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abridged index medicus (2) 2
allergy (2) 2
allergy and immunology (2) 2
analysis (2) 2
animals (2) 2
asthma (2) 2
asthma - immunology (2) 2
cells (2) 2
cytokines - metabolism (2) 2
immunology (2) 2
index medicus (2) 2
mice (2) 2
th2 cells - immunology (2) 2
adjuvants (1) 1
airway management (1) 1
allergen (1) 1
allergens (1) 1
allergic airway inflammation (1) 1
allergic sensitization (1) 1
allergies (1) 1
animal models (1) 1
antigens (1) 1
antigens, dermatophagoides - immunology (1) 1
antigens, fungal - immunology (1) 1
aspergillus fumigatus - immunology (1) 1
asthma - chemically induced (1) 1
asthma in children (1) 1
atopy (1) 1
cell line (1) 1
cells, cultured (1) 1
cellular signal transduction (1) 1
childhood asthma (1) 1
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circuit components (1) 1
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dendritic cells - immunology (1) 1
disease susceptibility (1) 1
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dust (1) 1
environment (1) 1
environmental exposure - adverse effects (1) 1
environmental factors (1) 1
exposure (1) 1
female (1) 1
females (1) 1
fetuses (1) 1
fibroblasts - immunology (1) 1
gene expression (1) 1
gene expression regulation (1) 1
gene-environment interactions (1) 1
gene-expression profiles (1) 1
genetic transcription (1) 1
house dust (1) 1
human subjects (1) 1
humans (1) 1
il-13 (1) 1
il-17a (1) 1
immune response (1) 1
immunoglobulins (1) 1
inflammation (1) 1
influence (1) 1
interleukin-13 - metabolism (1) 1
interleukin-13 receptor alpha2 subunit - genetics (1) 1
interleukin-13 responses (1) 1
interleukin-17 - metabolism (1) 1
life (1) 1
lung - immunology (1) 1
lymphocytes b (1) 1
lymphocytes t (1) 1
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male (1) 1
maternal exposure - adverse effects (1) 1
maternal exposures (1) 1
messenger-rna stabilization (1) 1
mice, inbred balb c (1) 1
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1. Full Text Maternal house dust mite exposure during pregnancy enhances severity of house dust mite–induced asthma in murine offspring
by Richgels, Phoebe K., MS and Yamani, Amnah, MS and Chougnet, Claire A., PhD and Lewkowich, Ian P., PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2017, Volume 140, Issue 5, pp. 1404 - 1415.e9
Background Atopic status of the mother and maternal exposure to environmental factors are associated with increased asthma risk. Moreover, animal models... 
Allergy and Immunology | allergen | maternal exposures | environment | pregnancy | Asthma | CELLS | SUSCEPTIBILITY | RISK | IMMUNOLOGY | GENE-ENVIRONMENT INTERACTIONS | TOTAL IGE | CHILDHOOD ASTHMA | CHILDREN | TRANSMISSION | ALLERGY | ALLERGIC SENSITIZATION | LIFE | Disease Susceptibility | Cytokines - metabolism | Dendritic Cells - immunology | Cells, Cultured | Male | Antigens, Fungal - immunology | Th2 Cells - immunology | Mice, Inbred Strains | Environmental Exposure - adverse effects | Pregnancy | Antigens, Dermatophagoides - immunology | Animals | Prenatal Exposure Delayed Effects - immunology | Aspergillus fumigatus - immunology | Asthma - immunology | Maternal Exposure - adverse effects | Female | Mice | Lung - immunology | Pyroglyphidae - immunology | Allergens | Dendritic cells | Pregnant women | Analysis | Animal models | Mites | Lymphocytes T | Macrophages | Atopy | Phagocytes | Rodents | Respiratory tract diseases | Antigens | Immunoglobulins | Immune response | Fetuses | House dust | Adjuvants | Exposure | Environmental factors | Allergies | Children & youth | Progeny | Sensitivity | Dust | Offspring | Disease transmission | Lymphocytes B | Influence | Females
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2. Full Text IL-17A enhances IL-13 activity by enhancing IL-13–induced signal transducer and activator of transcription 6 activation
by Hall, Sara L., MS and Baker, Theresa, MS and Lajoie, Stephane, PhD and Richgels, Phoebe K., MS and Yang, Yanfen, MS and McAlees, Jaclyn W., PhD and van Lier, Adelaide and Wills-Karp, Marsha, PhD and Sivaprasad, Umasundari, PhD and Acciani, Thomas H., PhD and LeCras, Timothy D., PhD and Myers, Jocelyn Biagini, PhD and Kovacic, Melinda Butsch, MPH, PhD and Lewkowich, Ian P., PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 2, pp. 462 - 471.e14
Background Increased IL-17A production has been associated with more severe asthma; however, the mechanisms whereby IL-17A can contribute to IL-13–driven... 
Allergy and Immunology | cytokines | IL-13 | IL-17A | signal transduction | Asthma | signal transducer and activator of transcription 6 | SEVERE ASTHMA | MESSENGER-RNA STABILIZATION | GENE-EXPRESSION PROFILES | ALLERGIC AIRWAY INFLAMMATION | DELTA-T-CELLS | IMMUNOLOGY | COMPLEMENT COMPONENT C3 | RECEPTOR ALPHA-2 | NEGATIVE REGULATOR | SMOOTH-MUSCLE | ALLERGY | INTERLEUKIN (IL)-13 RESPONSES | Cell Line | Cytokines - metabolism | Signal Transduction | Humans | Gene Expression Regulation | Receptors, Interleukin-17 - genetics | STAT6 Transcription Factor - genetics | Th2 Cells - immunology | STAT6 Transcription Factor - metabolism | Interleukin-13 - metabolism | Asthma - chemically induced | Mice, Knockout | Interleukin-17 - metabolism | Animals | Pneumonia - chemically induced | Asthma - immunology | Interleukin-13 Receptor alpha2 Subunit - genetics | Pneumonia - immunology | Fibroblasts - immunology | Mice | Mice, Inbred BALB C | Circuit components | Asthma in children | Genetic transcription | Cellular signal transduction | Analysis | Cells | Proteins | Studies | Human subjects | Pathology | Airway management | Cytokines | Pathogenesis | Smooth muscle | Inflammation | Gene expression
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