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Immunity, ISSN 1074-7613, 03/2018, Volume 48, Issue 3, pp. 514 - 529.e6
Microglia as tissue macrophages contribute to the defense and maintenance of central nervous system (CNS) homeostasis. Little is known about the epigenetic... 
HDACs | Alzheimer’s disease | microglia | epigenetics | Alzheimer's disease | HDAC2 | VARIANTS | MACROPHAGES | MONOCYTES | CNS | CENTRAL-NERVOUS-SYSTEM | IMMUNOLOGY | FATE | CELL | PRECURSORS | REVEALS | Microglia - metabolism | Cell Proliferation | Memory Disorders - genetics | Neurodegenerative Diseases - psychology | Epigenesis, Genetic | Transcriptome | Homeostasis | Memory Disorders - metabolism | Gene Expression Profiling | Neurogenesis - genetics | Microglia - immunology | Spatial Learning | Histone Deacetylase 1 - genetics | Disease Models, Animal | Histone Deacetylase 2 - genetics | Gene Expression | Neurodegenerative Diseases - pathology | Plaque, Amyloid - pathology | Gene Expression Regulation | Phagocytosis - immunology | Neurogenesis - immunology | Mice, Transgenic | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - metabolism | Mice, Knockout | Plaque, Amyloid - genetics | Animals | Plaque, Amyloid - metabolism | Mice | Histone Deacetylase 2 - metabolism | Histones - metabolism | Apoptosis | Histone Deacetylase 1 - metabolism | Brain | Histone deacetylase | Enzymes | Genomics | Cognitive ability | Central nervous system | Macrophages | Gene expression | Survival | Ablation | Microglia | Clonal deletion | Mutagenesis | Neurodegeneration | Cell cycle | DNA methylation | Epigenetics | Deletion | HDAC2 protein | Chemokines | Phagocytosis
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 08/2018, Volume 20, Issue 1, pp. 187 - 18
Background: In systemic sclerosis (SSc), autoantibodies provide the most accurate tool to predict the disease subset and pattern of organ involvement.... 
Autoantibodies | Immune complexes | Systemic sclerosis | Fibrosis | Fibroblasts | Toll-like receptors | Inflammation | TOPOISOMERASE-I AUTOANTIBODIES | ANTIBODIES | ACTIVATION | SJOGRENS-SYNDROME | AMERICAN-COLLEGE | LUPUS-ERYTHEMATOSUS | RHEUMATOLOGY | ANTIPHOSPHOLIPID SYNDROME | GROWTH-FACTOR RECEPTOR | PROGRESSIVE SYSTEMIC-SCLEROSIS | CLASSIFICATION CRITERIA | Skin - metabolism | Humans | Scleroderma, Systemic - pathology | Collagen Type I - genetics | Fibrosis - immunology | Gene Expression - immunology | Toll-Like Receptors - metabolism | Cytokines - genetics | Skin - pathology | Cytokines - immunology | Fibroblasts - metabolism | Skin - immunology | Collagen Type I - metabolism | Cytokines - metabolism | Intercellular Adhesion Molecule-1 - immunology | Toll-Like Receptors - immunology | Scleroderma, Systemic - genetics | Inflammation - immunology | Scleroderma, Systemic - immunology | Fibroblasts - pathology | Collagen Type I - immunology | Autoantibodies - immunology | Intercellular Adhesion Molecule-1 - metabolism | Phenotype | Intercellular Adhesion Molecule-1 - genetics | Toll-Like Receptors - genetics | Inflammation - genetics | Fibroblasts - immunology | Antigen-Antibody Complex - immunology | Care and treatment | Immune response | Systemic scleroderma | Scleroderma (Disease) | Development and progression | Genetic aspects | Health aspects | Lupus | Antigens | Immunoglobulins | Disease | Cytokines | Pathogenesis | Arthritis | RNA polymerase | Proteins | Autoimmune diseases | Scleroderma | Growth factors | Deoxyribonucleic acid--DNA | Cancer
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 10/2007, Volume 66, Issue 10, pp. 1327 - 1333
Journal Article
Haematologica, ISSN 0390-6078, 03/2019, Volume 104, Issue 4, pp. 819 - 826
Clinical studies have reported different diagnostic/predictive values of antibodies to domain 1 or 4/5 of β glycoproteinI in terms of risk of thrombosis and... 
Journal Article
Journal Article
JOURNAL OF AUTOIMMUNITY, ISSN 0896-8411, 12/2014, Volume 55, pp. 42 - 50
The thrombogenic effect of beta 2-glycoprotein I (beta 2GPI)-dependent anti-phospholipid antibodies (aPL) in animal models was found to be LPS dependent. Since... 
