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PloS one, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e100880 - e100880
... Liu [sup.1], David J. Figueroa [sup.1], Elisabeth A. Minthorn [sup.1], Mark A. Seefeld [sup.1], Meagan B. Rouse [sup.1], Sridhar K. Rabindran [sup.1], Dirk... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Ribosomal Protein S6 Kinases - metabolism | Pyrazoles - chemical synthesis | Antineoplastic Agents - chemical synthesis | Humans | Gene Expression Regulation, Neoplastic | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Pancreatic Neoplasms - drug therapy | PTEN Phosphohydrolase - antagonists & inhibitors | Female | Antineoplastic Agents - pharmacology | Drug Evaluation, Preclinical | MAP Kinase Kinase Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | Pyrazoles - pharmacology | PTEN Phosphohydrolase - genetics | Protein Kinase Inhibitors - chemical synthesis | Signal Transduction | Administration, Oral | MAP Kinase Kinase Kinases - genetics | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - enzymology | PTEN Phosphohydrolase - metabolism | Pancreatic Neoplasms - genetics | MAP Kinase Kinase Kinases - metabolism | Mice, SCID | Drug Synergism | Phosphatidylinositol 3-Kinases - genetics | Xenograft Model Antitumor Assays | Animals | Tumor Burden - drug effects | Cell Line, Tumor | Diamines - pharmacology | Mice | Protein Kinase Inhibitors - pharmacology | Blood Glucose - metabolism | Diamines - chemical synthesis | Ribosomal Protein S6 Kinases - antagonists & inhibitors | Ribosomal Protein S6 Kinases - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Cellular signal transduction | Glucose | Phosphotransferases | Dextrose | Tumors | Biotechnology | Phosphorylation | Animal models | Leukemia | Homeostasis | Oncology | AKT protein | Kinases | K-Ras protein | Anticancer properties | Feedback | Polyethylene glycol | Inhibition | Enzymes | Tumor cells | MAP kinase | Breast cancer | Bioavailability | Substrates | 1-Phosphatidylinositol 3-kinase | Studies | Signaling | Inhibitors | Pancreatic cancer | Mutation | Laboratory animals | ATP | PTEN protein | Cancer | Apoptosis | Index Medicus
Journal Article
The New England journal of medicine, ISSN 1533-4406, 11/2019, Volume 381, Issue 21, pp. 2020 - 2031
Patients with advanced non–small-cell lung cancer with a PD-L1 expression level of 1% or more of tumor cells were randomly assigned to receive nivolumab plus... 
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Antineoplastic Agents, Immunological - administration & dosage | Lung Neoplasms - drug therapy | Lung Neoplasms - mortality | Humans | Lung Neoplasms - metabolism | Middle Aged | Carcinoma, Non-Small-Cell Lung - metabolism | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Ipilimumab - adverse effects | Nivolumab - adverse effects | Carcinoma, Non-Small-Cell Lung - mortality | Antineoplastic Agents, Immunological - adverse effects | B7-H1 Antigen - metabolism | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Survival Analysis | Aged, 80 and over | Adult | Female | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Ipilimumab - administration & dosage | Nivolumab - administration & dosage | Lung cancer, Non-small cell | Drug therapy | Patient outcomes | Ipilimumab | Testing | Small cell lung carcinoma | Lung cancer | Non-small cell lung carcinoma | Oncology | Cancer therapies | Chemotherapy | Immunotherapy | PD-L1 protein | Biomarkers | Clinical medicine | Mutation | Drug dosages | Apoptosis | Tumors | Index Medicus | Abridged Index Medicus | B7-H1 Antigen | Carcinoma, Non-Small-Cell Lung | Antineoplastic Combined Chemotherapy Protocols | Cellular Biology | Lung Neoplasms | Life Sciences | Antineoplastic Agents, Immunological | Nivolumab
Journal Article
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 05/2006, Volume 103, Issue 20, pp. 7817 - 7822
... Sasaki § , James W. Horner*, Michael Eck*, Albert Mitchell ¶ , Yangping Sun ¶ , Ruqayyah Al-Hashem ¶ , Roderick T. Bronson ‡ , Sridhar K. Rabindran , Carolyn M... 
Polymerase chain reaction | Squamous cell carcinoma | Lungs | Reverse transcriptase polymerase chain reaction | RNA | Exons | Genomics | DNA | Genetic mutation | Tumors | Irreversible inhibitor | Lung cancer | Lung squamous cell carcinoma | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Erlotinib Hydrochloride | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Protein-Tyrosine Kinases - metabolism | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - metabolism | Receptor, Epidermal Growth Factor - metabolism | Protein Isoforms - metabolism | Quinazolines - metabolism | Cell Line | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Mice, Knockout | Quinolines - metabolism | Animals | Protein Kinase Inhibitors - therapeutic use | Quinazolines - therapeutic use | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Mice | Mutation | Tumor Suppressor Protein p14ARF - genetics | Tumor Suppressor Protein p14ARF - metabolism | Protein Isoforms - antagonists & inhibitors | Protein Isoforms - genetics | Protein Kinase Inhibitors - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Tyrosine | Research | Protein kinases | Oncology | Cell growth | Cells | Index Medicus | lung cancer | Biological Sciences | irreversible inhibitor | lung squamous cell carcinoma
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 05/2005, Volume 102, Issue 21, pp. 7665 - 7670
... ‡§ , Nadia Godin-Heymann ‡§ , Ross A. Okimoto ‡ , Brian W. Brannigan ‡ , Patricia L. Harris ‡ , David R. Driscoll ‡ , Panos Fidias †‡ , Thomas J. Lynch †‡ , Sridhar K... 
Biological Sciences | Receptors | Drug trafficking | Internalization | Liver | Cell lines | Lesions | Codons | Genetic mutation | Tumors | Resistance mechanisms | Molecular targeted therapy | Drug resistance | Tyrosine kinase inhibitor | Non-small cell lung cancer | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Erlotinib Hydrochloride | Receptor, Epidermal Growth Factor - genetics | Aniline Compounds | Humans | Lung Neoplasms - metabolism | Molecular Sequence Data | Immunoblotting | Quinolines - pharmacology | Receptor, ErbB-2 - physiology | Quinazolines - metabolism | Base Sequence | Organic Chemicals - pharmacology | Phosphorylation - drug effects | Tumor Cells, Cultured | Receptor, Epidermal Growth Factor - physiology | Lung Neoplasms - genetics | Neoplasm Recurrence, Local - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Mutation - genetics | Sequence Analysis, DNA | Drug Resistance - genetics | Signal Transduction - drug effects | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Neoplasm Recurrence, Local - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Aminoquinolines | Lung cancer | Research | Signaling | Mutation | Index Medicus | non-small cell lung cancer | molecular targeted therapy | drug resistance | tyrosine kinase inhibitor
Journal Article