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acute inferior myocardial infarction; cardiac remodeling, ventricular; cell transplantation; cell- and tissue-based therapy; pericytes; stem cells; angiogenic proteins; animals; cell differentiation; cell proliferation; cell survival; cells, cultured; coculture techniques; disease models, animal; fibrosis; hemodynamics; humans; mice, scid; myocardial contraction; myocardial infarction; myocardium; myocytes, cardiac; paracrine communication; pericytes; phenotype; recovery of function; saphenous vein; time factors; ventricular remodeling; neovascularization, physiologic; regeneration; stem cell transplantation; physiology; cardiology and cardiovascular medicine; medicine (1) 1
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Scientific Reports, ISSN 2045-2322, 07/2016, Volume 6, Issue 1, pp. 30639 - 30639
Journal Article
Journal Article
Endocrine, Metabolic and Immune Disorders - Drug Targets, ISSN 1871-5303, 06/2012, Volume 12, Issue 2, pp. 159 - 167
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2013, Volume 33, Issue 3, pp. 555 - 564
OBJECTIVE—Diabetes mellitus causes bone marrow (BM) microangiopathy. This study aimed to investigate the mechanisms responsible for BM endothelial dysfunction... 
diabetic microangiopathy | endothelial dysfunction | bone marrow | oxidative stress | RhoA | APOPTOSIS | PERMEABILITY | ANGIOGENESIS | PHOSPHORYLATION | NITRIC-OXIDE SYNTHASE | SELF-RENEWAL | AKT | INHIBITION | HEMATOPOIETIC STEM-CELLS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Diabetes Mellitus, Experimental - drug therapy | Phosphorylation | Cadherins - metabolism | Oxidative Stress | Bone Marrow Cells - enzymology | Diabetes Mellitus, Experimental - enzymology | Diabetes Mellitus, Experimental - genetics | Capillary Permeability - drug effects | Diabetic Angiopathies - etiology | Drug Implants | Male | rhoA GTP-Binding Protein - metabolism | Diabetes Mellitus, Type 1 - complications | rhoA GTP-Binding Protein - genetics | Proto-Oncogene Proteins c-akt - genetics | rho-Associated Kinases - antagonists & inhibitors | Antigens, CD - metabolism | Flow Cytometry | Transfection | Hypoglycemic Agents - administration & dosage | Time Factors | rho-Associated Kinases - metabolism | src-Family Kinases - metabolism | Antigens, Surface - metabolism | Bone Marrow Cells - drug effects | Diabetic Angiopathies - enzymology | Diabetes Mellitus, Experimental - complications | Diabetic Angiopathies - genetics | Proto-Oncogene Proteins c-akt - metabolism | Diabetes Mellitus, Type 1 - enzymology | Oxidation-Reduction | src-Family Kinases - antagonists & inhibitors | Cells, Cultured | Diabetes Mellitus, Type 1 - genetics | rho-Associated Kinases - genetics | Insulin - administration & dosage | Diabetic Angiopathies - drug therapy | Diabetes Mellitus, Type 1 - drug therapy | Animals | Signal Transduction - drug effects | rho GTP-Binding Proteins - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | Endothelial Cells - enzymology | Cell Movement | Endothelial Cells - drug effects | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 05/2015, Volume 116, Issue 10, pp. e81 - e94
RATIONALE:Optimization of cell therapy for cardiac repair may require the association of different cell populations with complementary activities.... 
Cardiac remodeling, ventricular | Pericytes | Acute inferior myocardial infarction | Cell transplantation | Cell- and tissue-based therapy | Stem cells | cell- and tissue-based therapy | PROGENITOR CELLS | stem cells | TISSUE KALLIKREIN | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | pericytes | REGENERATION | cardiac remodeling, ventricular | MODEL | CARDIOMYOCYTES | TRANSPLANTATION | acute inferior myocardial infarction | cell transplantation | IN-VITRO | ISCHEMIC CARDIOMYOPATHY | INTRACORONARY | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Cell Proliferation | Coculture Techniques | Humans | Recovery of Function | Stem Cell Transplantation | Ventricular Remodeling | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Pericytes - transplantation | Myocardial Infarction - physiopathology | Cell Differentiation | Saphenous Vein - cytology | Disease Models, Animal | Myocardial Contraction | Myocardial Infarction - surgery | Paracrine Communication | Cell Survival | Pericytes - metabolism | Cells, Cultured | Myocardium - pathology | Myocardial Infarction - metabolism | Mice, SCID | Myocytes, Cardiac - pathology | Myocytes, Cardiac - transplantation | Regeneration | Phenotype | Animals | Fibrosis | Myocytes, Cardiac - metabolism | Hemodynamics | Angiogenic Proteins - metabolism | Neovascularization, Physiologic | Index Medicus
Journal Article
Journal Article
2015
Diabetic subjects develop microangiopathy in the bone marrow (BM) resulting in depletion of stem cells (SC) and alteration of SC release upon tissue damage.... 
