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The New England Journal of Medicine, ISSN 0028-4793, 03/2017, Volume 376, Issue 9, pp. 848 - 855
Journal Article
Medecine/Sciences, ISSN 0767-0974, 05/2017, Volume 33, Issue 5, pp. 463 - 464
Journal Article
Nature Medicine, ISSN 1078-8956, 2014, Volume 20, Issue 4, pp. 398 - 407
Journal Article
Human Gene Therapy, ISSN 1043-0342, 02/2016, Volume 27, Issue 2, pp. 148 - 165
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 04/2018, Volume 378, Issue 16, pp. 1479 - 1493
Journal Article
Medecine sciences : M/S, 05/2017, Volume 33, Issue 5, p. 463
Journal Article
British Journal of Haematology, ISSN 0007-1048, 10/2017, Volume 179, Issue 2, p. 323
Summary In African-American patients with sickle cell disease (SCD), APOL1 G1 and G2 variants are associated with increased risk of sickle cell nephropathy... 
Tropism | Epidermal growth factor receptors | Complications | Genes | GSTM1 protein | Patients | GSTT1 protein | Glomerular filtration rate | Nephropathy | Glutathione transferase | Sickle cell disease | Genotypes | Age | Proteinuria | Kidney transplantation
Journal Article
Haematologica, ISSN 0390-6078, 05/2019, p. haematol.2018.213462
The most common treatment for patients with sickle cell disease is the chemotherapeutic, hydroxyurea, a therapy with pleiotropic effects, including increasing... 
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 514, Issue 7521, pp. 242 - 246
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4107 - 4121
Granulomatosis with polyangiitis (GPA) is a systemic necrotizing vasculitis that is associated with granulomatous inflammation and the presence of... 
PLASMACYTOID DENDRITIC CELLS | WEGENERS-GRANULOMATOSIS | MEDICINE, RESEARCH & EXPERIMENTAL | ANTINEUTROPHIL CYTOPLASMIC AUTOANTIBODY | HUMAN NEUTROPHILS | TGF-BETA | MACROPHAGE PHAGOCYTOSIS | ACTIVATED NEUTROPHILS | ANCA-ASSOCIATED VASCULITIS | NECROSIS-FACTOR-ALPHA | MEMORY T-CELLS | Granulocyte Colony-Stimulating Factor - physiology | T-Lymphocyte Subsets - immunology | Receptors, Interleukin-1 Type I - physiology | Dendritic Cells - immunology | Humans | Antibodies, Antineutrophil Cytoplasmic - immunology | Peritonitis - pathology | Granulomatosis with Polyangiitis - immunology | Granulomatosis with Polyangiitis - pathology | Myeloblastin - biosynthesis | Neutrophils - pathology | Granulomatosis with Polyangiitis - enzymology | Macrophages - physiology | Lung - pathology | Neutrophils - enzymology | Signal Transduction | Myeloblastin - immunology | Mice, Inbred C57BL | Cytokines - secretion | Neutrophils - immunology | Cellular Microenvironment | Myeloblastin - physiology | Nitric Oxide - physiology | Peritonitis - immunology | Autoantigens - physiology | Cell Membrane - enzymology | Animals | Myeloid Differentiation Factor 88 - deficiency | Autoantigens - immunology | Myeloid Differentiation Factor 88 - physiology | Mice | Apoptosis - physiology | Phagocytosis | Proteins | Ethics | Hypotheses | Consent | Rodents | Cloning | Inflammation | Kinases | Experiments | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Biology of Blood and Marrow Transplantation, ISSN 1083-8791, 03/2019, Volume 25, Issue 3, pp. S64 - S65
[beta]-globin gene transfer may reduce or eliminate complications in patients with sickle cell disease (SCD). LentiGlobin gene therapy (GT) comprises drug... 
Hemoglobin | Care and treatment | Sickle cell anemia | Gene therapy | Genes | Hematopoietic stem cells
Journal Article
BRITISH JOURNAL OF HAEMATOLOGY, ISSN 0007-1048, 10/2017, Volume 179, Issue 2, pp. 323 - 335
In African-American patients with sickle cell disease (SCD), APOL1 G1 and G2 variants are associated with increased risk of sickle cell nephropathy (SCN). To... 
sickle cell nephropathy | PROTEIN | PULMONARY-HYPERTENSION | RENAL-FUNCTION | ADULTS | GENETIC-VARIANTS | HAPTOGLOBIN | sickle cell disease | CHILDREN | ANEMIA | APOL1 | RISK VARIANTS | HEMATOLOGY | PROGRESSION
Journal Article
17.