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Gastroenterology, ISSN 0016-5085, 02/2015, Volume 148, Issue 2, pp. 427 - 439.e16
Journal Article
Nature Genetics, ISSN 1061-4036, 10/2013, Volume 45, Issue 10, pp. 1216 - 1220
Journal Article
Gut, ISSN 0017-5749, 02/2008, Volume 57, Issue 2, pp. 259 - 267
Acute pancreatitis is an inflammatory disease of the pancreas which, in its most severe form, is associated with multi-organ failure and death. Recently,... 
PROTEIN-KINASE-C | CYTOKINE PRODUCTION | LUNG INJURY | NECROSIS-FACTOR-ALPHA | SYSTEMIC INFLAMMATORY RESPONSE | ACINAR-CELLS | CERULEIN-INDUCED PANCREATITIS | TNF-ALPHA | B/REL ACTIVATION | GASTROENTEROLOGY & HEPATOLOGY | UBIQUITIN-PROTEASOME PATHWAY
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 14096 - 14
Elevated serum triglyceride concentration (seTG, > 1.7 mM or > 150 mg/dL) or in other words hypertriglyceridemia (HTG) is common in the populations of... 
ETIOLOGY | ACID ETHYL-ESTERS | MULTIDISCIPLINARY SCIENCES | HYPERLIPIDEMIA | RISK | NECROTIZING PANCREATITIS | ATLANTA CLASSIFICATION | HYPERTRIGLYCERIDEMIA | SEVERITY | EPIDEMIOLOGY | Systematic review | Pancreas | Hypertriglyceridemia | Mortality | Pancreatitis | Renal failure
Journal Article
Experimental Cell Research, ISSN 0014-4827, 2009, Volume 315, Issue 11, pp. 1975 - 1989
Acinar cells in pancreatitis die through apoptosis and necrosis, the roles of which are different. The severity of experimental pancreatitis correlates... 
Pancreatic acinar cell | Bcl-xL | CCK | Cytochrome c release | Caspase-3 | Mitochondrial membrane potential | APOPTOSIS | MEMBRANE PERMEABILITY TRANSITION | KAPPA-B | CANCER | CELL-DEATH | CELL BIOLOGY | ONCOLOGY | SMALL-MOLECULE INHIBITORS | X-L | ACINAR-CELLS | CASPASE ACTIVATION | TARGETING BCL-2 | Pancreatitis, Acute Necrotizing - genetics | Caspase 3 - metabolism | Sincalide - pharmacology | bcl-X Protein - genetics | Male | DNA Primers - genetics | Membrane Potential, Mitochondrial - drug effects | RNA, Messenger - metabolism | Necrosis | Proto-Oncogene Proteins c-bcl-2 - metabolism | bcl-X Protein - antagonists & inhibitors | Adenosine Triphosphate - metabolism | Base Sequence | Pancreatitis, Acute Necrotizing - metabolism | Disease Models, Animal | Pancreatitis, Acute Necrotizing - pathology | Gene Expression | Cytochromes c - metabolism | RNA, Messenger - genetics | Ceruletide - toxicity | Pancreas - drug effects | Pancreas - pathology | Rats | Mitochondria - metabolism | Mitochondria - drug effects | Pancreas - metabolism | Rats, Sprague-Dawley | Animals | Capsid Proteins | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Mice | In Vitro Techniques | bcl-X Protein - metabolism | Cytochrome c | Proteins | Analysis | Membrane potentials | Pancreatitis | Mitochondrial DNA | Cholecystokinin | Permeability | Mitochondria | Cellular biology | Gangrene | Pancreas | Medical disorders | Apoptosis | Index Medicus
Journal Article
International Journal of Inflammation, ISSN 2090-8040, 2012, Volume 2012, pp. 360685 - 10
Acute pancreatitis (AP) is a common emergency condition. In the majority of cases, it presents in a mild and self-limited form. However, about 20% of patients... 
Review
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, pp. e0165244 - e0165244
Journal Article
Inflammatory Bowel Diseases, ISSN 1078-0998, 04/2011, Volume 17, Issue 4, pp. 884 - 898
Journal Article