X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (9) 9
index medicus (8) 8
humans (6) 6
apoptosis (5) 5
mice (5) 5
rats (5) 5
mitochondria (4) 4
mitochondrial dna (4) 4
bcl-2-associated x protein - metabolism (3) 3
cell death (3) 3
cell line, tumor (3) 3
cells, cultured (3) 3
male (3) 3
mitochondria - metabolism (3) 3
permeability (3) 3
aging (2) 2
analgesics, opioid - pharmacology (2) 2
apoptosis - drug effects (2) 2
atherosclerosis (2) 2
biochemistry (2) 2
biochemistry & molecular biology (2) 2
calcium - metabolism (2) 2
cell biology (2) 2
cell cycle control (2) 2
cell-death (2) 2
central nervous system depressants (2) 2
cercopithecus aethiops (2) 2
cos cells (2) 2
cytochromes c - metabolism (2) 2
dose-response relationship, drug (2) 2
female (2) 2
history (2) 2
in vitro techniques (2) 2
induced apoptosis (2) 2
life sciences (2) 2
methadone (2) 2
methadone - pharmacology (2) 2
methadone hydrochloride (2) 2
mice, transgenic (2) 2
mitochondria - drug effects (2) 2
multidisciplinary sciences (2) 2
necrosis (2) 2
necrosis - chemically induced (2) 2
neurons - drug effects (2) 2
neurons - metabolism (2) 2
nitric-oxide (2) 2
oxidative stress (2) 2
p27 (2) 2
p38 mitogen-activated protein kinases - metabolism (2) 2
pharmacology & pharmacy (2) 2
protein phosphorylation (2) 2
protein transport - drug effects (2) 2
proteins (2) 2
sh-sy5y cells (2) 2
transgenic animal models (2) 2
acetylcholine (1) 1
acetylcholine - pharmacology (1) 1
adaptor proteins, signal transducing - genetics (1) 1
adaptor proteins, signal transducing - metabolism (1) 1
aequorin (1) 1
age (1) 1
aging - metabolism (1) 1
amyotrophic lateral sclerosis (1) 1
amyotrophic lateral sclerosis - metabolism (1) 1
amyotrophic-lateral-sclerosis (1) 1
animals, newborn (1) 1
anti-oxidant (1) 1
aorta - metabolism (1) 1
aorta - pathology (1) 1
aorta, thoracic - drug effects (1) 1
aorta, thoracic - enzymology (1) 1
aorta, thoracic - physiopathology (1) 1
apolipoproteins e - genetics (1) 1
apoptosi (1) 1
apoptosis inducing factor - analysis (1) 1
apoptosis inducing factor - metabolism (1) 1
architecture (1) 1
architecture guidebooks. history 20th century spainbarcelona (1) 1
arquitectura (1) 1
arquitectura moderna (1) 1
article (1) 1
article subject (1) 1
articles (1) 1
aspartate (1) 1
atherosclerosis - metabolism (1) 1
atherosclerosis - pathology (1) 1
atp (1) 1
avena - genetics (1) 1
avena - metabolism (1) 1
b-cll (1) 1
barcelona (1) 1
barcelona buildings, structures (1) 1
barcelona guidebooks. buildings, structures, etc (1) 1
bax (1) 1
bcl-2 (1) 1
bcl-2 family-members (1) 1
bcl-2 homologous antagonist-killer protein - metabolism (1) 1
bcl-2 homologous antagonist-killer protein - physiology (1) 1
bcl-2-associated x protein - physiology (1) 1
bcl-x protein - metabolism (1) 1
more...
Library Location Library Location
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 03/2013, Volume 110, Issue 12, p. E1102
  Oxidative stress is a widely recognized cause of cell death associated with neurodegeneration, inflammation, and aging. Tyrosine nitration in these... 
Proteins | Oxidative stress | Neurons | Aging | Amyotrophic lateral sclerosis | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2013, Volume 110, Issue 12, pp. 4449 - 4450
Journal Article
The Journal of Neuroscience, ISSN 0270-6474, 2015, Volume 35, Issue 19, pp. 7349 - 7364
The protein NOS1AP/CAPON mediates signaling from a protein complex of NMDA receptor, PSD95 and nNOS. The only stroke trial for neuroprotectants that showed... 
