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Journal Article
Clinical Endocrinology, ISSN 0300-0664, 08/2018, Volume 89, Issue 2, pp. 226 - 232
The second edition Bethesda System for Reporting Thyroid Cytology estimates 6%-18% malignancy rate of category III (B3) and 10%-40% for category IV (B4)... 
atypia of undetermined significance | indeterminate thyroid cytology | suspicious for follicular neoplasm | Bethesda | molecular marker | gene expression classifier | Surgery | Benign | Cytology | Thyroid gland | Malignancy | Gene expression | Thyroid | Nodules
Journal Article
Oncogene, ISSN 0950-9232, 2018, Volume 38, Issue 14, pp. 2565 - 2579
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, pp. e0134901 - e0134901
NF-kappa B signaling plays an important role in tumor cell proliferation, cell survival, angiogenesis, invasion, metastasis and drug/radiation resistance.... 
ACTIVATION | INVASION | INHIBITION | METASTASIS | MANAGEMENT | EFFICACY | INDUCED APOPTOSIS | MULTIDISCIPLINARY SCIENCES | CELL-LINES | CARCINOMA | DOCETAXEL | Caspase 7 - metabolism | Cell Death - radiation effects | Taxoids - pharmacology | Humans | Caspase 3 - metabolism | NF-kappa B - metabolism | Antineoplastic Agents - therapeutic use | Combined Modality Therapy | Bortezomib - pharmacology | Molecular Targeted Therapy | Thyroid Neoplasms - therapy | Signal Transduction - drug effects | Cell Line, Tumor | Inhibitory Concentration 50 | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Cell Death - drug effects | Signal Transduction - radiation effects | Neoplasm Staging | Cell Proliferation - radiation effects | Radiation, Ionizing | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Cell proliferation | Biotechnology | Histone deacetylase | Transcription factors | Iodine radioisotopes | Toxicity | Cytotoxicity | Taxane | Kinases | Cancer therapies | Metastases | Angiogenesis | Signal transduction | Ionizing radiation | Cell growth | Tumor necrosis factor-TNF | Inhibition | Deoxyribonucleic acid--DNA | Thyroid | NF-κB protein | Bortezomib | Cell survival | Iodine 131 | Hydroxamic acid | Pharmacology | Radiation therapy | Tumor cell lines | Metabolism | Patients | Proteasome inhibitors | Medicine | Signaling | Chemotherapy | Thyroid cancer | Inhibitors | Antibiotics | Cell death | Cell lines | Radiation tolerance | Diabetes | Synergistic effect | Endocrinology | Tumors | Apoptosis | Cancer | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 02/2012, Volume 90, Issue 2, pp. 331 - 339
Complete congenital stationary night blindness (cCSNB) is a clinically and genetically heterogeneous group of retinal disorders characterized by nonprogressive... 
COMPLETE FORM | LIGHT RESPONSE | CHANNEL | MGLUR6 | SYNAPTIC-TRANSMISSION | MOUSE MODEL | GENETICS & HEREDITY | NYCTALOPIN | RICH REPEAT PROTEIN | MUTATIONS | CONE ELECTRORETINOGRAM | Electroretinography - methods | Night Blindness - metabolism | Humans | Dark Adaptation - genetics | Retinal Rod Photoreceptor Cells - metabolism | Gene Knockdown Techniques - methods | Myopia - metabolism | Receptors, Metabotropic Glutamate - genetics | Retinal Bipolar Cells - physiology | Chromosome Mapping - methods | Genetic Diseases, X-Linked | Night Blindness - genetics | Retinal Bipolar Cells - metabolism | Eye Diseases, Hereditary | Signal Transduction | Mice, Inbred C57BL | Zebrafish | Mice, Inbred C3H | Myopia - genetics | Animals | Myopia - physiopathology | Pedigree | Night Blindness - physiopathology | Retinal Rod Photoreceptor Cells - physiology | Heterozygote | Mice | Receptors, G-Protein-Coupled - genetics | Mutation | Chromosome mapping | Usage | Gene mutations | Retinal bipolar cells | Causes of | Physiological aspects | Night blindness | Genetic aspects | Research | Analysis | Genetic disorders | Proteins | Rodents | Blindness | Gene expression | Cells | Index Medicus | Animal models | Antibodies | Insertion | stationary night blindness | Retina | Guanine nucleotide-binding protein | Electroretinograms | Depolarization | Signal transduction | Vision | Bipolar cells | Gene mapping | DNA sequencing | Report
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2009, Volume 106, Issue 35, pp. 15043 - 15048
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 4168 - 4168
Journal Article
American Journal of Surgery, The, ISSN 0002-9610, 2015, Volume 210, Issue 6, pp. 1162 - 1169
Abstract Background This study evaluates the outcomes of a protocol to manage hypocalcemia after thyroidectomy (TTX). Methods A review of prospectively... 
