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Journal of Biological Chemistry, ISSN 0021-9258, 02/2015, Volume 290, Issue 9, pp. 5470 - 5483
Despite the effectiveness of surgery or radiation therapy for the treatment of early-stage prostate cancer (PCa), there is currently no effective strategy for... 
DOUBLE-STRANDED-RNA | RECOGNITION | VIRUS INFECTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | RIG-I | ACID/CATIONIC LIPOSOME COMPLEX | NF-KAPPA-B | ADAPTER PROTEIN | UP-REGULATION | BETA | Poly I-C - genetics | Prostatic Neoplasms - metabolism | Humans | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Male | Interferon Regulatory Factor-3 - genetics | Interferon-Induced Helicase, IFIH1 | STAT1 Transcription Factor - metabolism | Prostatic Neoplasms - genetics | Transfection | RNA Interference | Adjuvants, Immunologic - metabolism | Interferon-beta - genetics | Toll-Like Receptor 3 - genetics | DEAD-box RNA Helicases - metabolism | Prostatic Neoplasms - pathology | Ubiquitin-Protein Ligases - metabolism | Receptors, Cell Surface - metabolism | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | STAT1 Transcription Factor - genetics | Toll-Like Receptor 3 - metabolism | Blotting, Western | DEAD-box RNA Helicases - genetics | Microscopy, Confocal | Interferon-beta - metabolism | Poly I-C - metabolism | Proto-Oncogene Proteins pp60(c-src) - genetics | Proto-Oncogene Proteins pp60(c-src) - metabolism | Cell Line, Tumor | Interferon Regulatory Factor-3 - metabolism | Androgens - metabolism | Ubiquitin-Protein Ligases - genetics | Receptors, Cell Surface - genetics | Prostate Cancer | Signal Transduction | Toll-like Receptor (TLR) | Double-stranded RNA (dsRNA) | Endosome | Innate Immunity
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2013, Volume 110, Issue 24, pp. 9812 - 9817
Toll-like receptor 3 (TLR3) is a key effector of the innate immune system against viruses. Activation of TLR3 exerts an antitumoral effect through a mechanism... 
MicroRNA | Cell lines | Toll like receptors | Small interfering RNA | Breast cancer | Prostate cancer | Prostate | Tumors | Apoptosis | Cancer | Combined chemotherapy | Inflammation | CELLS | APOPTOSIS | METHYLATION | STIMULATION | MULTIDISCIPLINARY SCIENCES | ALPHA | combined chemotherapy | FEATURES | BREAST-CANCER | inflammation | PROSTATE-CANCER | GENE-EXPRESSION | PROMOTER | Neoplasms - metabolism | Apoptosis - drug effects | Humans | Apoptosis - genetics | Immunoblotting | Male | Receptors, Retinoic Acid - genetics | DNA (Cytosine-5-)-Methyltransferases - metabolism | DNA Methylation | Prostatic Neoplasms - genetics | Neoplasms - genetics | Female | Toll-Like Receptor 3 - genetics | Gene Expression Regulation, Neoplastic - drug effects | Prostatic Neoplasms - prevention & control | Tretinoin - pharmacology | Prostatic Neoplasms - pathology | Poly I-C - pharmacology | Receptors, Retinoic Acid - metabolism | Toll-Like Receptor 3 - agonists | Reverse Transcriptase Polymerase Chain Reaction | Toll-Like Receptor 3 - metabolism | Xenograft Model Antitumor Assays | DNA (Cytosine-5-)-Methyltransferases - genetics | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Cell Line, Tumor | Mice | MicroRNAs - genetics | Neoplasms - pathology | Toll-Like Receptor 3 | Gene Expression Regulation, Neoplastic | Breast Neoplasms | Neoplasms | Life Sciences | Prostatic Neoplasms | DNA (Cytosine-5-)-Methyltransferase | MicroRNAs | Receptors, Retinoic Acid | Poly I-C | Tretinoin | Biological Sciences
Journal Article
Journal Article
Journal Article
Biology of Reproduction, ISSN 0006-3363, 10/2008, Volume 79, Issue 4, pp. 766 - 775
Journal Article
Neoplasia, ISSN 1476-5586, 07/2010, Volume 12, Issue 7, pp. 539 - 549
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2013, Volume 17, Issue 6, pp. 713 - 722
Toll‐Like receptors (TLRs) are a family of evolutionary conserved transmembrane proteins that recognize highly conserved molecules in pathogens. TLR‐expressing... 
cancer therapy | clinical trials | TLR | prostate infections | innate immunity | prostate carcinoma | Prostate infections | Clinical trials | Prostate carcinoma | Innate immunity | Cancer therapy | MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNE-RESPONSE | TOLL-LIKE-RECEPTOR-4 | TUMOR-CELLS | TNF-ALPHA PRODUCTION | CUTTING EDGE | CELL BIOLOGY | PHASE-II TRIAL | EPITHELIAL-CELLS | INFLAMMATION | AGONIST | Prostatic Hyperplasia - pathology | Prostatic Hyperplasia - immunology | Humans | Male | Prostatic Hyperplasia - complications | Prostatic Neoplasms - immunology | Bacterial Infections - complications | Prostate - pathology | Prostate - immunology | Bacterial Infections - immunology | Prostate - drug effects | Prostatic Hyperplasia - drug therapy | Prostatic Neoplasms - drug therapy | Adaptive Immunity | Biomarkers - metabolism | Prostatic Neoplasms - pathology | Gene Expression | Signal Transduction | Bacterial Infections - drug therapy | Toll-Like Receptors - immunology | Clinical Trials as Topic | Immunity, Innate | Bacterial Infections - pathology | Toll-Like Receptors - genetics | Immunologic Factors - pharmacology | Care and treatment | Prostate cancer | Health aspects | Cancer | Therapy | Deregulation | Hyperplasia | Infections | Genomes | Kinases | Immunity | Inflammatory diseases | Anticancer properties | Proteins | Signal transduction | Receptors | Evolutionary conservation | Bacteria | Immune system | Antigens | Pathogens | Adenosine | Immune response | Cytokines | Dendritic cells | Organs | Priming | Inflammation | Adaptive immunity | Membrane proteins | Androgens | Biopsy | Diagnostic software | Antitumor activity | Ligands | In vivo methods and tests | Diagnostic systems | Mutation | Prostate | Apoptosis | Tumors | Immunologic Factors | Biological Markers | Prostatic Hyperplasia | Life Sciences | Prostatic Neoplasms | Toll-Like Receptors | Bacterial Infections | Reviews
Journal Article
Journal Article
Oncotarget, ISSN 1949-2553, 07/2018, Volume 9, Issue 56, pp. 30905 - 30918
In human prostate cancer (PCa), the neuroendocrine cells, expressing the prostate cancer stem cell (CSC) marker CD44, may be resistant to androgen ablation and... 
Alternative splicing | Invasiveness | Prostate stem cells | CD44
Journal Article
Journal Article
Journal Article