Dissertation
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2019, Volume 39, Issue 6, pp. 1113 - 1124
OBJECTIVE—To determine the role of the oncofetal protein TPBG (trophoblast glycoprotein) in normal vascular function and reparative vascularization. APPROACH... 
endothelial cells | glycoproteins | aspartic acid | immunohistochemistry | pericytes | BINDING-PROTEIN | CXCR4 | CITED2 MUTATION | BETA-ARRESTIN | HEART | INTERNALIZATION | CELL-MIGRATION | CHEMOKINE RECEPTOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | 5T4 ONCOFETAL ANTIGEN | TRANSCRIPTION FACTOR
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2015, Volume 35, Issue 3, pp. 675 - 688
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 10/2014, Volume 21, Issue 11, pp. 1591 - 1604
Journal Article
Molecular Therapy, ISSN 1525-0016, 12/2015, Volume 23, Issue 12, pp. 1854 - 1866
Reparative response by bone marrow (BM)-derived progenitor cells (PCs) to ischemia is a multistep process that comprises the detachment from the BM endosteal... 
Journal Article
Endocrine, metabolic & immune disorders drug targets, 06/2012, Volume 12, Issue 2, p. 159
Diabetes mellitus is considered a cardiovascular disease owing to its prevalent association with cardiovascular morbidity and mortality. Cardiovascular events... 
Severity of Illness Index | Osteoclasts - pathology | Oxidative Stress | Humans | Bone Marrow - blood supply | Cellular Microenvironment | Hematopoietic Stem Cells - pathology | Rats | Wound Healing | Animals | Diabetic Angiopathies - pathology | Diabetic Angiopathies - complications | Mice
Journal Article
Pharmacology and Therapeutics, ISSN 0163-7258, 03/2017, Volume 171, pp. 30 - 42
Pericytes are a heterogeneous population of cells located in the blood vessel wall. They were first identified in the 19th century by Rouget, however their... 
Pericytes | Perivascular stem cells | Diabetic retinopathy | Diabetic nephropathy | Pericyte fibrosis | Cancer stem cells | DIABETIC-RETINOPATHY | Cancer stern cells | VEGF-A | BLOOD-BRAIN-BARRIER | MESENCHYMAL STEM-CELLS | EPITHELIUM-DERIVED FACTOR | HIGH GLUCOSE | PHARMACOLOGY & PHARMACY | STROMAL CELLS | TUMOR-GROWTH | GLYCATION END-PRODUCTS | ENDOTHELIAL GROWTH-FACTOR | Neoplasms - therapy | Animals | Diabetes Mellitus - physiopathology | Vascular Diseases - therapy | Diabetes Mellitus - therapy | Humans | Pericytes - cytology | Ischemia - therapy | Vascular Diseases - physiopathology | Ischemia - physiopathology | Molecular Targeted Therapy | Neoplasms - pathology | Blood circulation disorders | Therapeutics | Homeopathy | Materia medica and therapeutics | Impotence | Diabetic nephropathies | T cells | Muscle proteins | Cardiovascular agents | Blood coagulation factor VIII | Interleukins | Chronic kidney failure | Type 1 diabetes | Stem cells | Vascular endothelial growth factor | Mitogens | Protein kinases | Growth factors | Index Medicus | UUO, unilateral ureteric obstruction | EGFR, EGF receptor | EMT, epithelial-mesenchymal transition | SFT, solitary fibrous tumour | DAN, diabetic autonomous neuropathy | ECM, extracellular matrix | NRF2, nuclear factor (erythroid-derived 2)-like 2 | IL-6, interleukin 6 | PSC, perivascular stem cell | BBB, blood-brain barrier | SOD, super oxide dismutase | HIF, hypoxia inducible factor | ED, erectile dysfunction | SDF-1, stromal derived factor 1 | RAGE, receptors of AGEs | CKD, chronic kidney disease | MMP, matrix metalloproteinases | MDSC, myeloid-derived suppressor cells | DN, diabetic nephropathy | TGF β, transforming growth factor β | PDL-1, programmed death-ligand 1 | ANG2, angiopoietin-2 | Olmfl3, Olfactomedin-like 3 | VEGF, vascular endothelial growth factor | AGE, Advanced Glycation End-Products | EC, endothelial cells | NG2, neural | R, ischemia-reperfusion | MSC, mesenchymal stromal cell | MF-EGF8, milk fat globule epidermal growth factor VIII | IL-8, interleukin 8 | glial antigen 2 | T1D, type 1 diabetic | PDGFb, platelet-derived growth factor B | MAPK, mitogen-activated protein kinase | PDGFRβ, platelet derived growth factor receptor β | DR, diabetic retinopathy | CNS, blood-retinal barrier | HB-EGF, heparin-binding EGF-like growth factor | HPC, hemangiopericytoma | PDR, proliferative diabetic retinopathy | DME, diabetic macular oedema | GFR, glomerular filtration rate | MI, myocardial infarction | SMA, smooth muscle actin | CSC, cancer stem cell | NPDR, non-proliferative diabetic retinopathy | T2D, type 2 diabetic | ROS, reactive oxygen species | Treg, regulatory T cells | BRB, blood-retina barrier | PKC, protein kinase C | DPN, diabetic peripheral neuropathy | TME, tumour microenvironment | ANG1, angiopoietin-1 | GSC, glioblastoma CSC | iPS, induced pluripotent stem cells | GSI, g-secretase inhibitor | FGF-9, fibroblastic growth factor 9 | PEDF, Pigment Epithelium-Derived Factor
Journal Article
Journal Article
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