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Lancet neurology, ISSN 1474-4422, 2015, Volume 14, Issue 4, pp. 388 - 405
Summary Increasing evidence suggests that Alzheimer's disease pathogenesis is not restricted to the neuronal compartment, but includes strong interactions with... 
Neurology | CEREBRAL AMYLOID ANGIOPATHY | POSITRON-EMISSION-TOMOGRAPHY | TRAUMATIC BRAIN-INJURY | NITRIC-OXIDE SYNTHASE | ANTIINFLAMMATORY PREVENTION TRIAL | CENTRAL-NERVOUS-SYSTEM | RANDOMIZED CONTROLLED-TRIAL | MILD COGNITIVE IMPAIRMENT | TRANSGENIC MOUSE MODEL | CLINICAL NEUROLOGY | GENOME-WIDE ASSOCIATION | Humans | Astrocytes - pathology | Brain Injuries - complications | Brain Injuries - metabolism | Alzheimer Disease - pathology | Inflammation - metabolism | Microglia - immunology | Astrocytes - immunology | Alzheimer Disease - prevention & control | Inflammation Mediators - metabolism | Microglia - pathology | Inflammation - diagnosis | Alzheimer Disease - immunology | Disease Models, Animal | Inflammation Mediators - immunology | Severity of Illness Index | Immunization | Obesity - complications | Locus Coeruleus - pathology | Risk Factors | Clinical Trials as Topic | Inflammation - immunology | Biomarkers - blood | Immunity, Innate | Disease Progression | Protein Folding | Obesity - metabolism | Animals | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Nootropic Agents - administration & dosage | Alzheimer Disease - metabolism | Biomarkers - cerebrospinal fluid | Alzheimer Disease - genetics | Phagocytosis | Alzheimer's disease | Analysis | Genotype & phenotype | Neurosciences | Brain research | Pathogenesis | Cognitive ability | Ligands | Physiology | Mutation | Alzheimers disease | Gene expression | Immune system
Journal Article