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Biophysical Journal, ISSN 0006-3495, 05/2017, Volume 112, Issue 9, p. 1739
Mechanical interactions between a cell and its environment, or between cells, influence key developmental and physiologic processes as well as many aspect of... 
Cues | Matrix | Cell survival | Cellular biology | Leukocyte migration | War | Mechanical stimuli | Differentiation | Cells | Cell migration
Journal Article
Biophysical Journal, ISSN 0006-3495, 05/2017, Volume 112, Issue 9, pp. 1739 - 1741
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 04/2012, Volume 197, Issue 3, pp. 351 - 360
Journal Article
Cancer Cell, ISSN 1535-6108, 03/2012, Volume 21, Issue 3, pp. 418 - 429
Pancreatic ductal adenocarcinomas (PDAs) are characterized by a robust fibroinflammatory response. We show here that this desmoplastic reaction generates... 
INTERSTITIAL FLUID PRESSURE | SOLID TUMORS | ONCOLOGY | COMPARING STANDARD PANCREATICODUODENECTOMY | HUMAN OSTEOSARCOMA XENOGRAFTS | RANDOMIZED-TRIAL | HEAD ADENOCARCINOMA | TUMOR VASCULATURE | EXTENDED LYMPHADENECTOMY | CANCER | BLOOD-PRESSURE | CELL BIOLOGY | Adenocarcinoma - pathology | Cell Adhesion Molecules - administration & dosage | Stromal Cells - pathology | Pancreatic Neoplasms - blood supply | Deoxycytidine - pharmacology | Hyaluronic Acid - physiology | Polyethylene Glycols - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Pancreatic Neoplasms - drug therapy | Deoxycytidine - therapeutic use | Stromal Cells - drug effects | Hyaluronoglucosaminidase - administration & dosage | Drug Evaluation, Preclinical | Hyaluronoglucosaminidase - therapeutic use | Tumor Microenvironment - drug effects | Animals, Genetically Modified | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Microvessels - drug effects | Adenocarcinoma - drug therapy | Carcinoma, Pancreatic Ductal - pathology | Extracellular Fluid - drug effects | Polyethylene Glycols - administration & dosage | Adenocarcinoma - blood supply | Hyaluronoglucosaminidase - pharmacology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Hyaluronic Acid - metabolism | Mice | Carcinoma, Pancreatic Ductal - blood supply | Cell Adhesion Molecules - therapeutic use | Deoxycytidine - analogs & derivatives | Enzymes | Medical colleges | Care and treatment | Glycosaminoglycans | Oncology, Experimental | Research | Chemotherapy | Universities and colleges | Drug therapy | Hyaluronic acid | Public health | Tumors | Cancer
Journal Article
Oncogene, ISSN 0950-9232, 12/2009, Volume 28, Issue 49, pp. 4326 - 4343
Mammographically dense breast tissue is one of the greatest risk factors for developing breast carcinoma, yet the associated molecular mechanisms remain... 
3D matrix/focal adhesion | Breast cancer | Mechanotransduction | Epithelial morphogenesis | Cell-extracellular matrix interaction | Breast tissue density | CANCER RISK | ACTIVATED PROTEIN-KINASE | mechanotransduction | BIOCHEMISTRY & MOLECULAR BIOLOGY | breast cancer | MALIGNANT PHENOTYPE | CARCINOMA IN-SITU | CELL BIOLOGY | MAMMOGRAPHIC DENSITY | STRESS FIBERS | ONCOLOGY | epithelial morphogenesis | EPITHELIAL-SPECIFIC DISRUPTION | GROWTH-FACTORS | GENETICS & HEREDITY | EXTRACELLULAR-MATRIX | FOCAL ADHESION KINASE | breast tissue density | cell-extracellular matrix interaction | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Cell Count | Humans | Breast Neoplasms - etiology | Gene Expression Profiling | Extracellular Signal-Regulated MAP Kinases - metabolism | Mammary Glands, Human - metabolism | Mechanotransduction, Cellular - physiology | Carcinoma - etiology | Extracellular Matrix - chemistry | Breast Neoplasms - metabolism | Cell Transformation, Neoplastic - genetics | Extracellular Matrix - physiology | Gene Expression - physiology | Female | Mammary Glands, Human - physiology | Extracellular Signal-Regulated MAP Kinases - physiology | Focal Adhesion Kinase 1 - metabolism | Mechanotransduction, Cellular - genetics | Mammary Glands, Human - cytology | Risk Factors | Cells, Cultured | Focal Adhesion Kinase 1 - physiology | Cell Transformation, Neoplastic - metabolism | Phenotype | Breast Neoplasms - genetics | Carcinoma - genetics | Carcinoma - metabolism | Physiological aspects | Development and progression | Genetic aspects | Research | Gene expression | Protein kinases | Risk factors | Genotype & phenotype | Cellular biology | 3D-Matrix | Focal Adhesion | Cell-Extracellular Matrix Interaction | Epithelial Morphogenesis | Breast Cancer | Breast Tissue Density | Tumor-Stroma Interaction
Journal Article
Cancer Research, ISSN 0008-5472, 01/2019, Volume 79, Issue 2, pp. 372 - 386
Pancreatic ductal adenocarcinoma (PDA) remains one of the deadliest forms of cancer, in part, because it is largely refractory to current therapies. The... 
INHIBITION | ONCOLOGY | STELLATE CELLS | EXTRACELLULAR-MATRIX | PHYSICAL BARRIERS | HALOFUGINONE | PHASE-I | CANCER | TUMOR MICROENVIRONMENT | FIBROBLASTS | CHEMOTHERAPY
Journal Article
Biophysical Journal, ISSN 0006-3495, 03/2017, Volume 112, Issue 5, pp. 1023 - 1036
Journal Article
Nature Communications, ISSN 2041-1723, 04/2017, Volume 8, Issue 1, p. 14923
Directed migration by contact guidance is a poorly understood yet vital phenomenon, particularly for carcinoma cell invasion on aligned collagen fibres. We... 
MATRIX | PANCREATIC DUCTAL ADENOCARCINOMA | INVASION | COLLAGEN | ACTIN | MICROENVIRONMENT | PERSISTENCE | MULTIDISCIPLINARY SCIENCES | DYNAMICS | REORGANIZATION | MECHANOTRANSDUCTION
Journal Article