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Genome Biology, ISSN 1474-7596, 2017, Volume 18, Issue 1, pp. 1 - 21
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2011, Volume 31, Issue 45, pp. 16298 - 16308
Journal Article
by Chen, Shu-Huang and Chen, Lu and Ge, Bing and Casale, Francesco Paolo and Vasquez, Louella and Kwan, Tony and Garrido-Martín, Diego and Watt, Stephen and Yan, Ying and Kundu, Kousik and Ecker, Simone and Datta, Avik and Richardson, David and Burden, Frances and Mead, Daniel and Mann, Alice L and Fernandez, Jose Maria and Rowlston, Sophia and Wilder, Steven P and Farrow, Samantha and Shao, Xiaojian and Lambourne, John J and Redensek, Adriana and Albers, Cornelis A and Amstislavskiy, Vyacheslav and Ashford, Sofie and Berentsen, Kim and Bomba, Lorenzo and Bourque, Guillaume and Bujold, David and Busche, Stephan and Caron, Maxime and Cheung, Warren and Delaneau, Oliver and Dermitzakis, Emmanouil T and Elding, Heather and Colgiu, Irina and Bagger, Frederik O and Flicek, Paul and Habibi, Ehsan and Iotchkova, Valentina and Janssen-Megens, Eva and Kim, Bowon and Lehrach, Hans and Lowy, Ernesto and Mandoli, Amit and Matarese, Filomena and Maurano, Matthew T and Morris, John A and Pancaldi, Vera and Pourfarzad, Farzin and Rehnstrom, Karola and Rendon, Augusto and Risch, Thomas and Sharifi, Nilofar and Simon, Marie-Michelle and Sultan, Marc and Valencia, Alfonso and Walter, Klaudia and Wang, Shuang-Yin and Frontini, Mattia and Antonarakis, Stylianos E and Clarke, Laura and Yaspo, Marie-Laure and Beck, Stephan and Guigo, Roderic and Rico, Daniel and Martens, Joost H.A and Ouwehand, Willem H and Kuijpers, Taco W and Paul, Dirk S and Stunnenberg, Hendrik G and Stegle, Oliver and Downes, Kate and Pastinen, Tomi and Soranzo, Nicole
Cell, ISSN 0092-8674, 11/2016, Volume 167, Issue 5, pp. 1398 - 1414.e24
Journal Article
Biological Psychiatry, ISSN 0006-3223, 05/2018, Volume 83, Issue 9, pp. S7 - S7
Journal Article
JOURNAL OF NEUROSCIENCE, ISSN 0270-6474, 11/2011, Volume 31, Issue 45, pp. 16298 - 16308
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2011, Volume 31, Issue 38, pp. 13412 - 13419
Journal Article
Genome biology, ISSN 1465-6906, 2017, Volume 18, Issue 1, p. 18
Journal Article
Journal Article
FASEB Journal, ISSN 0892-6638, 2011, Volume 25, Issue 12, pp. 4240 - 4252
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 48, pp. 12736 - 12747
CNS injury-induced hemorrhage and tissue damage leads to excess iron, which can cause secondary degeneration. The mechanisms that handle this excess iron are... 
Iron chelation | Inflammation | Free radicals | Hemorrhage | CNS injury | Macrophage | FUNCTIONAL RECOVERY | free radicals | GLYCOSYLPHOSPHATIDYLINOSITOL-ANCHORED FORM | RAT | macrophage | 2-CHLOROBENZOYL HYDRAZONE | NEUROSCIENCES | CELL-DEATH | INDUCED CARDIOTOXICITY | inflammation | iron chelation | NITRIC-OXIDE | PYRIDOXAL ISONICOTINOYL HYDRAZONE | OVERLOAD DISEASE | EXPRESSION | hemorrhage | Iron Metabolism Disorders - etiology | Neuroprotective Agents - therapeutic use | Neurons - pathology | Spinal Cord - drug effects | Spinal Cord - metabolism | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | Spinal Cord Injuries - complications | Oxidative Stress - physiology | Ceruloplasmin - pharmacology | Neuroprotective Agents - pharmacology | Recovery of Function - physiology | Neurons - metabolism | Neurons - drug effects | Disease Models, Animal | Hemorrhage - physiopathology | Iron Metabolism Disorders - drug therapy | Mice, Inbred C57BL | Hemorrhage - complications | Treatment Outcome | Antioxidants - pharmacology | Ceruloplasmin - therapeutic use | Iron - metabolism | Mice, Knockout | Antioxidants - therapeutic use | Hemorrhage - drug therapy | Iron Metabolism Disorders - physiopathology | Animals | Mice | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Oxidative Stress - drug effects | Iron - toxicity | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2010, Volume 30, Issue 9, pp. 3220 - 3226
The inflammatory response is thought to contribute to secondary damage after spinal cord injury (SCI). Polyunsaturated fatty acids (PUFAs) play an important... 
