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Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 2, pp. 607 - 620.e15
Background Patients with heterozygous germline mutations in phosphatase and tensin homolog deleted on chromosome 10 (PTEN) experience autoimmunity and lymphoid... 
Allergy and Immunology | PHTS | autoimmunity | immunologic synapse | PTEN | Regulatory T cells | phosphatases | phosphoinositide 3-kinase | PH domain leucine-rich repeat protein phosphatase | HOMEOSTASIS | PROTEIN | KINASE | PROMOTER MUTATIONS | RILEY-RUVALCABA-SYNDROME | IMMUNOLOGY | ALLERGY | RESOLUTION MELTING ANALYSIS | AKT-MTOR | LYMPHOID HYPERPLASIA | EXPRESSION | Autoimmunity | Humans | Hyperplasia | Middle Aged | Phosphoprotein Phosphatases - metabolism | Male | Immunological Synapses - metabolism | Young Adult | Forkhead Transcription Factors - metabolism | Membrane Potential, Mitochondrial | Adult | Child | Hamartoma Syndrome, Multiple - immunology | Lymphocyte Subsets - physiology | T-Lymphocytes, Regulatory - physiology | PTEN Phosphohydrolase - genetics | Hamartoma Syndrome, Multiple - genetics | Signal Transduction | Cells, Cultured | B-Lymphocytes - physiology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Mutation - genetics | Protein Transport | Adolescent | Protein Binding | Aged | Medical colleges | Care and treatment | Phosphatases | Gene mutations | Analysis | Children's hospitals | Genomics | Medical genetics | Genetic research | T cells | Tumors | Hamartoma | Proteins | Blood & organ donations | Hematology | Biopsy | Lymphocytes | T cell receptors | Mutation | Kinases | Metabolism | Phosphatase | FACS, Fluorescence-activated cell sorting | TCR, T-cell receptor | mTOR complex 1 | NHERF1, Na+ | H+-exchanger 3 regulatory factor | MALT, Mucosa-associated lymphoid tissue | APC, Allophycocyanin | SHIP, Src homology domain 2–containing inositol phosphatase | HRM, High Resolution DNA Melting | FITC, Fluorescein isothiocyanate | PP2A, Protein phosphatase 2A | mTORC1, PTEN | PHTS, PTEN hamartoma tumor syndrome | PerCP, Peridinin-chlorophyll-protein complex | CS, Cowden syndrome | POD, Peroxidase | Tmem, Memory T | TMRE, Tetramethylrhodamine-ethylester | Immune Deficiencies, Infection, and Systemic Immune Disorders | IC50, Inhibitory concentration of 50 | CTLA-4, Cytotoxic T lymphocyte–associated antigen 4 | PHLPP, PH domain leucine-rich repeat protein phosphatase | AKT | DLG1, Scaffold protein discs, large homolog 1 | FOXP3, Forkhead box P3 | ICAM-1, Intercellular adhesion molecule 1 | iTreg, In vitro induced regulatory T | PE, Phycoerythrin | PI3K, Phosphoinositide 3-kinase | mTOR, Mammalian target of rapamycin | PTEN, Phosphatase and tensin homologue deleted on chromosome 10
Journal Article
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 12/2004, Volume 108, Issue 6, pp. 467 - 470
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2012, Volume 287, Issue 24, pp. 20056 - 20069
Journal Article
PLoS Genetics, ISSN 1553-7390, 03/2012, Volume 8, Issue 3, p. e1002572
Journal Article
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 12/2017, Volume 134, Issue 6, pp. 905 - 922
In search of novel germline alterations predisposing to tumors, in particular to gliomas, we studied a family with two brothers affected by anaplastic gliomas,... 
Pathology | Prostate carcinoma | Neurosciences | Medicine & Public Health | Glioma | Type I interferon signaling | Whole-exome sequencing | Aicardi–Goutières syndrome | DNA-BINDING DOMAIN | RNA | IDH1 | SUSCEPTIBILITY | PATHOLOGY | SAMHD1 | RIBONUCLEOTIDES | NEUROSCIENCES | GENOME | CLINICAL NEUROLOGY | AICARDI-GOUTIERES-SYNDROME | Aicardi-Goutieres syndrome | GRADE | MUTATIONS | Neoplasms - metabolism | RNA-Binding Proteins - genetics | Adenosine Deaminase - metabolism | Humans | DNA Repair Enzymes - genetics | Male | Neoplasms - genetics | Tumor Suppressor Proteins - genetics | Interferon Type I - metabolism | Adult | Female | Protein Stability | Fibroblasts - metabolism | Ribonuclease H - genetics | Genetic Predisposition to Disease | Adenosine Deaminase - genetics | Cells, Cultured | Isocitrate Dehydrogenase - genetics | Molecular Dynamics Simulation | Mice, Knockout | Neoplasms - drug therapy | DNA Modification Methylases - genetics | Phenotype | Animals | Polymorphism, Single Nucleotide | Neoplasms - pathology | RNA-Binding Proteins - metabolism | Cohort Studies | Carcinoma | Gliomas | Genes | Development and progression | Interferon | Biological response modifiers | Health aspects | Risk factors | Cancer | Transformation | Tumor cells | Brain tumors | Peripheral blood | Calcification | Tumorigenesis | Prostate | Minority & ethnic groups | Tumors
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2007, Volume 104, Issue 48, pp. 19055 - 19060
The novel keratinocyte-specific chemokine CCL27 plays a critical role in the organization of skin-associated immune responses by regulating T cell homing under... 
