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2003, ISBN 9780612845282
Hsp73, 72 and 90 expression was investigated in the western painted turtle following bacterial infection, forced dive and reoxygenation. Following infection,... 
Zoology | Animals | Anatomy & physiology
Dissertation
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2012, Volume 109, Issue 3, pp. 965 - 970
Microglial priming predisposes the brain to neurodegeneration and affects disease progression. The signal to switch from the quiescent to the primed state is... 
Spinal cord | Nervous system diseases | Cytokines | Central nervous system | Central nervous system diseases | Mice | Infections | Alzheimers disease | White matter | Microglia | SYSTEMIC INFLAMMATION | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | ACTIVATION | MESSENGER-RNA | COMPLEMENT REGULATORY PROTEIN | MULTIDISCIPLINARY SCIENCES | DISEASE | CRRY | FACTOR-H | AGED MICE | Inflammation - pathology | Up-Regulation | Humans | Lipopolysaccharides - administration & dosage | Receptors, Complement - deficiency | Encephalomyelitis, Autoimmune, Experimental - immunology | Receptors, Complement - metabolism | Microglia - immunology | Spinal Cord - pathology | Cell Shape | Inflammation Mediators - metabolism | Microglia - pathology | Encephalomyelitis, Autoimmune, Experimental - pathology | Mice, Inbred C57BL | Complement C3 - immunology | Spinal Cord - immunology | Models, Immunological | Animals | Cross-Priming - immunology | Complement Pathway, Alternative - immunology | Lipopolysaccharides - pharmacology | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Complement C3b - immunology | Development and progression | Multiple sclerosis | Proteins | Brain | Signal transduction | Rodents | Complement receptors | Animal models | Complement component C3b | Deposits | Crry protein | Neurodegenerative diseases | Inflammation | Substantia alba | Alternative pathway | Experimental allergic encephalomyelitis | Lipopolysaccharides | Neurological diseases | Complement component C3 | Index Medicus | Biological Sciences
Journal Article
Immunity, ISSN 1074-7613, 12/2015, Volume 43, Issue 6, pp. 1160 - 1173
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2017, Volume 12, Issue 5, pp. e0177815 - e0177815
Mycobacterium leprae (M. leprae) infection causes nerve damage and the condition worsens often during and long after treatment. Clearance of bacterial antigens... 
PERIPHERAL-NERVE | SYSTEM | MEMBRANE ATTACK COMPLEX | MULTIDISCIPLINARY SCIENCES | WALLERIAN DEGENERATION | REACTIONAL STATES | NECROSIS-FACTOR-ALPHA | 2 PARTS | REVERSAL REACTIONS | IMMUNOLOGY | EXPRESSION | Immunohistochemistry | Granuloma - metabolism | Complement C3d - immunology | Complement Membrane Attack Complex - metabolism | Leprosy - microbiology | Humans | Middle Aged | Male | Complement Membrane Attack Complex - immunology | Skin Diseases - pathology | Complement Activation - immunology | Lipopolysaccharides | Young Adult | T-Lymphocytes - metabolism | Adult | Female | Child | Leprosy - pathology | Skin Diseases - immunology | Leprosy - immunology | Bacterial Load | Granuloma - pathology | Biopsy | Adolescent | Biomarkers | Granuloma - immunology | T-Lymphocytes - immunology | Leprosy | Immune response | Mycobacterium leprae | Research | T cells | Peripheral nerves | Immune clearance | Activation | Complement | Lymphocytes T | Genomes | Infections | Immunity | Cell activation | Immunology | Nerves | Gangrene | Skin diseases | Bacteria | Membrane attack complex | Tumor necrosis factor-TNF | Lesions | Damage | Erythema nodosum | Erythema nodosum leprosum | Deposition | Activation analysis | Antigens | Patients | Studies | Pathology | Axons | Complement activation | Skin | Immunoreactivity | Erythema | Index Medicus
Journal Article
Journal Article
Journal Article