Surgery | Post-thyroidectomy | Protocol | Parathyroid hormone | Hypocalcemia
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2012, Volume 123, Issue 1, pp. 44 - 59
NeuroD1 encodes a basic helix‐loop‐helix transcription factor involved in the development of neural and endocrine structures, including the retina and pineal... 
transcriptome profiling | microarray | retina | NeuroD1 | pineal gland | gene expression | NUCLEAR IMPORT | S-ANTIGEN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL FATE | NEUROSCIENCES | RETINAL PHOTORECEPTOR | LEBER CONGENITAL AMAUROSIS | MEKA PHOSDUCIN | MOUSE MODEL | TRANSCRIPTION FACTOR | EXPRESSION | GLAND | Opsins - genetics | Pineal Gland - metabolism | Pineal Gland - ultrastructure | Cell Survival - genetics | Bromodeoxyuridine | RNA, Messenger - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Retina - cytology | Microarray Analysis | Pineal Gland - cytology | Basic Helix-Loop-Helix Transcription Factors - deficiency | Mucoproteins - genetics | Electroretinography | Microscopy, Electron, Transmission | Photoreceptor Cells, Vertebrate - ultrastructure | Basic Helix-Loop-Helix Transcription Factors - genetics | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Mice, Knockout | Transcription Factors - metabolism | Animals | Analysis of Variance | Mucoproteins - deficiency | Opsins - metabolism | Mice | Photoreceptor Cells, Vertebrate - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Retinal Degeneration - pathology | Genetic research | Anopheles | Genetic transcription | Ophthalmology | Analysis | Genes | Studies | Gene expression | Neurons | Recombination | Transcription factors | retinal degeneration | Pineal gland | Homeostasis | Retina | Rod outer segment membranes | Gene deletion | Survival | Cre recombinase | Promoters | Beta2 protein | Electroretinograms | Pinealocytes | DNA microarrays | Photoreceptors | Helix-loop-helix proteins | Age | Index Medicus
Journal Article
Oncogenesis, ISSN 2157-9024, 02/2018, Volume 7, Issue 2, pp. 23 - 14
Advanced stages of papillary and anaplastic thyroid cancer continue to be plagued by a dismal prognosis, which is a result of limited effective therapies for... 
BREAST-CANCER | ONCOLOGY | KINASE | PTEN | DASATINIB | CELL-LINES | MEK INHIBITORS | IDENTIFICATION | REVEALS | FAMILY | VEMURAFENIB | Tyrosine | Phosphorylation | Melanoma | MAP kinase | Tumor cell lines | Cancer therapies | 1-Phosphatidylinositol 3-kinase | Thyroid cancer | Medical prognosis | Cell lines | Protein-tyrosine kinase receptors | Mutation | Protein-tyrosine kinase | Thyroid
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 11/2010, Volume 35, Issue 12, pp. 2440 - 2449
Journal Article