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Science, ISSN 0036-8075, 12/2010, Volume 330, Issue 6009, pp. 1390 - 1393
Although the proteins BAX and BAK are required for initiation of apoptosis at the mitochondria, how BAX and BAK are activated remains unsettled. We provide in... 
T lymphocytes | Mitochondria | Cytokines | Thymocytes | Neurons | Cell death | REPORTS | Cytochromes | Mice | Potassium | Apoptosis | NEURONAL APOPTOSIS | CYTOCHROME-C | MITOCHONDRIAL APOPTOSIS | MECHANISM | MULTIDISCIPLINARY SCIENCES | BH3 DOMAINS | RELEASE | JNK PATHWAY | PROTEINS | BCL-2 FAMILY-MEMBERS | MEMBRANE PERMEABILIZATION | BH3 Interacting Domain Death Agonist Protein - deficiency | T-Lymphocytes - physiology | bcl-2-Associated X Protein - chemistry | Protein Multimerization | Stress, Physiological | bcl-2 Homologous Antagonist-Killer Protein - genetics | BH3 Interacting Domain Death Agonist Protein - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Membrane Proteins - deficiency | Caspases - metabolism | Bcl-2-Like Protein 11 | Apoptosis Regulatory Proteins - deficiency | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Neurons - physiology | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cytochromes c - metabolism | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Permeability | Proto-Oncogene Proteins - deficiency | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Models, Biological | Cerebellum - cytology | Intracellular Membranes - metabolism | bcl-2 Homologous Antagonist-Killer Protein - chemistry | Protein research | Genetic aspects | Mitochondrial DNA | Biochemical genetics | Research | Properties | Methods | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 362
N6-methyladenosine (m6A) is the most abundant mRNA modification. However, the role of m6A in regulating β-cell function is unknown. To address the functional... 
Cell proliferation | RNA modification | Secretion | Diabetes mellitus | Methyltransferase | Transgenic mice | Glucose | Tamoxifen | Gene expression | Insulin | Feeding | Beta cells | 3' Untranslated regions | Clonal deletion | Cell death | Rodents | Diabetes | Insulin-like growth factor-binding protein 5
Journal Article
1976, Tai 1 ban., Zhongguo fang zhi cong shu Hua bei di fang, Volume di 365 hao, 3 v. (1108 p.)
Book
Nature Cell Biology, ISSN 1465-7392, 10/2015, Volume 17, Issue 10, pp. 1270 - 1281
Multidomain pro-apoptotic BAX and BAK, once activated, permeabilize mitochondria to trigger apoptosis, whereas anti-apoptotic BCL-2 members preserve... 
CYTOCHROME-C | MITOCHONDRIAL APOPTOSIS | ACTIVATION | CONFORMATIONAL-CHANGE | MEMBERS | PROAPOPTOTIC BAX | BH3 DOMAINS | PUMA | BH3-ONLY PROTEINS | MEMBRANE PERMEABILIZATION | CELL BIOLOGY | bcl-2 Homologous Antagonist-Killer Protein - genetics | Immunoblotting | BH3 Interacting Domain Death Agonist Protein - genetics | Cytochromes c - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Embryo, Mammalian - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Bcl-2-Like Protein 11 | Mitochondria - genetics | RNA Interference | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Fibroblasts - metabolism | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cytochromes c - metabolism | Mice, Inbred C57BL | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Intestine, Small - cytology | Embryo, Mammalian - cytology | Models, Biological | Fibroblasts - cytology | Intestine, Small - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Apoptosis | Genotype | Genetic aspects | Properties | Cell death | Cellular control mechanisms | Index Medicus
Journal Article
Journal of Structural Engineering, ISSN 0733-9445, 02/2016, Volume 142, Issue 2, p. 4015122
AbstractIn this study, a novel force-based frame element with delocalization is developed to address localization issues induced by material strain softening.... 
Technical Papers | Force-based frame element | Strain softening | Analysis and computation | Delocalization | Plastic hinge | Implicit gradient method | R/C FRAMES | NONLINEAR-ANALYSIS | DAMAGE | CONCRETE | ISSUES | ENGINEERING, CIVIL | CONSTRUCTION & BUILDING TECHNOLOGY | BEAM-COLUMN MODEL | PLASTICITY | Hinges | Research | Analysis | Deformations (Mechanics)
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 08/2018, Volume 238, Issue 2, pp. 151 - 163
Journal Article
International Journal of Non-Linear Mechanics, ISSN 0020-7462, 07/2016, Volume 83, pp. 15 - 25
In the present paper, a kind of stochastic damage hysteretic model is proposed to describe the damage and hysteretic behaviors of concrete material. According... 
Micromechanical approach | Stochastic damage model | Sub-hysteresis | Hysteretic behavior | Plastic strain | MECHANICS | CONSTITUTIVE MODEL | FORMULATION | PLASTICITY | Analysis | Concrete | Models | Concretes | Fracture mechanics | Mathematical analysis | Mathematical models | Stochasticity | Damage | Hysteresis | Strain
Journal Article
The Proceedings of the ... International Offshore and Polar Engineering Conference, ISSN 1098-6189, 01/2018, Volume 3, p. 94
In order to study the influences of grid scale on the wave breaking, three different computational grids are adopted in the simulation of the bow wave breaking... 
Temperature | Atmospheric pressure | Computer simulation | Bow waves | Computational grids | Direction | Wave breaking | Free surfaces
Journal Article
IEEE Transactions on Geoscience and Remote Sensing, ISSN 0196-2892, 11/2018, Volume 56, Issue 11, pp. 6537 - 6546
Journal Article
杂文月刊:文摘版, ISSN 1009-2218, 2014, Issue 10, pp. 7 - 7
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 09/2019, Volume 1865, Issue 9, p. 2138
Defects in the development, maintenance or expansion of [beta]-cell mass can result in impaired glucose metabolism and diabetes. N.sup.6-methyladenosine... 
Glucose metabolism | Pancreatic beta cells | Methyltransferases | RNA | Cell death | Liver | Glucose | Cell differentiation | Genetic translation | Insulin | Cells | Dextrose
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 34, pp. 56768 - 56779
Mutations in the gene for Immediate Early Response 3 Interacting Protein 1 (IER3IP1) cause permanent neonatal diabetes mellitus in human. The mechanisms... 
IER3IP1 | Cell death and proliferation | Beta-cell | APOPTOSIS | ACTIVATION | PHOSPHORYLATION | BIM | ENDOPLASMIC-RETICULUM STRESS | cell death and proliferation | CELL BIOLOGY | UNFOLDED PROTEIN RESPONSE | beta-cell | BAX | EPILEPSY | PUMA | EXPRESSION
Journal Article
Composites Science and Technology, ISSN 0266-3538, 01/2019, Volume 169, pp. 45 - 51
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2018, Volume 503, Issue 4, p. 2866
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1016/j.bbrc.2018.08.057 As the... 
Enzymes | Inositol | Cell death | Analysis | Leukemia | Lymphomas | Tumor proteins | Mediation | Protein binding
Journal Article
DIABETES, ISSN 0012-1797, 07/2018, Volume 67, Issue Supplement 1, p. 2103
Conference Proceeding
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