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1. Full Text Polio: The Odyssey of Eradication by Thomas Abraham (review)
by Renne, Elisha P
Journal of Interdisciplinary History, ISSN 0022-1953, 2019, Volume 50, Issue 2, pp. 268 - 269
Prevention | Poliomyelitis | Abraham, Thomas, ---- author | Developing countries | History
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2. Full Text Polio: The Odyssey of Eradication. By Thomas Abraham (London, C. Hurst & Co., 2018) 252 pp. $29.95
by Renne, Elisha P
The Journal of Interdisciplinary History, ISSN 0022-1953, 08/2019, Volume 50, Issue 2, pp. 268 - 269
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3. Full Text Mast Cells Increase Vascular Permeability by Heparin-Initiated Bradykinin Formation In Vivo
by Oschatz, Chris and Maas, Coen and Lecher, Bernd and Jansen, Thomas and Björkqvist, Jenny and Tradler, Thomas and Sedlmeier, Reinhard and Burfeind, Peter and Cichon, Sven and Hammerschmidt, Sven and Müller-Esterl, Werner and Wuillemin, Walter A and Nilsson, Gunnar and Renné, Thomas
Immunity, ISSN 1074-7613, 02/2011, Volume 34, Issue 2, pp. 258 - 268
Activated mast cells trigger edema in allergic and inflammatory disease. We report a paracrine mechanism by which mast cell-released heparin increases vascular... 
COAGULATION-FACTOR-XII | CONTACT SYSTEM | ACTIVATION | ENZYME | HEREDITARY ANGIOEDEMA | CHONDROITIN SULFATE-E | ADVERSE CLINICAL EVENTS | ALLERGIC REACTIONS | RECEPTOR | MECHANISMS | IMMUNOLOGY | Capillary Permeability - physiology | Plasma | Capillary Leak Syndrome - physiopathology | Kallikrein-Kinin System - physiology | Complement C1 Inhibitor Protein - physiology | Heparin - secretion | Male | Leukocytes - physiology | Factor XII - physiology | Paracrine Communication - physiology | Immunoglobulin E - immunology | Heparin - physiology | Hypotension - physiopathology | Passive Cutaneous Anaphylaxis - physiology | Hypotension - etiology | Bradykinin - biosynthesis | Capillary Leak Syndrome - etiology | Rats | Cell Adhesion | Skin - blood supply | Edema - etiology | Animals | Mast Cells - secretion | Signal Transduction - physiology | Edema - physiopathology | Mice | Enzyme Activation | Endothelial Cells - pathology | Bradykinin - genetics | Heparan sulfate | Rodents | Blood pressure | Permeability | Pressure transducers | Veins & arteries
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4. Full Text Jennifer Cole and Lynn M. Thomas, eds. Love in Africa. Chicago: The University of Chicago Press, 2009. 280 pp. Illustrations. Bibliography. List of Contributors. Index. $63.00. Cloth; $23.00. Paper
by Renne, Elisha P
African Studies Review, ISSN 0002-0206, 04/2010, Volume 53, Issue 1, pp. 213 - 214
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5. Full Text Differential VASP phosphorylation controls remodeling of the actin cytoskeleton
by Benz, Peter M and Blume, Constanze and Seifert, Stefanie and Wilhelm, Sabine and Waschke, Jens and Schuh, Kai and Gertler, Frank and Münzel, Tomas and Renné, Thomas
Journal of Cell Science, ISSN 0021-9533, 2009, Volume 122, Issue 21, pp. 3954 - 3965
Proteins of the Enabled/vasodilator-stimulated phosphoprotein (Ena/VASP) family link signal transduction pathways to actin cytoskeleton dynamics. VASP is... 
