subarachnoid Hemorrhage - pathology | Enzyme Inhibitors - therapeutic use | Rats, Sprague-Dawley | MAP Kinase Signaling System | Subarachnoid Hemorrhage - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Cerebral Arteries - metabolism | Butadienes - therapeutic use | Proteome - metabolism | Subarachnoid Hemorrhage - drug therapy | Mitogen-Activated Protein Kinase 1 - metabolism | Nitriles - therapeutic use | Complications and side effects | Subarachnoid hemorrhage | Analysis | Genetic aspects | Cerebral arteries | Research | Gene expression | Risk factors | Subarachnoid space | Networks | Aneurysms | Smooth muscle | mRNA | Intracranial pressure | Hemorrhage | Blood | Arteries | Protein turnover | Proteins | Cerebrovascular system | Ischemia | Protein folding | Cerebral blood flow | Network analysis | Blood pressure | Inhibition | Leakage | Spectroscopy | Extracellular signal-regulated kinase | MAP kinase | Mass spectroscopy | Metabolism | Blood flow | 14-3-3 protein | Protein expression | Scientific imaging | MEK1 | 2 inhibition | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, p. e41852
Background: Global ischemic stroke is one of the most prominent consequences of cardiac arrest, since the diminished blood flow to the brain results in cell... 
INCOMPLETE ISCHEMIA | 5-HT1B RECEPTOR | 2-VESSEL OCCLUSION | BRAIN-INJURY | MULTIDISCIPLINARY SCIENCES | SUBARACHNOID HEMORRHAGE | GLOBAL CEREBRAL-ISCHEMIA | CA1 NEURONS | RAT-BRAIN | BLOOD-FLOW | UP-REGULATION | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Rats, Wistar | Endothelin-1 - pharmacology | Rats | Male | Viper Venoms - pharmacology | Serotonin - pharmacology | Vasoconstriction - drug effects | Prosencephalon - drug effects | Gene Expression Regulation - drug effects | Animals | Carotid Arteries - physiopathology | Prosencephalon - blood supply | Serotonin - analogs & derivatives | Receptor, Serotonin, 5-HT1B - metabolism | Receptor, Endothelin B - metabolism | Prosencephalon - metabolism | Ischemic Attack, Transient - metabolism | Ischemic Attack, Transient - physiopathology | Immunohistochemistry | Brain | Brain damage | Endothelin | Ischemia | Heart | Forebrain | Smooth muscle | Kinases | Serotonin S1 receptors | Hemorrhage | Contraction | Arteries | Western blotting | Cerebrovascular system | Receptors | Reperfusion | Cerebral blood flow | Rodents | Blood pressure | Heart diseases | Stroke | Serotonin | Subarachnoid hemorrhage | Contractility | Muscles | Muscle contraction | Blood flow | Neurological diseases | Perfusion | Hindbrain | Receptor mechanisms | Brain injury | Veins & arteries | Clinical Medicine | Other Clinical Medicine | Annan klinisk medicin | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 12/2012, Volume 9, Issue 1, pp. 274 - 274
Background: Subarachnoid hemorrhage (SAH) is associated with high morbidity and mortality. It is suggested that the associated inflammation is mediated through... 
TNFα | IL-1β | Phosphorylated ERK1/2 | Subarachnoid hemorrhage | MMP-9 | IL-6 | TNF alpha | VASOSPASM | IL-1 beta | NECROSIS-FACTOR-ALPHA | CEREBROSPINAL-FLUID | NEUROSCIENCES | RECEPTOR ANTAGONIST | MEK/ERK PATHWAY | MATRIX METALLOPROTEINASES | NERVOUS-SYSTEM INJURY | ISCHEMIC BRAIN-INJURY | CEREBRAL-BLOOD-FLOW | INFLAMMATORY CYTOKINES | Tumor Necrosis Factor-alpha - metabolism | Motor Activity - drug effects | Male | Astrocytes - enzymology | Extracellular Signal-Regulated MAP Kinases - metabolism | Glial Fibrillary Acidic Protein - metabolism | Matrix Metalloproteinase 9 - metabolism | Time Factors | Matrix Metalloproteinase 9 - genetics | Exploratory Behavior - drug effects | Statistics, Nonparametric | Cytokines - genetics | Subarachnoid Hemorrhage - drug therapy | Disease Models, Animal | Astrocytes - drug effects | Blood Transfusion, Autologous - adverse effects | Cytokines - metabolism | Nervous System Diseases - drug therapy | Rats | Subarachnoid Hemorrhage - complications | Subarachnoid Hemorrhage - pathology | Enzyme Inhibitors - therapeutic use | Rats, Sprague-Dawley | Subarachnoid Hemorrhage - metabolism | Animals | Signal Transduction - drug effects | Cerebral Arteries - enzymology | Cerebral Arteries - pathology | Nervous System Diseases - etiology | Butadienes - therapeutic use | Signal Transduction - physiology | Mitogen-Activated Protein Kinases - metabolism | Nitriles - therapeutic use | Studies | Proteins | Brain damage | Cytokines | Kinases | Veins & arteries | Immunohistochemistry | sensorimotor system | Mortality | Extracellular signal-regulated kinase | MAP kinase | Inflammation | Matrix metalloproteinase | Arteries | Blood | Gelatinase B | Morbidity | Western blotting | Interleukin 6 | Signal transduction | Ischemia | subarachnoid hemorrhage | Tumor necrosis factor- alpha | Neurologi | Clinical Medicine | Neurology | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin
Journal Article
Journal of Cerebral Blood Flow and Metabolism, ISSN 0271-678X, 07/2016, Volume 36, Issue 7, pp. 1195 - 1201
Transient global cerebral ischemia is often followed by delayed disturbances of cerebral blood flow, contributing to neuronal injury. The pathophysiological... 