T lymphocytes | Messenger RNA | Skin cancers | Keratinocytes | Antibodies | Skin | Mice | Chemokines | Lymph nodes | Tumors | EGDR/R pathway | CCL27/CTACK | Immune surveillance | Cancer | RAS | MULTIDISCIPLINARY SCIENCES | skin | RECEPTOR | KERATINOCYTES | CANCER-THERAPY | IMMUNOTHERAPY | INFLAMMATION | SKIN-ASSOCIATED CHEMOKINE | GROWTH | cancer | immune surveillance | T-CELLS | PROGRESSION | Carcinoma, Basal Cell - genetics | Carcinoma, Squamous Cell - genetics | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Antibodies, Monoclonal - therapeutic use | Melanoma, Experimental - metabolism | Precancerous Conditions - metabolism | Photosensitivity Disorders - metabolism | Proto-Oncogene Proteins p21(ras) - physiology | Extracellular Signal-Regulated MAP Kinases - metabolism | Melanoma, Experimental - immunology | Chemokine CCL27 - genetics | Photosensitivity Disorders - immunology | Chemokine CCL27 - antagonists & inhibitors | Neoplasm Proteins - genetics | Receptor, Epidermal Growth Factor - physiology | Chemokine CCL27 - physiology | Signal Transduction | Skin Neoplasms - immunology | Neoplasm Proteins - biosynthesis | Antibodies, Monoclonal - pharmacology | Down-Regulation | Mice, Inbred C57BL | Carcinoma, Basal Cell - immunology | Tumor Escape - physiology | Carcinoma, Squamous Cell - immunology | Animals | Melanoma, Experimental - genetics | Precancerous Conditions - immunology | Keratinocytes - metabolism | Skin Neoplasms - genetics | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Chemokine CCL27 - biosynthesis | Cytotoxicity, Immunologic | Prevention | Immune response | Research | T cells | Health aspects | Skin tumors | EGDR | Biological Sciences | R pathway | CCL27 | CTACK
Journal Article
by Rhea van den Bruck and Patrick P Weil and Thomas Ziegenhals and Philipp Schreiner and Stefan Juranek and Daniel Gödde and Silvia Vogel and Frauke Schuster and Valerie Orth and Johannes Dörner and Daniel Pembaur and Meike Röper and Stefan Störkel and Hubert Zirngibl and Stefan Wirth and Andreas C W Jenke and Jan Postberg and Nikolas Boy and Jana Heringer and Gisela Haege and Esther M Glahn and Georg F Hoffmann and Sven F Garbade and Peter Burgard and Stefan Kölker and Cho-Ming Chao and Faady Yahya and Alena Moiseenko and Amit Shrestha and Negah Ahmadvand and Jennifer Quantius and Jochen Wilhelm and Elie El-Agha and Klaus-Peter Zimmer and Saverio Bellusci and Christian Staufner and Holger Prokisch and Stephan Seeliger and Matthias Müller and Andreas Hippe and Henrik Steinkraus and Roland Wauer and Burkhard Lachmann and Sigrun R Hofmann and Christian M Hedrich and Jakob Zierk and Farhad Arzideh and Rainer Haeckel and Wolfgang Rascher and Manfred Rauh and Markus Metzler and Sebastian Thieme and Joanna Bandoła and Cornelia Richter and Martin Ryser and Arshad Jamal and Michelle P Ashton and Malte von Bonin and Matthias Kuhn and Ezio Bonifacio and Reinhard Berner and Sebastian Brenner and Johanna Hammersen and Cristina Has and Nora Naumann-Bartsch and Daniel Stachel and Dimitra Kiritsi and Stephan Söder and Mathilde Tardieu and Leena Bruckner-Tuderman and Holm Schneider and F Bohne and D Langer and R Cencic and T Eggermann and U Zechner and J Pelletier and F Zepp and T Enklaar and D Prawitt and Martin Pech and Markus Weckmann and Femke-Anouska Heinsen and Andre Franke and Christine Happle and Anna-Maria Dittrich and Gesine Hansen and Oliver Fuchs and Erika von Mutius and Brian G Oliver and Matthias V Kopp and Claudia Paret and Alexandra Russo and Johanna Theruvath and Bettina Keller and Khalifa El Malki and Nadine Lehmann and Arthur Wingerter and Marie A Neu and Gerhold-Ay Aslihan and ...
Molecular and Cellular Pediatrics, 05/2017, Volume 4, Issue 1, pp. 1 - 9
Journal Article
Journal Article
Journal Article
International Journal of Cancer, ISSN 0020-7136, 08/2001, Volume 93, Issue 3, pp. 445 - 449
Journal Article
PLoS Genetics, ISSN 1553-7390, 03/2012, Volume 8, Issue 3
Medulloblastoma is the most common malignant brain tumor in children. A subset of medulloblastoma originates from granule cell precursors (GCPs) of the... 
Medulloblastoma | Physiological aspects | Development and progression | Genotype | Genetic aspects | Research | Gene expression | Cancer
Journal Article