TUMOR-NECROSIS-FACTOR | INFLAMMATION | MOUSE MODEL | TRAFFICKING | FATTY-ACIDS | TRANS-RETINOIC ACID | TNF-ALPHA | CERAMIDE DEFICIENCY | EXPRESSION | NEUROSCIENCES | CELL-DEATH | Neuroprotective Agents - therapeutic use | Tumor Necrosis Factor-alpha - metabolism | Docosahexaenoic Acids - agonists | Microglia - metabolism | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | Oxidative Stress - physiology | Arachidonic Acid - blood | Nerve Degeneration - physiopathology | Cytoprotection - physiology | Arachidonic Acid - antagonists & inhibitors | Neuroprotective Agents - pharmacology | Nerve Degeneration - prevention & control | Cytoprotection - drug effects | Inflammation Mediators - metabolism | Anti-Inflammatory Agents - therapeutic use | Recovery of Function - physiology | Female | Inflammation Mediators - antagonists & inhibitors | Anticarcinogenic Agents - pharmacology | Anticarcinogenic Agents - therapeutic use | Disease Models, Animal | Biomarkers - metabolism | Drug Administration Schedule | Microglia - drug effects | Spinal Cord Injuries - metabolism | Administration, Oral | Anti-Inflammatory Agents - pharmacology | Fenretinide - pharmacology | Gene Expression Regulation - physiology | Fatty Acids, Unsaturated - metabolism | Gene Expression Regulation - drug effects | Fenretinide - therapeutic use | Animals | Docosahexaenoic Acids - blood | Mice | Mice, Inbred BALB C | Spinal Cord Injuries - physiopathology | Oxidative Stress - drug effects | Nerve Degeneration - drug therapy | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
Glia, ISSN 0894-1491, 04/2011, Volume 59, Issue 4, pp. 603 - 614
Prostaglandin D 2 (PGD 2 ) is a potent inflammatory mediator, which is implicated in both the initiation and resolution of inflammation in peripheral... 
spinal cord injury | inflammation | prostaglandins | Spinal cord injury | Prostaglandins | Inflammation | MOUSE-BRAIN | FUNCTIONAL RECOVERY | ALLERGIC INFLAMMATION | AXONAL REGENERATION | NEUROSCIENCES | DP1 RECEPTOR | MESSENGER-RNA | TRANSFORMING GROWTH-FACTOR-BETA-1 | CENTRAL-NERVOUS-SYSTEM | D SYNTHETASE | CELLULAR-LOCALIZATION | Microglia - metabolism | Spinal Cord - drug effects | Spinal Cord - metabolism | Recovery of Function - drug effects | Galectin 3 - metabolism | Spinal Cord Injuries - genetics | Immunoenzyme Techniques | Piperidines - pharmacology | Recovery of Function - physiology | Female | Isomerases - genetics | Interleukin-6 - metabolism | Interleukin-6 - genetics | Microglia - drug effects | Spinal Cord Injuries - metabolism | Galectin 3 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Macrophages - metabolism | Animals | Analysis of Variance | Macrophages - drug effects | Mice | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Isomerases - metabolism | Enzymes | Central nervous system | Data processing | Macrophages | Hemopoiesis | Microglia | Antioxidants | Interleukin 6 | Cell activation | Prostaglandin D2 synthase | prostaglandin D2 | CXCL10 protein | Chromium | Chemokines | Metallothionein
Journal Article