Vasodilator-stimulated phosphoprotein (VASP) | Actin turnover | Serine/threonine kinase | Ena/VASP family | LISTERIA-MONOCYTOGENES | F-ACTIN | VASODILATOR-STIMULATED-PHOSPHOPROTEIN | DEPENDENT PROTEIN-KINASE | CELL-ADHESION | ENA/VASP PROTEINS | INTACT HUMAN PLATELETS | CELL BIOLOGY | FIBROBLAST MOTILITY | FOCAL ADHESION | SERUM RESPONSE FACTOR | Cell Adhesion Molecules - chemistry | Cell Line | Microfilament Proteins - chemistry | Cell Adhesion Molecules - genetics | Phosphorylation | Humans | Mice, Inbred C57BL | Actins - metabolism | Cytoskeleton - genetics | Phosphoproteins - genetics | Phosphoproteins - metabolism | Cell Adhesion Molecules - metabolism | Phosphoproteins - chemistry | Actins - genetics | Mice, Knockout | Protein Transport | Animals | Cytoskeleton - metabolism | Mice | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Ena | threonine kinase | VASP family | Serine
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6. Full Text The procoagulant and proinflammatory plasma contact system
by Renné, Thomas
Seminars in Immunopathology, ISSN 1863-2297, 1/2012, Volume 34, Issue 1, pp. 31 - 41
The contact system is a plasma protease cascade that is initiated by coagulation factor XII activation on cardiovascular cells. The system starts procoagulant... 
Leakage | Plasma | Edema | Biomedicine | Immunology | Hereditary angioedema | Bradykinin | Internal Medicine | Factor XII | Thrombosis | CHONDROITIN SULFATE | BLOOD-COAGULATION | MAST-CELLS | ADVERSE CLINICAL EVENTS | VASCULAR-PERMEABILITY | IMMUNOLOGY | PATHOLOGY | ARTERIAL THROMBUS FORMATION | COAGULATION-FACTOR-XII | IN-VIVO | HAGEMAN-FACTOR | Factor XII - immunology | Kallikreins - immunology | Blood Platelets - immunology | Humans | Platelet Activation - immunology | Hypersensitivity - immunology | Kinins - genetics | Bacterial Infections - genetics | Blood Coagulation - immunology | Bacterial Infections - immunology | Hypersensitivity - genetics | Factor XII - genetics | Kallikreins - genetics | Plasma - immunology | Thrombosis - immunology | Mice, Transgenic | Inflammation - immunology | Angioedemas, Hereditary - immunology | Animals | Inflammation - genetics | Blood Coagulation - genetics | Thrombosis - genetics | Mice | Kinins - immunology | Angioedemas, Hereditary - genetics | Anticoagulants (Medicine) | Glycosaminoglycans | Bacterial infections | Proteases | Health aspects | Genetically modified organisms | Pathogens | Proteinase | Coagulation | polyphosphates | Data processing | Allergic diseases | Inflammation | Inflammatory diseases | Infection | angioedema | Reviews | Genetic engineering | Heparin | Platelets | Coagulation factor XII
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7. Full Text Intrinsic Pathway of Coagulation and Arterial Thrombosis
by Gailani, David and Renné, Thomas
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2007, Volume 27, Issue 12, pp. 2507 - 2513
Formation of a fibrin clot is mediated by a group of tightly regulated plasma proteases and cofactors. While this system is essential for minimizing blood loss... 
Arterial thrombosis | Coagulation | Intrinsic pathways | VENOUS THROMBOSIS | DEEP-VEIN THROMBOSIS | coagulation | VON-WILLEBRAND-FACTOR | VERTEBRATE BLOOD-COAGULATION | KALLIKREIN-KININ SYSTEM | ACUTE MYOCARDIAL-INFARCTION | PERIPHERAL VASCULAR DISEASE | 14 SWISS FAMILIES | intrinsic pathways | arterial thrombosis | FACTOR-XI DEFICIENCY | CORONARY-HEART-DISEASE | HEMATOLOGY | TISSUE FACTOR | Reperfusion Injury - etiology | Factor XII Deficiency - blood | Humans | Reperfusion Injury - blood | Factor XI - antagonists & inhibitors | Protease Inhibitors - pharmacology | Thromboembolism - prevention & control | Infarction, Middle Cerebral Artery - complications | Factor XII Deficiency - genetics | Thrombosis - blood | Thrombosis - prevention & control | Protease Inhibitors - therapeutic use | Thromboembolism - genetics | Disease Models, Animal | Hemostasis - drug effects | Factor XI - genetics | Factor XII - genetics | Anticoagulants - therapeutic use | Hemostasis - genetics | Infarction, Middle Cerebral Artery - blood | Mice, Knockout | Factor XI Deficiency - blood | Animals | Thromboembolism - blood | Blood Coagulation - drug effects | Anticoagulants - pharmacology | Factor XII - antagonists & inhibitors | Factor XII - metabolism | Blood Coagulation - genetics | Thrombosis - genetics | Mice | Factor XI - metabolism | Fibrin - metabolism | Factor XI Deficiency - genetics
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8. Full Text Coagulation factor XII in thrombosis and inflammation
by Maas, Coen and Renne, Thomas
Blood, ISSN 0006-4971, 04/2018, Volume 131, Issue 17, pp. 1903 - 1909
Combinations of proinflammatory and procoagulant reactions are the unifying principle for a variety of disorders affecting the cardiovascular system. The... 
