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Genes and Cancer, ISSN 1947-6019, 09/2017, Volume 8, Issue 9-10, pp. 682 - 694
Jun N-terminal kinases or JNKs have been shown to be involved in a wide array of signaling events underlying tumorigenesis and tumor progression. Through its... 
Ferroptosis | Cell death | Necroptosis | JNK | Apoptosis | apoptosis | ferroptosis | Review | cell death | necroptosis
Journal Article
Cell, ISSN 0092-8674, 04/2016, Volume 165, Issue 3, pp. 643 - 655
Oncogenic activation of RAS genes via point mutations occurs in 20%–30% of human cancers. The development of effective RAS inhibitors has been challenging,... 
rigosertib | RAS-binding domain | PI3K | MAPK | RAS | RAF | POINT MUTATIONS | BLADDER-CARCINOMA ONCOGENE | MUTANT | SELECTIVE INHIBITOR | ACTIVATION | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | KINASE | BINDING | TRANSFORMING PROPERTIES | CELL BIOLOGY | Glycine - analogs & derivatives | Phosphorylation | Humans | Molecular Sequence Data | ras Proteins - metabolism | Crystallography, X-Ray | Proto-Oncogene Proteins - chemistry | Sulfones - pharmacology | MAP Kinase Signaling System | Cell Cycle Proteins - chemistry | Pancreatic Neoplasms - drug therapy | Glycine - chemistry | Sulfones - chemistry | Nuclear Magnetic Resonance, Biomolecular | Dimerization | Glycine - administration & dosage | Protein-Serine-Threonine Kinases - metabolism | Proto-Oncogene Proteins B-raf - metabolism | Proto-Oncogene Proteins B-raf - chemistry | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | RNA-Binding Proteins - chemistry | Cell Cycle Proteins - metabolism | Models, Molecular | Sequence Alignment | Animals | Glycine - pharmacology | Signal Transduction - drug effects | Mice, Nude | Mice | Protein-Serine-Threonine Kinases - chemistry | Cell Transformation, Neoplastic - drug effects | RNA-Binding Proteins - metabolism | Sulfones - administration & dosage | Proteins | Medical colleges | Gene mutations | Protein binding | Jewish schools | Phosphotransferases
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2009, Volume 106, Issue 51, pp. 21689 - 21694
Journal Article
Nature Medicine, ISSN 1078-8956, 12/2011, Volume 17, Issue 12, pp. 1619 - 1626
Journal Article
Genes and Cancer, ISSN 1947-6019, 2010, Volume 1, Issue 10, pp. 979 - 993
Hematopoiesis is the cumulative result of intricately regulated signaling pathways that are mediated by cytokines and their receptors. Studies conducted over... 
STAT | JAK2 inhibitors | JAK | Myeloproliferative disorders | Cytokine receptor | cytokine receptor | Review | myeloproliferative disorders
Journal Article
Genes and Cancer, ISSN 1947-6019, 2012, Volume 3, Issue 11-12, pp. 658 - 669
The cell cycle is regulated in part by cyclins and their associated serine/threonine cyclin-dependent kinases, or CDKs. CDK4, in conjunction with the D-type... 
CDK4 | Targeted therapy | Knockout | Cell cycle | Cancer | knockout | targeted therapy | cancer | cell cycle | Monographs
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e58355
Journal Article
Journal Article
Genes and Cancer, ISSN 1947-6019, 2012, Volume 3, Issue 5-6, pp. 447 - 454
The development of inhibitors against Abl has changed the landscape for the treatment of chronic myelogenous leukemia (CML) and cancer in general. Beginning... 
imatinib | ponatinib | X-ray crystal structure | dasatinib | nilotinib | BCR-ABL | Monographs
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2005, Volume 102, Issue 6, pp. 1992 - 1997
..., Anthony D. Kang, Kimberly A. Robell, M. V. Ramana Reddy* † , and E. Premkumar Reddy †‡ The Fels Institute for Cancer Research and Molecular Biology, Temple University... 
Biological Sciences | Phosphorylation | K562 cells | Medical treatment | Cell lines | Mice | Chronic myeloid leukemia | Inhibitory concentration 50 | Dosage | Genetic mutation | Blood | APOPTOSIS | substrate-competitive | INTERLEUKIN-3 | STI571 | MULTIDISCIPLINARY SCIENCES | MECHANISMS | Gleevec | ON012380 | INDEPENDENCE | PHILADELPHIA-CHROMOSOME | GROWTH | TYROSINE KINASE INHIBITOR | LEUKEMIA | Protein-Tyrosine Kinases - metabolism | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Drug Resistance, Neoplasm | Piperazines - metabolism | Antineoplastic Agents - therapeutic use | Piperazines - chemistry | Pyrimidines - chemistry | Pyrimidines - metabolism | Antineoplastic Agents - metabolism | Protein-Tyrosine Kinases - genetics | Fusion Proteins, bcr-abl | Adenosine Triphosphate - metabolism | Cell Death | Female | Molecular Structure | Recombinant Proteins - metabolism | Benzene Derivatives - metabolism | Benzene Derivatives - therapeutic use | Piperazines - therapeutic use | Recombinant Proteins - genetics | Antineoplastic Agents - chemistry | Imatinib Mesylate | Animals | Mice, Nude | Pyrimidines - therapeutic use | K562 Cells | Benzamides | Mutation | Benzene Derivatives - chemistry | Protein-Tyrosine Kinases - antagonists & inhibitors | Care and treatment | Research
Journal Article