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Journal Article
PLoS ONE, ISSN 1932-6203, 06/2018, Volume 13, Issue 6, pp. e0199371 - e0199371
Background Improving the knowledge of angiomyolipoma physiopathology might help in refining its pharmacological treatment. We investigated if angiomyolipoma... 
PATHOGENESIS | BREAST-CANCER CELLS | MATRIX METALLOPROTEINASES | METASTASIS | TUBEROUS SCLEROSIS COMPLEX | PULMONARY LYMPHANGIOLEIOMYOMATOSIS | MULTIDISCIPLINARY SCIENCES | EPIDERMAL-GROWTH-FACTOR | ESTROGEN | EPITHELIOID ANGIOMYOLIPOMA | FACTOR RECEPTOR | Kidney - surgery | Cell Proliferation - genetics | Estrogens - pharmacology | Kidney - pathology | Humans | Gene Expression Regulation, Neoplastic | Male | Angiomyolipoma - pathology | Wound Healing | Angiomyolipoma - genetics | Sequence Analysis, DNA | Cell Movement - genetics | Matrix Metalloproteinase 2 - genetics | Kidney - metabolism | Chemokine CXCL12 - genetics | Matrix Metalloproteinase 9 - genetics | Angiomyolipoma - surgery | Female | Primary Cell Culture | Angiomyolipoma - metabolism | Phenotype | Genetic aspects | Research | Diagnosis | Cell migration | Kidney cancer | Cell proliferation | Health sciences | Pathogenesis | Estrogen | Smooth muscle | mRNA | Metastasis | Kinases | Estradiol | Cell adhesion & migration | Receptors | Epidermal growth factor | Clonal deletion | Rodents | Deletion | Angiomyolipoma | Phenotypes | Wound healing | Genetic control | Incubation | Quantum dots | Organs | Pharmacology | Breast cancer | Tamoxifen | Patients | Gelatinase B | Gelatinase A | Pathology | TSC2 protein | Sex hormones | Hypotheses | Hospitals | Lungs | Womens health | Medical prognosis | Females | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 02/2019, Volume 116, Issue 6, pp. 2226 - 2231
Malignant pleural mesothelioma (MPM) is an aggressive malignancy associated with exposure to asbestos, with poor prognosis and no effective therapies. The... 
Growth hormone-releasing hormone | Malignant pleural mesothelioma | GHRH antagonists | GHRH receptor | malignant pleural mesothelioma | APOPTOSIS | DIAGNOSIS | ACTIVATION | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | LUNG-CANCER CELLS | P21 | PROMOTES SURVIVAL | UPDATE | growth hormone-releasing hormone | EXPRESSION | Lung Neoplasms - drug therapy | Mesothelioma - pathology | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Receptors, Neuropeptide - metabolism | Pleural Neoplasms - drug therapy | Pleural Neoplasms - metabolism | Receptors, Pituitary Hormone-Regulating Hormone - metabolism | Antineoplastic Agents - pharmacology | Receptors, Pituitary Hormone-Regulating Hormone - genetics | Pleural Neoplasms - pathology | Disease Models, Animal | Cell Survival - drug effects | Gene Expression | Receptors, Neuropeptide - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Growth Hormone-Releasing Hormone - genetics | Mesothelioma - drug therapy | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Growth Hormone-Releasing Hormone - metabolism | Animals | Mesothelioma - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Growth Hormone-Releasing Hormone - antagonists & inhibitors | Cell proliferation | Alternative splicing | Biotechnology | Reactive oxygen species | Asbestos | Matrix metalloproteinases | Lung cancer | Xenotransplantation | Mesothelioma | Antagonists | Malignancy | Cell adhesion & migration | Mitochondria | Rodents | Xenografts | Inhibition | Vascular endothelial growth factor | Growth factors | Cell survival | Insulin | Chemotherapy | Medical prognosis | Cell lines | Growth hormones | Growth hormone | Cell migration | Cancer | Apoptosis | Index Medicus | Biological Sciences
Journal Article
Cancer Letters, ISSN 0304-3835, 10/2017, Volume 405, pp. 38 - 45
Malignant pleural mesothelioma (MPM) is a rare, aggressive cancer caused by asbestos exposure. An inherited predisposition has been suggested to explain... 
Homologous recombination repair | Mesothelioma | Germline mutation | DNA repair genes | Asbestos exposure | POPULATION | PANCREATIC-CANCER | SUSCEPTIBILITY | RISK | CROCIDOLITE ASBESTOS | PREVALENCE | BREAST-CANCER | MELANOMA | CARCINOGENESIS | ONCOLOGY | BAP1 MUTATIONS | Lung Neoplasms - genetics | Asbestos - toxicity | Genetic Predisposition to Disease | Pleural Neoplasms - genetics | Humans | Middle Aged | Risk Factors | Male | Mesothelioma - genetics | DNA Repair - genetics | Ubiquitin Thiolesterase - genetics | Pleural Neoplasms - etiology | Carcinogens - toxicity | Environmental Exposure - adverse effects | Lung Neoplasms - etiology | Tumor Suppressor Proteins - genetics | Aged, 80 and over | Germ-Line Mutation | Female | Aged | Environmental Pollutants - toxicity | Mesothelioma - etiology | Biotechnology | Asbestos | Gene mutations | DNA damage | Genes | DNA | Genetic research | Development and progression | Genetic aspects | Disease susceptibility | DNA repair | Asbestos industry | Genomics | Homologous recombination | Homology | Genomes | Family medical history | Carcinogenesis | Ovarian cancer | FANCC protein | Carcinogens | Repair | Deoxyribonucleic acid--DNA | BRCA2 protein | BRCA1 protein | Melanoma | Breast cancer | Exposure | Patients | Studies | Pancreatic cancer | Mutation | Health risk assessment | Cancer | Tumors | Index Medicus
Journal Article
European Respiratory Journal, ISSN 0903-1936, 05/2017, Volume 49, Issue 5, pp. 1602006 - 1602006
Journal Article