POLYPHOSPHATE NANOPARTICLES | PLATELET POLYPHOSPHATES | SAFE THROMBOPROTECTION | BLOOD-COAGULATION | HEREDITARY ANGIOEDEMA | CHAIN FACTOR-XII | IN-VIVO | CONTACT SYSTEM ACTIVATION | HEPARAN-SULFATE PROTEOGLYCANS | ANGIOEDEMA TYPE-III | HEMATOLOGY
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9. Full Text Defective glycosylation of coagulation factor XII underlies hereditary angioedema type III
by Björkqvist, Jenny and De Maat, Steven and Lewandrowski, Urs and Di Gennaro, Antonio and Oschatz, Chris and Schönig, Kai and Nöthen, Markus M and Drouet, Christian and Braley, Hal and Nolte, Marc W and Sickmann, Albert and Panousis, Con and Maas, Coen and Renné, Thomas
Journal of Clinical Investigation, ISSN 0021-9738, 08/2015, Volume 125, Issue 8, pp. 3132 - 3146
Hereditary angioedema type III (HAEIII) is a rare inherited swelling disorder that is associated with point mutations in the gene encoding the plasma protease... 
C1 INHIBITOR | MEDICINE, RESEARCH & EXPERIMENTAL | CONTACT SYSTEM | PLASMA KALLIKREIN | MOLECULAR-WEIGHT KININOGEN | FACTOR 12 GENE | IN-VIVO | HAGEMAN-FACTOR | VASCULAR-PERMEABILITY | NORMAL C1-INHIBITOR | MISSENSE MUTATIONS | Bradykinin - metabolism | Factor XII - genetics | Humans | Hereditary Angioedema Type III - metabolism | Antibodies, Neutralizing - pharmacology | Mutation, Missense | Glycosylation - drug effects | Hereditary Angioedema Type III - genetics | Mice, Knockout | Hereditary Angioedema Type III - pathology | Animals | Factor XII - metabolism | Blood Coagulation | Hereditary Angioedema Type III - drug therapy | Adult | Female | Mice | Amino Acid Substitution | Bradykinin - genetics | Disease Models, Animal | Angioneurotic edema | Abnormalities | Physiological aspects | Genetic aspects | Glycosylation | Research | Blood coagulation factors | Risk factors | Peptides | Rodents | Mutation | Permeability | Gene expression | Patients | Molecular weight
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10. Full Text Adverse Events Associated with Testosterone Administration
by Basaria, Shehzad and Coviello, Andrea D and Travison, Thomas G and Storer, Thomas W and Farwell, Wildon R and Jette, Alan M and Eder, Richard and Tennstedt, Sharon and Ulloor, Jagadish and Zhang, Anqi and Choong, Karen and Lakshman, Kishore M and Mazer, Norman A and Miciek, Renee and Krasnoff, Joanne and Elmi, Ayan and Knapp, Philip E and Brooks, Brad and Appleman, Erica and Aggarwal, Sheetal and Bhasin, Geeta and Bhasin, Shalender and Hede-Brierley, Leif and Bhatia, Ashmeet and Collins, Lauren and LeBrasseur, Nathan and Fiore, Louis D
The New England Journal of Medicine, ISSN 0028-4793, 07/2010, Volume 363, Issue 2, pp. 109 - 122
In a randomized trial, men 65 years of age or older who had low serum testosterone levels and limitations in mobility were assigned to either placebo or... 
