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Nature Cell Biology, ISSN 1465-7392, 09/2010, Volume 12, Issue 9, pp. 863 - 875
Accumulation of unwanted/misfolded proteins in aggregates has been observed in airways of patients with cystic fibrosis (CF), a life-threatening genetic... 
APOPTOSIS | UNFOLDED PROTEIN RESPONSE | BECLIN 1 | DOWN-REGULATION | ENDOPLASMIC-RETICULUM | MICE | GENE-TRANSFER | TISSUE TRANSGLUTAMINASE | NEURODEGENERATIVE DISEASES | CELL-DEATH | CELL BIOLOGY | Reactive Oxygen Species - metabolism | Respiratory Mucosa - drug effects | Sequestosome-1 Protein | Humans | Transglutaminases - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Autophagy - drug effects | Young Adult | Cystamine - therapeutic use | Inflammation - metabolism | Autophagy - genetics | Nasal Polyps - metabolism | Mice, Inbred CFTR | Respiratory Mucosa - cytology | Cystic Fibrosis - metabolism | Membrane Proteins - genetics | Mice, Transgenic | Protein Transport - genetics | Apoptosis Regulatory Proteins - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Models, Biological | Cystic Fibrosis - genetics | Transglutaminases - metabolism | Adolescent | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mice | GTP-Binding Proteins | Cystic Fibrosis - drug therapy | Protein Binding - physiology | Microtubule-Associated Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Salicylates - pharmacology | Epithelial Sodium Channels - genetics | Apoptosis Regulatory Proteins - genetics | Adult | Cystamine - pharmacology | Membrane Proteins - metabolism | Acetylcysteine - therapeutic use | Beclin-1 | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Cell Line | Small Ubiquitin-Related Modifier Proteins - metabolism | Heat-Shock Proteins - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Antioxidants - pharmacology | Mice, Inbred Strains | Nasal Polyps - drug therapy | Antioxidants - therapeutic use | Animals | Acetylcysteine - pharmacology | Adaptor Proteins, Signal Transducing - genetics | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Organometallic Compounds - pharmacology | Complications and side effects | Active oxygen | Gene mutations | Lung diseases | Physiological aspects | Cystic fibrosis | Genetic aspects | Research | Health aspects | Risk factors | Index Medicus
Journal Article
Journal of Translational Medicine, ISSN 1479-5876, 01/2013, Volume 11, Issue 1, pp. 5 - 5
Journal Article
Gut, ISSN 0017-5749, 03/2010, Volume 59, Issue 3, pp. 311 - 319
Background An unresolved question in coeliac disease is to understand how some toxic gliadin peptides, in particular p31-43, can initiate an innate response... 
PATHOGENIC T-CELLS | ACTIVATED RECEPTOR-GAMMA | ACID | PERMEABILITY | INFLAMMATION | ULCERATIVE-COLITIS | DISEASE | STORAGE DISORDERS | CROSS-LINKING | ROSIGLITAZONE | GASTROENTEROLOGY & HEPATOLOGY
Journal Article
Journal of Translational Medicine, ISSN 1479-5876, 01/2015, Volume 13, Issue 1, pp. 32 - 32
Journal Article
Journal of Immunology, ISSN 0022-1767, 2008, Volume 180, Issue 11, pp. 7697 - 7705
Cystic fibrosis (CF), the most common life-threatening inherited disease in Caucasians, is due to mutations in the CF transmembrane conductance regulator... 
BREAST-CANCER CELLS | PROTEIN | INHIBITION | AGGRESOMES | RECEPTOR-GAMMA | EPITHELIAL-CELLS | DISEASE | AIRWAY INFLAMMATION | KAPPA-B | IMMUNOLOGY | CROSS-LINKS
Journal Article
Gastroenterology, ISSN 0016-5085, 11/2005, Volume 129, Issue 5, pp. 1400 - 1413
Journal Article
Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2014, Volume 193, Issue 10, pp. 5347 - 5349
Journal Article
Drug Discovery Today: Disease Models, ISSN 1740-6757, 2004, Volume 1, Issue 4, pp. 417 - 423
Autoimmune thyroiditis is an organ-specific autoimmune disease characterized by both B-cell and T-cell responses. Despite extensive research to underpin the... 
Journal Article
Gut, ISSN 0017-5749, 03/2010, Volume 59, Issue 3, pp. 311 - 319
  Background An unresolved question in coeliac disease is to understand how some toxic gliadin peptides, in particular p31-43, can initiate an innate response... 
Celiac disease | Lysosomes | Physiological aspects | Development and progression | Natural immunity | Transglutaminases | Research | Inflammatory bowel disease | Oxidative stress | Disease | Peptides | Cytokines | Rodents | Cystic fibrosis | Inflammation | Gene expression | Small intestine
Journal Article