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Pigment Cell & Melanoma Research, ISSN 1755-1471, 09/2019, Volume 32, Issue 5, pp. 687 - 696
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 02/2019, Volume 11, Issue 2, p. n/a
Journal Article
JOURNAL OF VIROLOGY, ISSN 0022-538X, 10/2019, Volume 93, Issue 20
Cytomegalovirus (CMV) is a ubiquitous betaherpesvirus that infects many different cell types. Human CMV (HCMV) has been found in several solid tumors, and it... 
immunomodulation | LATENT | melanoma | CD8(+) T-CELLS | macrophages | GLIOBLASTOMA | HIGHLY PATHOGENIC SIV | VIROLOGY | cytomegalovirus | MEMORY INFLATION | INFLAMMATION | immunotherapy | DISSEMINATION | LYMPHOMA | EXPRESSION
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 2019, Volume 18, Issue 9, pp. 1637 - 1648
Combined BRAF and MEK inhibition is a standard of care in patients with advanced BRAF-mutant melanoma, but acquired resistance remains a challenge that limits... 
SURVIVAL | THERAPY | ONCOLOGY | KINASE | PLK1 | ACQUIRED-RESISTANCE | COBIMETINIB | COMBINATION | EXPRESSION | DISCOVERY | VEMURAFENIB
Journal Article
Journal of virology, 08/2019
Cytomegalovirus (CMV) is a ubiquitous β-herpesvirus that infects many different cell types. Human (H)CMV has been found in several solid tumors and it has been... 
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 28 - 14
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 105 - 105
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 03/2017, Volume 16, Issue 3, pp. 516 - 528
Patients with metastatic uveal melanoma usually die within 1 year of diagnosis, emphasizing an urgent need to develop new treatment strategies. The liver is... 
CELL LUNG-CANCER | BRAF-MUTATED MELANOMA | ONCOLOGY | GNAQ | REGENERATION | RESISTANCE | LY2801653 | COMBINATION | TUMOR-GROWTH | SF3B1 | SOMATIC MUTATIONS | Proto-Oncogene Proteins c-met - metabolism | Apoptosis - drug effects | Humans | Hepatic Stellate Cells - metabolism | Apoptosis - genetics | Drug Resistance, Neoplasm | JNK Mitogen-Activated Protein Kinases - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Melanoma - genetics | Pyrimidinones - pharmacology | Antineoplastic Agents - pharmacology | Uveal Neoplasms - metabolism | Melanoma - metabolism | GTP-Binding Protein alpha Subunits, Gq-G11 - genetics | Uveal Neoplasms - genetics | Uveal Neoplasms - pathology | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Melanoma - pathology | Uveal Neoplasms - drug therapy | Hepatocyte Growth Factor - metabolism | Signal Transduction - drug effects | Melanoma - drug therapy | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Adaptor Proteins, Signal Transducing - metabolism | Cell Cycle - drug effects | Pyridones - pharmacology | Phosphorylation | Liver | AKT protein | Metastasis | Kinases | Metastases | Proteins | Liver cancer | Conditioning | Explants | Stellate cells | Hepatocyte growth factor | MEK inhibitors | Melanoma | Poly(ADP-ribose) polymerase | MAP kinase | Resistance factors | Patients | 1-Phosphatidylinositol 3-kinase | Ron protein | Signaling | Inhibitors | Hepatocytes | Protein kinase | Stromal cells | Isoforms | Cancer | Apoptosis | melanoma | MEK | skin cancers | signal transduction | liver | uveal melanoma
Journal Article
Nature communications, ISSN 2041-1723, 04/2018, Volume 9, Issue 1, pp. 1404 - 1
In the original version of this Article, financial support was not fully acknowledged. The PDF and HTML versions of the Article have now been corrected to... 
SUMO protein | Phosphorylation | Education | Melanoma | Extracellular signal-regulated kinase | BASIC (programming language) | Sox10 protein
Journal Article
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 01/2018, Volume 17, Issue 1, pp. 84 - 95
Journal Article
Cancer Research, ISSN 0008-5472, 10/2018, Volume 78, Issue 19, pp. 5680 - 5693
MEK-ERK1/2 signaling is elevated in melanomas that are wild-type for both BRAF and NRAS (WT/WT), but patients are insensitive to MEK inhibitors.... 
BREAST-CANCER | BRAF-MUTATED MELANOMA | ONCOLOGY | EPIDERMAL-GROWTH-FACTOR | ADAPTIVE RESISTANCE | CUTANEOUS MELANOMA | PHASE-3 TRIAL | RAF INHIBITORS | FEEDBACK INHIBITION | UP-REGULATION | TUMOR MICROENVIRONMENT | NRAS melanoma | MEK | cancer-associated fibroblasts | NRG1 | wild-type BRAF | ErbB3
Journal Article