Excitotoxic lesion | NMDA receptor | Protein interaction | nNOS/NOS1 | Target identification | NOS1AP/CAPON | protein interaction | THERMAL HYPERALGESIA | NMDA RECEPTORS | INDUCED APOPTOSIS | NITRIC-OXIDE SYNTHASE | PDZ DOMAIN | target identification | PSD-95/NNOS INTERFACE | NEUROSCIENCES | CELL-DEATH | SLICE CULTURES | BETA-FINGER | excitotoxic lesion | SMALL-MOLECULE INHIBITORS | Humans | Cercopithecus aethiops | N-Methylaspartate - pharmacology | Cell Death - genetics | Guanine Nucleotide Exchange Factors - metabolism | Tumor Suppressor Proteins - genetics | Membrane Proteins - metabolism | Neurons - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Cell Death - drug effects | Neurons - drug effects | T-Lymphoma Invasion and Metastasis-inducing Protein 1 | Organ Culture Techniques | Animals, Newborn | Protein Structure, Tertiary | Recombinant Proteins - metabolism | Guanine Nucleotide Exchange Factors - genetics | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Models, Molecular | Rats | Excitatory Amino Acid Agonists - pharmacology | Recombinant Proteins - genetics | Mutation - genetics | Animals | Nitric Oxide Synthase Type I - metabolism | Adaptor Proteins, Signal Transducing - genetics | Signal Transduction - physiology | Nitric Oxide Synthase Type I - genetics | Adaptor Proteins, Signal Transducing - metabolism | In Vitro Techniques | COS Cells | nNOS | NOS1AP | NOS1 | CAPON
Journal Article
Biogerontology, ISSN 1389-5729, 12/2007, Volume 8, Issue 6, pp. 663 - 672
We studied vascular function in quiescent aortas from senescence-accelerated resistant (SAM-R1) and prone (SAM-P8) mice. Myographical studies of thoracic aorta... 
Life Sciences | Geriatrics/Gerontology | Vessels | Aging | Lipid peroxidation | Developmental Biology | Vascular function | SAM | Cell Biology | Endothelium | vascular function | endothelium | ACETYLCHOLINE | CARDIOVASCULAR-SYSTEM | THORACIC AORTA | RELAXATION | MECHANISMS | lipid peroxidation | GERIATRICS & GERONTOLOGY | PATHWAY | NITRIC-OXIDE | MICE | aging | HYPERTENSION | vessels | AGE | Nitric Oxide Synthase - antagonists & inhibitors | Endothelium, Vascular - drug effects | Male | Endothelium, Vascular - enzymology | Dose-Response Relationship, Drug | Phenylephrine - pharmacology | Potassium Chloride - pharmacology | Models, Animal | Aorta, Thoracic - drug effects | Malondialdehyde - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | Aorta, Thoracic - physiopathology | Mice, Inbred Strains | Vasoconstriction - drug effects | Indomethacin - pharmacology | Cyclooxygenase Inhibitors - pharmacology | Aorta, Thoracic - enzymology | Animals | Prostaglandin-Endoperoxide Synthases - metabolism | Mice | Nitric Oxide Synthase - metabolism | Vasodilation - drug effects | In Vitro Techniques | Lipid Peroxidation | Nitric Oxide - metabolism | Aging - metabolism | Rodents | Muscular system
Journal Article
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 03/2006, Volume 97, Issue 4, pp. 735 - 743
Excessive cellular proliferation is thought to contribute to neointimal lesion development during atherosclerosis and restenosis after angioplasty. Inhibition... 
atherosclerosis | transgenic animal models | protein phosphorylation | cell cycle control | p27 | Protein phosphorylation | Transgenic animal models | Atherosclerosis | Cell cycle control
Journal Article
Seminars in Arthritis and Rheumatism, ISSN 0049-0172, 08/2019, Volume 49, Issue 1, pp. 126 - 135
Tocilizumab (TCZ) has shown efficacy in clinical trials on giant cell arteritis (GCA). Real-world data are scarce. Our objective was to assess efficacy and... 
Giant cell arteritis | Biological therapy | Large-vessel vasculitis | Tocilizumab | REFRACTORY UVEITIS | EFFICACY | POLYMYALGIA-RHEUMATICA | CLASSIFICATION | BEHCETS-DISEASE | INDUCTION | RHEUMATOLOGY | MAINTENANCE | THERAPY | RECEPTOR TOCILIZUMAB | REMISSION | Medicine, Experimental | Medical research
Journal Article