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Science, ISSN 0036-8075, 12/2015, Volume 350, Issue 6266, pp. 1317 - 1318
Journal Article
Oncotarget, ISSN 1949-2553, 05/2016, Volume 7, Issue 22, pp. 32172 - 32183
Journal Article
Cancer Cell, ISSN 1535-6108, 06/2018, Volume 33, Issue 6, pp. 949 - 951
To avoid reactive oxygen species (ROS)-induced cell death, cancer cells increase their antioxidant defense system. In this issue of , Takahashi et al. identify... 
TRPA1 | CHANNEL | ONCOLOGY | CELL BIOLOGY | Antioxidants | Cell death | Cancer
Journal Article
Science, ISSN 0036-8075, 10/2011, Volume 334, Issue 6055, pp. 525 - 528
Germline mutations of the breast cancer 1 (BRCA1) gene are a major cause of familial breast and ovarian cancer. The BRCA1 protein displays E3 ubiquitin ligase... 
Research fellowships | Proteins | Kaplan Meier estimator | Phosphoproteins | DNA damage | REPORTS | Breast cancer | Mice | Genetic mutation | Tumors | Cancer | BREAST-CANCER | BACH1 PHOSPHOPEPTIDE | COMPLEX | STRUCTURAL BASIS | MULTIDISCIPLINARY SCIENCES | MUTATION | PHOSPHOPEPTIDE RECOGNITION | BRCA1/BARD1 HETERODIMER | MICE | IDENTIFICATION | TUMORIGENESIS | Embryonic Stem Cells - metabolism | Basic-Leucine Zipper Transcription Factors - metabolism | Pancreatic Neoplasms - metabolism | Protein Multimerization | Mammary Neoplasms, Experimental - metabolism | Phosphoproteins - metabolism | RING Finger Domains | Mammary Neoplasms, Experimental - genetics | BRCA1 Protein - metabolism | Tumor Suppressor Proteins - chemistry | Protein Interaction Domains and Motifs | Genes, BRCA1 | BRCA1 Protein - chemistry | Disease Models, Animal | Tumor Suppressor Proteins - metabolism | Cells, Cultured | Mutant Proteins - genetics | Ubiquitin-Protein Ligases - metabolism | Mutant Proteins - metabolism | Pancreatic Neoplasms - genetics | Basic-Leucine Zipper Transcription Factors - genetics | Ubiquitin-Protein Ligases - chemistry | Animals | Mutant Proteins - chemistry | Protein Binding | Ligands | Ligases | Physiological aspects | Tumor suppressor genes | Genetic aspects | Research | Health aspects | Protein binding | Biochemistry | Mutation | Binding sites
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 6, pp. 1027 - 1036
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 01/2013, Volume 210, Issue 1, pp. 115 - 123
Journal Article
Cell Reports, ISSN 2211-1247, 04/2018, Volume 23, Issue 1, pp. 58 - 67
A hallmark of advanced prostate cancer (PC) is the concomitant loss of PTEN and p53 function. To selectively eliminate such cells, we screened cytotoxic... 
prostate cancer | ATP synthase | glucose | deguelin | mitochondria | RapidCaP | complex I | metabolism | Pten | ATP | MORTALITY | ROTENONE | ACTIVATION | THERAPY | OXIDOREDUCTASE | METFORMIN USE | SUPPRESSION | METASTATIC PROSTATE-CANCER | GENOME | DEGUELIN | CELL BIOLOGY
Journal Article
01/2012, ISBN 9781267270696
Familial cases of breast and ovarian cancer are often attributed to germline mutations of the BRCA1 tumor suppressor gene. Although the mechanisms of BRCA1... 
Pathology | Genetics | Oncology
Dissertation
Annual Review of Cancer Biology, ISSN 2472-3428, 3/2017, Volume 1, Issue 1, pp. 79 - 98
Reactive oxygen species (ROS), now appreciated for their cellular signaling capabilities, have a dual role in cancer. On the one hand, ROS can promote... 
cancer | signaling | antioxidants | therapy | oxidative stress | ROS | STEM-CELLS | CYSTEINE-SULFINIC ACID | CELL LUNG-CANCER | SIGNAL-TRANSDUCTION | BREAST-CANCER | ONCOLOGY | REDOX REGULATION | TUMOR-SUPPRESSOR | HYDROGEN-PEROXIDE | SUPEROXIDE-DISMUTASE
Journal Article