SKELETAL-MUSCLE | MEDICINE, GENERAL & INTERNAL | MUSCLE STRENGTH | PLACEBO-CONTROLLED TRIALS | CARDIOVASCULAR-DISEASE | PHYSICAL FUNCTION | DOUBLE-BLIND | OLDER MEN | RANDOMIZED CONTROLLED-TRIAL | ANABOLIC-ANDROGENIC STEROIDS | BODY-COMPOSITION | Double-Blind Method | Gels | Obesity - complications | Humans | Risk Factors | Kaplan-Meier Estimate | Logistic Models | Male | Administration, Cutaneous | Walking | Exercise Test | Aged, 80 and over | Hypertension - complications | Testosterone - deficiency | Aged | Cardiovascular Diseases - chemically induced | Muscle Strength - drug effects | Testosterone - adverse effects | Testosterone - blood | Hyperlipidemias - complications | Testosterone - therapeutic use | Testosterone | Complications and side effects | Aged men | Dosage and administration | Cardiovascular diseases | Health aspects | Risk factors | Design | Heart failure | Mens health | Androgens | Heart attacks | Older people | Men | Aging | Data collection
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11. Full Text Plasmin is a natural trigger for bradykinin production in patients with hereditary angioedema with factor XII mutations
by de Maat, Steven, MSc and Björkqvist, Jenny, PhD and Suffritti, Chiara, PhD and Wiesenekker, Chantal P., MSc and Nagtegaal, Willem and Koekman, Arnold, BSc and van Dooremalen, Sanne, BSc and Pasterkamp, Gerard, PhD and de Groot, Philip G., PhD and Cicardi, Marco, PhD and Renné, Thomas, PhD and Maas, Coen, PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 138, Issue 5, pp. 1414 - 1423.e9
Background Patients with angioedema experience unpredictable attacks of tissue swelling in which bradykinin is implicated. Several distinct mutations in Factor... 
Allergy and Immunology | kininogen | Angioedema | plasmin | bradykinin | plasma kallikrein | Factor XII | C1 esterase inhibitor | KALLIKREIN CLEAVAGE | CONTACT SYSTEM ACTIVATION | IMMUNOLOGY | FIBRINOLYTIC SYSTEM | NORMAL C1-INHIBITOR | DEFICIENCY | C1 INHIBITOR | F12 GENE | ALLERGY | HAGEMAN-FACTOR | COAGULATION | ALTEPLASE TREATMENT | Pregnancy | Bradykinin - metabolism | Factor XII - genetics | Angioedemas, Hereditary - metabolism | Factor XII - metabolism | Humans | Antifibrinolytic Agents - pharmacology | Female | Complement C1 Inhibitor Protein - metabolism | Mutation | Fibrinolysin - metabolism | Aminocaproic Acid - pharmacology | Angioneurotic edema | Care and treatment | Genetic aspects | Bradykinin | Medical research | Medicine, Experimental | Kallikrein | Proteins | Plasma | Life sciences | Patients
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12. Full Text Application of High-Sensitivity Troponin in Suspected Myocardial Infarction
by Neumann, Johannes T and Twerenbold, Raphael and Ojeda, Francisco and Sörensen, Nils A and Chapman, Andrew R and Shah, Anoop S.V and Anand, Atul and Boeddinghaus, Jasper and Nestelberger, Thomas and Badertscher, Patrick and Mokhtari, Arash and Pickering, John W and Troughton, Richard W and Greenslade, Jaimi and Parsonage, William and Mueller-Hennessen, Matthias and Gori, Tommaso and Jernberg, Tomas and Morris, Niall and Liebetrau, Christoph and Hamm, Christian and Katus, Hugo A and Münzel, Thomas and Landmesser, Ulf and Salomaa, Veikko and Iacoviello, Licia and Ferrario, Marco M and Giampaoli, Simona and Kee, Frank and Thorand, Barbara and Peters, Annette and Borchini, Rossana and Jørgensen, Torben and Söderberg, Stefan and Sans, Susana and Tunstall-Pedoe, Hugh and Kuulasmaa, Kari and Renné, Thomas and Lackner, Karl J and Worster, Andrew and Body, Richard and Ekelund, Ulf and Kavsak, Peter A and Keller, Till and Lindahl, Bertil and Wild, Philipp and Giannitsis, Evangelos and Than, Martin and Cullen, Louise A and Mills, Nicholas L and Mueller, Christian and Zeller, Tanja and Westermann, Dirk and Blankenberg, Stefan
The New England Journal of Medicine, ISSN 0028-4793, 06/2019, Volume 380, Issue 26, pp. 2529 - 2540
Data from 15 international cohorts of patients with suspected acute MI were used to create and validate a risk-assessment tool integrating high-sensitivity... 
