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The EMBO Journal, ISSN 0261-4189, 07/2015, Volume 34, Issue 13, pp. 1759 - 1772
Journal Article
Cell, ISSN 0092-8674, 09/2012, Volume 150, Issue 6, p. 1103
[beta] cell dysfunction with subsequent apoptosis is considered a significant contributor to the development of type 2 diabetes. Emerging data from Talchai et... 
Type 2 diabetes | Pancreatic beta cells
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 03/2009, Volume 20, Issue 6, pp. 1728 - 1736
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 03/2009, Volume 20, Issue 6, pp. 1728 - 1736
Journal Article
Nature cell biology, ISSN 1465-7392, 06/2019, Volume 21, Issue 6, pp. 792 - 792
In the version of this article originally published, the Gene Expression Omnibus (GEO) accession number listed in the data availability section was incorrectly... 
Cell culture | Error correction | Ribonucleic acid--RNA | Gene expression | Clustering | Stem cells
Journal Article
Journal Article
Cell, ISSN 0092-8674, 09/2012, Volume 150, Issue 6, pp. 1103 - 1104
β cell dysfunction with subsequent apoptosis is considered a significant contributor to the development of type 2 diabetes. Emerging data from Talchai et al.... 
BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY
Journal Article
Pancreatology, ISSN 1424-3903, 2017, Volume 17, Issue 3, pp. S8 - S8
Intravenous delivery of adeno-associated virus encoding urocortin2 (AAV.Ucn2) normalized blood glucose in high fat diet (HFD) and db/db mouse models of type II... 
Endocrinology & Metabolism | Gastroenterology and Hepatology | Rodents | Pancreas transplantation | Diabetes | Pancreas | Corticotropin-releasing hormone | Insulin | Metabolic disorders
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, pp. e72213 - e72213
Mutations in the human homolog of the Vhlh gene [encoding the von-Hippel Lindau (VHL) protein] lead to tumor development. In mice, depletion of Vhlh in... 
GLUCAGON-SECRETION | REGULATOR | ALPHA-CELLS | GLUCOSE | STABILIZATION | MULTIDISCIPLINARY SCIENCES | BETA-CELLS | IN-VIVO | INSULIN-SECRETION | GENE-EXPRESSION | HYPOXIA | Embryo, Mammalian | Hypoxia-Inducible Factor 1 - metabolism | Pancreatic Polypeptide-Secreting Cells - metabolism | Hypoxia - metabolism | Hypoxia-Inducible Factor 1 - genetics | Insulin-Secreting Cells - metabolism | Gene Expression Regulation, Developmental | Gene Deletion | Glucagon-Secreting Cells - metabolism | Endocrine System - metabolism | Pancreatic Polypeptide-Secreting Cells - pathology | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Somatostatin-Secreting Cells - pathology | Signal Transduction | Glucagon-Secreting Cells - pathology | Cells, Cultured | Endocrine System - pathology | Mice, Knockout | Hypoxia - genetics | Insulin - metabolism | Animals | Hypoxia - pathology | Glucose - metabolism | Glucagon - metabolism | Mice | Insulin-Secreting Cells - pathology | Somatostatin-Secreting Cells - metabolism | Homeostasis - genetics | Glucose metabolism | Glucagon | Physiological aspects | Homeostasis | Glucose | Insulin | Dextrose | Cell culture | Homology | Lethality | Defects | Rodents | Gastroenterology | Standard deviation | Pancreas | Secretion | Hypoglycemia | Metabolism | Gene expression | Epithelium | Embryos | Depletion | Hypoxia | VHL protein | Islets of Langerhans | Diabetes | Mutation | Neoplasia | Endocrinology | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, pp. e0164714 - e0164714
Journal Article