Anti-beta 2-glycoprotein I antibodies | ANTIBODIES | beta 2-Glycoprotein I | VIVO | ANTIPHOSPHOLIPID-SYNDROME | Toll-like receptor 4 | TOLL-LIKE RECEPTOR | Innate immunity | IMMUNOLOGY | Endothelial cells | LPS | PATHOGENESIS | BETA-GLYCOPROTEIN I | CELL ACTIVATION | PATHWAY
Journal Article
Blood, ISSN 0006-4971, 05/2003, Volume 101, Issue 9, pp. 3495 - 3500
Antiphospholipid syndrome (APS) Is an autoimmune disease characterized by the persistent presence of antiphospholipid and recurrent thrombosis or fetal. loss.... 
BACTERIAL LIPOPOLYSACCHARIDE | IN-VITRO | DENDRITIC CELLS | TOLL-LIKE RECEPTORS | FACTOR-KAPPA-B | ADHESION MOLECULES | ANTI-BETA-GLYCOPROTEIN-I ANTIBODIES | HEMATOLOGY | EXPRESSION | MONONUCLEAR PHAGOCYTES | CELL ANTIBODIES | Protein Kinases - metabolism | Phosphorylation | Receptors, Immunologic - physiology | Humans | Antibodies, Antiphospholipid - immunology | Recombinant Fusion Proteins - physiology | Interleukin-1 Receptor-Associated Kinases | NF-kappa B - metabolism | Antiphospholipid Syndrome - complications | TNF Receptor-Associated Factor 2 | Thrombophilia - etiology | Thrombophilia - immunology | Transfection | E-Selectin - genetics | Gene Deletion | E-Selectin - biosynthesis | Adult | Female | Antibodies, Monoclonal - immunology | Genes, Reporter | Endothelium, Vascular - immunology | Proteins - physiology | beta 2-Glycoprotein I | Antibodies, Monoclonal - pharmacology | Myeloid Differentiation Factor 88 | Antigens, Differentiation - physiology | Antiphospholipid Syndrome - immunology | Gene Expression Regulation - drug effects | Proteins - genetics | Adaptor Proteins, Signal Transducing | Glycoproteins - immunology | TNF Receptor-Associated Factor 6 | Lipopolysaccharides - pharmacology | Signal Transduction - physiology | Antigens, Differentiation - genetics | Protein Processing, Post-Translational | Receptors, Immunologic - genetics | Antibodies, Antiphospholipid - pharmacology | Cell Line, Transformed
Journal Article
The Open Rheumatology Journal, ISSN 1874-3129, 12/2018, Volume 12, Issue Suppl-1, M5, pp. 248 - 260
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 10/2013, Volume 15, Issue 5, pp. R151 - R151
Introduction: T helper (Th)-17 cells are increased in systemic sclerosis (SSc). We therefore assessed whether Th17 cells could modulate the inflammatory and... 
RHEUMATOID-ARTHRITIS | MATRIX METALLOPROTEINASES | SYNOVIAL FIBROBLASTS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | GENE-EXPRESSION | SKIN FIBROSIS | NECROSIS-FACTOR-ALPHA | RHEUMATOLOGY | NF-KAPPA-B | T-CELLS | PULMONARY-FIBROSIS | Interleukin-8 - genetics | Gene Expression - drug effects | Skin - metabolism | Humans | Scleroderma, Systemic - pathology | Culture Media, Conditioned - pharmacology | Male | Interleukin-17 - pharmacology | Dose-Response Relationship, Drug | Collagen Type I - genetics | Th17 Cells - metabolism | Radioimmunoassay | Inflammation Mediators - metabolism | Female | Chemokine CCL2 - metabolism | Interleukin-8 - metabolism | Matrix Metalloproteinase 1 - genetics | Skin - pathology | Fibroblasts - metabolism | Collagen Type I - metabolism | Enzyme-Linked Immunosorbent Assay | Scleroderma, Systemic - metabolism | Cells, Cultured | Scleroderma, Systemic - genetics | Chemokine CCL2 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Tumor Necrosis Factor-alpha - pharmacology | Fibroblasts - drug effects | Matrix Metalloproteinase 1 - metabolism | Interferon-gamma - pharmacology | Culture Media, Conditioned - metabolism | Proteins | Medical equipment and supplies industry | Interleukins | Systemic scleroderma | Medical test kit industry | Scleroderma (Disease) | High-definition television | Skin | Biological response modifiers | Enzyme-linked immunosorbent assay
Journal Article