channel | smooth muscle | cerebral blood flow | ischemia | Neurovascular coupling | NEURONAL-ACTIVITY | K-IR channel | RAT | BRAIN-INJURY | MODEL | BLOOD-FLOW | FOREBRAIN ISCHEMIA | NEUROSCIENCES | CONSTRICTION | CARDIAC-ARREST | ENDOCRINOLOGY & METABOLISM | HEMATOLOGY | ISCHEMIA/REPERFUSION | RESUSCITATION
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92417
Background: Global cerebral ischemia following cardiac arrest is associated with increased cerebral vasoconstriction and decreased cerebral blood flow,... 
CO2 REACTIVITY | RECOVERY | ACTIVATION | CARDIAC-ARREST | TRANSIENT FOREBRAIN ISCHEMIA | THERAPEUTIC HYPOTHERMIA | ADULT RATS | MULTIDISCIPLINARY SCIENCES | DEATH | BLOOD-FLOW | NEURONAL DAMAGE | MAP Kinase Kinase 1 - antagonists & inhibitors | Butadienes - pharmacology | Nitriles - pharmacology | Rats, Wistar | Cerebrovascular Circulation - drug effects | Rats | MAP Kinase Kinase 1 - metabolism | Treatment Outcome | MAP Kinase Kinase 2 - metabolism | Vasoconstriction - drug effects | Hypoxia, Brain - prevention & control | Up-Regulation - drug effects | Receptor, Endothelin B - genetics | Animals | MAP Kinase Kinase 2 - antagonists & inhibitors | Brain Ischemia - drug therapy | Brain Ischemia - pathology | Receptor, Serotonin, 5-HT1B - genetics | Butadienes - therapeutic use | Receptor, Serotonin, 5-HT1B - metabolism | Receptor, Endothelin B - metabolism | Drug Evaluation, Preclinical | Nitriles - therapeutic use | Prevention | Brain | Cerebral ischemia | Neurons | Cell death | Patient outcomes | Brain damage | Mesencephalon | Kinases | Damage prevention | Serotonin S1 receptors | Carotid arteries | Arteries | Cerebrovascular system | Receptors | Ischemia | Cerebral blood flow | Rodents | Surgery | Blood pressure | Inhibition | Heart diseases | Stroke | Cell survival | Mortality | Vasoconstriction | Contractility | Survival | Muscle contraction | Blood flow | Studies | Delayed response | Brain research | Inhibitors | Perfusion | Hypoxia | Immunofluorescence | Brain injury | Hippocampus | Apoptosis | Veins & arteries | Animal cognition | Clinical Medicine | Other Clinical Medicine | Annan klinisk medicin | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
Journal Article
BMC Neuroscience, ISSN 1471-2202, 03/2013, Volume 14, Issue 1, pp. 34 - 34
Background: Upregulation of vasoconstrictor receptors in cerebral arteries, including endothelin B (ETB) and 5-hydroxytryptamine 1B (5-HT1B) receptors, has... 
5-Hydroxytryptamine receptor | Cerebral blood flow | Subarachnoid hemorrhage | Neurological outcome | Endothelin receptor | 5-HT1B RECEPTOR | ACTIVATION | VASOSPASM | CLAZOSENTAN | MODEL | BLOOD-FLOW | NEUROSCIENCES | ARTERIES | REDUCTION | DOUBLE-BLIND | RAT-BRAIN | Motor Activity - physiology | MAP Kinase Signaling System - physiology | Cerebrovascular Circulation - physiology | Nitriles - pharmacology | Area Under Curve | Brain Ischemia - metabolism | Carbon Isotopes | Male | Antipyrine - analogs & derivatives | Up-Regulation - physiology | Receptor, Serotonin, 5-HT1B - genetics | Receptor, Serotonin, 5-HT1B - metabolism | Blood Pressure - physiology | Disease Models, Animal | Butadienes - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Subarachnoid Hemorrhage - complications | Brain Ischemia - etiology | Laser-Doppler Flowmetry | Rats, Sprague-Dawley | Up-Regulation - drug effects | Nervous System Diseases - prevention & control | Receptor, Endothelin B - genetics | Animals | Analysis of Variance | Signal Transduction - drug effects | Cerebral Arteries - metabolism | Nervous System Diseases - etiology | Signal Transduction - physiology | Receptor, Endothelin B - metabolism | Brain Ischemia - mortality | Cerebral ischemia | Endothelin | Analysis | Cerebral circulation | Health aspects | Mitogens | Protein kinases | Blood vessels | Pathology | Brain | Kinases | Rodents | Veins & arteries
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 12/2014, Volume 11, Issue 1, pp. 207 - 207
Journal Article