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by Rion, N and Ruegg, M
DEVELOPMENT, ISSN 0950-1991, 04/2019, Volume 146, Issue 7, p. dev178491
In development and during regeneration in adults, muscle fibres develop from muscle progenitor cells, and the proliferation, differentiation and fusion of... 
DEVELOPMENTAL BIOLOGY
Journal Article
Cell Metabolism, ISSN 1550-4131, 05/2012, Volume 15, Issue 5, pp. 725 - 738
Mammalian target of rapamycin complex 2 (mTORC2) phosphorylates and activates AGC kinase family members, including Akt, SGK1, and PKC, in response to... 
TRANSCRIPTION FACTORS | LIPID-METABOLISM | INSULIN-RESISTANCE | ELEMENT-BINDING PROTEIN-1C | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | MOTIF PHOSPHORYLATION | DIABETIC-NEPHROPATHY | GLUCOSE-UTILIZATION | CELL-GROWTH | PHOSPHOINOSITIDE 3-KINASE | CELL BIOLOGY | Glucose Intolerance - metabolism | Phosphorylation | Liver - enzymology | TOR Serine-Threonine Kinases - metabolism | Homeostasis | Glycogen Synthase Kinase 3 beta | Male | Hepatocytes - metabolism | Mechanistic Target of Rapamycin Complex 2 | Hyperglycemia - genetics | Proto-Oncogene Proteins c-akt - genetics | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Forkhead Transcription Factors - metabolism | Trans-Activators - genetics | Hyperinsulinism - genetics | Insulin - genetics | Lipogenesis | Proto-Oncogene Proteins c-akt - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Gluconeogenesis | Hyperinsulinism - metabolism | Glucose Intolerance - genetics | Signal Transduction | Liver - metabolism | Glucokinase - genetics | Glucose - genetics | Lipid Metabolism | Forkhead Transcription Factors - genetics | Glucokinase - metabolism | Glycogen Synthase Kinase 3 - metabolism | Mice, Knockout | Hyperglycemia - metabolism | Proteins - genetics | Insulin - metabolism | Animals | Proteins - metabolism | Trans-Activators - deficiency | Glycogen Synthase Kinase 3 - genetics | Sterol Regulatory Element Binding Protein 1 - genetics | Glucose - metabolism | Glycolysis | Trans-Activators - metabolism | Forkhead Box Protein O1 | Mice | Transcription Factors | Glucose metabolism | Hyperglycemia | Glucose | Isoenzymes | Dextrose | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 11/2004, Volume 6, Issue 11, pp. 1122 - 1128
Journal Article
Journal Article
Cell Metabolism, ISSN 1550-4131, 2008, Volume 8, Issue 5, pp. 399 - 410
Journal Article
Cell Metabolism, ISSN 1550-4131, 2008, Volume 8, Issue 5, pp. 411 - 424
Journal Article
细胞研究:英文版, ISSN 1001-0602, 2017, Volume 27, Issue 5, pp. 604 - 605
Long non-coding RNAs (lncRNAs) belong to the ever-increasing number of transcripts that are thought not to encode proteins. A recent study has now identified a... 
MUSCLE | MTORC1 | REGENERATION | CELL BIOLOGY | Regeneration | Peptides | RNA, Long Noncoding | Cytoplasm | Mechanistic Target of Rapamycin Complex 1 | Research Highlight
Journal Article
Nucleic acids research, ISSN 0305-1048, 05/2019, Volume 47, Issue 14, pp. 7618 - 7632
Spinal Muscular Atrophy results from loss-of-function mutations in SMN1 but correcting aberrant splicing of SMN2 offers hope of a cure. However, current splice... 
Index Medicus | RNA and RNA-protein complexes
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2011, Volume 121, Issue 6, pp. 2181 - 2196
Diabetic nephropathy (DN) is among the most lethal complications that occur in type 1 and type 2 diabetics. Podocyte dysfunction is postulated to be a critical... 
EPITHELIAL-MESENCHYMAL TRANSITION | KIDNEY-DISEASE | MAMMALIAN TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | TUBEROUS SCLEROSIS COMPLEX | SLIT DIAPHRAGM | GLOMERULAR PROTEIN | ANGIOTENSIN-II | NEPHRIN EXPRESSION | ENDOPLASMIC-RETICULUM STRESS | NEPHROTIC SYNDROME | Phosphorylation | Ribosomal Protein S6 Kinases - metabolism | Diabetic Nephropathies - etiology | Diabetes Mellitus, Type 2 - genetics | Multiprotein Complexes | Podocytes - enzymology | Diabetic Nephropathies - drug therapy | Endoplasmic Reticulum - metabolism | Male | Mechanistic Target of Rapamycin Complex 1 | Mice, Mutant Strains | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Glomerular Basement Membrane - pathology | Regulatory-Associated Protein of mTOR | Cell Differentiation | Membrane Proteins - metabolism | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Disease Models, Animal | Carrier Proteins - physiology | Proteins - physiology | Sirolimus - therapeutic use | Mice, Inbred C57BL | Podocytes - pathology | Mice, Knockout | Carrier Proteins - genetics | Tumor Suppressor Proteins - physiology | Adaptor Proteins, Signal Transducing | Glomerular Mesangium - pathology | Animals | Podocytes - drug effects | Podocytes - physiology | Mice | Protein Processing, Post-Translational | TOR Serine-Threonine Kinases | Enzyme Activation | Proteins - antagonists & inhibitors | Diabetic nephropathies | Index Medicus | Abridged Index Medicus
Journal Article
Skeletal Muscle, ISSN 2044-5040, 09/2012, Volume 2, Issue 1, pp. 18 - 18
Laminin-α2-deficient congenital muscular dystrophy (MDC1A) is a severe muscle-wasting disease for which no curative treatment is available. Antagonists of the... 
Smad2/3 | Fibrosis | Notexin | TGF-β | Angiotensin II | losartan | Muscle regeneration | Skeletal muscle
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1446 - 1458
Journal Article