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Cancer Research, ISSN 0008-5472, 10/2014, Volume 74, Issue 19 Supplement, pp. 4763 - 4763
Journal Article
Nature, ISSN 0028-0836, 03/2015, Volume 519, Issue 7541, pp. 102 - 105
The BCR-ABL1 fusion gene is a driver oncogene in chronic myeloid leukaemia and 30-50% of cases of adult acute lymphoblastic leukaemia(1). Introduction of ABL1... 
CELLS | PONATINIB | MECHANISM | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | RESISTANCE | FOLLOW-UP | PATIENTS RECEIVING IMATINIB | BCR-ABL INHIBITOR | CHRONIC MYELOID-LEUKEMIA | T315I MUTANT | Proto-Oncogene Proteins c-abl - antagonists & inhibitors | Humans | Molecular Conformation | Imidazoles - chemistry | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Crystallography, X-Ray | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Protein Kinase Inhibitors - chemistry | Angiogenesis Inhibitors - therapeutic use | Imidazoles - therapeutic use | Phosphorylation - drug effects | Fusion Proteins, bcr-abl - chemistry | Cell Line | Proto-Oncogene Proteins c-abl - genetics | Indazoles - chemistry | Crystallization | Proto-Oncogene Proteins c-abl - chemistry | Angiogenesis Inhibitors - pharmacology | Models, Molecular | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Imidazoles - pharmacology | Drug Repositioning | Indazoles - pharmacology | Drug Resistance, Neoplasm - genetics | Fusion Proteins, bcr-abl - genetics | Vascular Endothelial Growth Factor Receptor-2 - chemistry | Protein Kinase Inhibitors - therapeutic use | Fusion Proteins, bcr-abl - antagonists & inhibitors | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Protein Binding | Proto-Oncogene Proteins c-abl - metabolism | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Kidney Neoplasms - drug therapy | Fusion Proteins, bcr-abl - metabolism | Indazoles - therapeutic use | Angiogenesis Inhibitors - chemistry | Drug Screening Assays, Antitumor | Axitinib | Genetic aspects | Research | Gene mutations | Drug resistance | Analysis | Phosphorylation | Inhibitor drugs | Leukemia | Bone marrow | Mutation | Kinases | Drug dosages | Patients | Index Medicus
Journal Article
Trends in Cell Biology, ISSN 0962-8924, 2004, Volume 14, Issue 11, pp. 639 - 647
Journal Article
Cancer Research, ISSN 0008-5472, 04/2010, Volume 70, Issue 8 Supplement, pp. 3129 - 3129
Journal Article
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 676 - 676
Journal Article
Cancer Research, ISSN 0008-5472, 07/2004, Volume 64, Issue 13, pp. 4585 - 4592
Journal Article
2006, Methods in Enzymology, ISBN 9780121828127, Volume 407, 15
(Ras‐related and estrogen‐regulated growth inhibitor), a gene that encodes a small GTP binding and hydrolyzing protein (GTPase) of the Ras superfamily, was... 
RAS-RELATED GTPASES | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | BREAST CARCINOMAS | BIOCHEMICAL RESEARCH METHODS | SUPERFAMILY | RIT | FAMILY | MEMBER | Amino Acid Sequence | Gene Expression | Sequence Alignment | Animals | GTP Phosphohydrolases - metabolism | GTP Phosphohydrolases - genetics | Humans | Molecular Sequence Data | Genes, Tumor Suppressor
Book Chapter
2006, Proteins and Cell Regulation, ISBN 1402043287, Volume 4
Ras proteins activate cytoplasmic signaling cascades that mediate responses in growth, cellular differentiation, and survival. Therefore, it is not surprising... 
Biomedicine | Human Physiology | Cancer Research | Molecular Medicine | Pharmacology/Toxicology | Cell Biology
Book Chapter
Book Chapter
2010, ISBN 0123741459, Volume 2
This chapter summarizes the general features of R-Ras proteins that are shared with Ras proteins, and then highlights unique features of each R-Ras protein.... 
Book Chapter
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