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Journal Article
PLoS ONE, ISSN 1932-6203, 05/2016, Volume 11, Issue 5, p. e0155944
Duchenne muscular dystrophy (DMD) is an X-linked muscle disease caused by mutations in the dystrophin gene. The pathology of DMD manifests in patients with... 
RHEUMATOID-ARTHRITIS | CONTROLLED-TRIAL | INTERLEUKIN-1 | INHIBITION | LONG-TERM BENEFITS | REDUCES SKELETAL | MULTIDISCIPLINARY SCIENCES | SKELETAL-MUSCLE DEGENERATION | DUCHENNE MUSCULAR-DYSTROPHY | TNF-ALPHA | ANXIETY-LIKE BEHAVIOR | Interleukin 1 Receptor Antagonist Protein - administration & dosage | Muscular Dystrophy, Duchenne - drug therapy | Humans | Motor Activity - drug effects | Dose-Response Relationship, Drug | Forelimb - physiopathology | Animals | Signal Transduction - drug effects | Interleukin-1beta - metabolism | Muscular Dystrophy, Duchenne - physiopathology | Injections, Subcutaneous | Muscular Dystrophy, Duchenne - immunology | Mice, Inbred mdx | Female | Mice | Interleukin 1 Receptor Antagonist Protein - pharmacology | Forelimb - drug effects | Disease Models, Animal | Locomotion - drug effects | Disease | Interleukin | Stimulation | Arthritis | Muscular dystrophy | Lipopolysaccharides | Proteins | Signal transduction | Splenocytes | Duchenne's muscular dystrophy | Interleukin 1 | Toll-like receptors | Tumor necrosis factor-TNF | Mathematical models | Inhibition | Grip strength | Dystrophin | Age | Recombinant | Phenotypes | Parameters | Cytokines | Coordination | Interleukin 1 receptor antagonist | Medical treatment | Muscles | Blocking | Inflammation | Gene expression | Disease control | Skeletal muscle | Medicine | Pathology | Signaling | Inhibitors | Anxieties | Correlation analysis | Rheumatoid arthritis | Dystrophy | Diabetes | Mutation | Laboratory animals
Journal Article
Journal Article
The Journal of Pathology, ISSN 0022-3417, 10/2013, Volume 231, Issue 2, pp. 199 - 209
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 9, p. e12981
The dysferlin deficient SJL/J mouse strain is commonly used to study dysferlin deficient myopathies. Therefore, we systematically evaluated behavior in... 
ENCEPHALOMYELITIS | CARDIOMYOCYTE APOPTOSIS | BIOLOGY | RHO-KINASE INHIBITOR | GIRDLE MUSCULAR-DYSTROPHY | SKELETAL-MUSCLES | KAPPA-B | MODEL | MYOPATHY | EXPRESSION | T-CELLS | Membrane Proteins - genetics | Humans | Mice, Inbred C57BL | Muscular Dystrophies, Limb-Girdle - physiopathology | Dysferlin | Muscular Dystrophies, Limb-Girdle - drug therapy | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Male | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - administration & dosage | Mice, Knockout | Membrane Proteins - deficiency | Protein Kinase Inhibitors - administration & dosage | Animals | Muscle Contraction | Muscular Dystrophies, Limb-Girdle - metabolism | Muscular Dystrophies, Limb-Girdle - genetics | Mice | Drug Evaluation, Preclinical | Disease Models, Animal | Medical research | Creatine kinase | Body weight | Medicine, Experimental | Comparative analysis | Creatine | Health sciences | Disease | Standardization | Clinical trials | Biology | Soleus muscle | Kinases | Macrophages | Muscular dystrophy | Proteins | Ischemia | Lymphocytes | Rodents | Degeneration | Grip strength | Open-field behavior | Cytokines | Cardiomyocytes | Pharmacology | Inflammation | Rho-associated kinase | Children & youth | Inflammatory bowel disease | Medicine | Regeneration | Signaling | Monocytes | Weight reduction | Apoptosis | Myopathy
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e65970
Background: Current treatments for idiopathic inflammatory myopathies (collectively called myositis) focus on the suppression of an autoimmune inflammatory... 
CAPACITY | MYOADENYLATE DEAMINASE DEFICIENCY | METABOLISM | MULTIDISCIPLINARY SCIENCES | ENDOPLASMIC-RETICULUM STRESS | AUTOIMMUNE MYOSITIS | UP-REGULATION | HUMAN SKELETAL-MUSCLE | INTENSE EXERCISE | DAMAGE | Ribose - metabolism | Adenosine Monophosphate - metabolism | Body Weight - drug effects | Myositis - genetics | Male | Mice, Transgenic | Muscle, Skeletal - metabolism | Histocompatibility Antigens Class I - genetics | Ribose - pharmacology | Animals | Metabolic Networks and Pathways | Muscle Contraction - drug effects | Muscle, Skeletal - drug effects | AMP Deaminase - metabolism | Behavior, Animal - drug effects | Female | Ribose - administration & dosage | Mice | Muscle, Skeletal - pathology | Dietary Supplements | Myositis - drug therapy | Disease Models, Animal | Enzymes | Genetic disorders | Body weight | Muscles | Inflammation | Batteries | Gene expression | Monosaccharides | Metabolites | Analysis | Immunotherapy | Myositis | Genetic engineering | Adenylic acid | Health aspects | Sugars | Disease | Inflammatory response | Arthritis | Transgenic animals | Ribose | Rodents | Animal tissues | Grip strength | Supplementation | Stress response | Age | Statistical analysis | Adenosine monophosphate | AMP | Abnormalities | Dietary supplements | Transgenic mice | Adenosine deaminase | Myalgia | Metabolism | Skeletal muscle | Medicine | Musculoskeletal system | Immunosuppression | Major histocompatibility complex | D-Ribose | Ribokinase | Muscle function | Endoplasmic reticulum
Journal Article
Journal of inherited metabolic disease, ISSN 0141-8955, 06/2019
Abundance of urea cycle enzymes in the liver is regulated by dietary protein intake. Although urea cycle enzyme levels rise in response to a high-protein (HP)... 
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 04/2012, Volume 14, Issue 2, pp. 209 - 209
Journal Article