CARDIAC TROPONIN | POPULATION | MEDICINE, GENERAL & INTERNAL | EARLY-DIAGNOSIS | BIOMARKER | ALGORITHM | VALIDATION | RULE-OUT | OUTCOMES | I ASSAY | Myocardial Infarction - diagnosis | Myocardial Infarction - blood | Risk Assessment - methods | Emergency Service, Hospital | Prognosis | Humans | Middle Aged | Male | Biomarkers - blood | Troponin I - blood | Sensitivity and Specificity | Troponin - blood | Adult | Female | Aged | Cohort Studies | Care and treatment | Health risk assessment | Analysis | Heart attack | Myocardial infarction | Heart attacks | Clinical trials | Probability | Patients | Troponin T | Studies | Troponin | Pain | Troponin I | Calcium-binding protein | Electrocardiography | Population | Acute coronary syndromes | Cardiovascular diseases | Sampling | Index Medicus | Abridged Index Medicus
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13. Full Text A Case of Severe Ebola Virus Infection Complicated by Gram-Negative Septicemia
by Kreuels, Benno and Wichmann, Dominic and Emmerich, Petra and Schmidt-Chanasit, Jonas and de Heer, Geraldine and Kluge, Stefan and Sow, Abdourahmane and Renné, Thomas and Günther, Stephan and Lohse, Ansgar W and Addo, Marylyn M and Schmiedel, Stefan
The New England Journal of Medicine, ISSN 0028-4793, 12/2014, Volume 371, Issue 25, pp. 2394 - 2401
A relief worker who became infected with the Ebola virus while working in Sierra Leone during the largest recorded Ebola epidemic was evacuated to Hamburg,... 
MEDICINE, GENERAL & INTERNAL | Fluid Therapy | Humans | Bacteremia - drug therapy | Anti-Infective Agents - therapeutic use | Gram-Negative Bacterial Infections - drug therapy | Male | RNA, Viral - blood | Sierra Leone | Ebolavirus - genetics | Viral Load | Ebolavirus - isolation & purification | Hemorrhagic Fever, Ebola - therapy | Diarrhea - therapy | Hepatitis B, Chronic - complications | Bacteremia - etiology | Adult | Critical Care | Diarrhea - etiology | Gram-Negative Bacterial Infections - etiology | Hemorrhagic Fever, Ebola - complications | Parenteral Nutrition | Complications and side effects | Ebola virus infections | Respiratory insufficiency | Septicemia | Encephalopathy | Risk factors | Ebola virus | Urine | Bacterial infections | Medical treatment | Ribonucleic acid--RNA | Sweat
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14. Full Text Factor XII: a drug target for safe interference with thrombosis and inflammation
by Kenne, Ellinor and Renné, Thomas
Drug Discovery Today, ISSN 1359-6446, 09/2014, Volume 19, Issue 9, pp. 1459 - 1464
Data from experimental animal models revealed an essential role for factor XII (FXII) in thrombotic occlusive diseases. In contrast to other blood coagulation... 
COAGULATION-FACTOR-XII | PLATELET POLYPHOSPHATES | HEAVY-CHAIN | BLOOD-COAGULATION | HEREDITARY ANGIOEDEMA | IN-VIVO | HAGEMAN-FACTOR | PHARMACOLOGY & PHARMACY | MONOCLONAL-ANTIBODY | KININOGEN BINDING-SITE | CONTACT ACTIVATION | Thrombosis - physiopathology | Humans | Antibodies - adverse effects | Anticoagulants - adverse effects | Heparin - adverse effects | Antibodies - pharmacology | Animals | Factor XIIa - antagonists & inhibitors | Inflammation - drug therapy | Anticoagulants - pharmacology | Factor XII - antagonists & inhibitors | Thrombosis - drug therapy | Hemorrhage - chemically induced | Extracorporeal Membrane Oxygenation - methods | Heparin - pharmacology | Disease Models, Animal | Inflammation - physiopathology | Drug therapy | Thrombosis | Blood clot
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