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Journal of Investigative Dermatology, ISSN 0022-202X, 03/2012, Volume 132, Issue 3, pp. 882 - 886
The innate immune system must recognize and rapidly respond to microbial pathogens, providing a first line of host defense. This is accomplished through an... 
MYCOBACTERIUM-TUBERCULOSIS | ACTIVATION | PEPTIDOGLYCAN | RECOGNITION | ADJUVANT ACTIVITY | INFLAMMASOME | TOLL-LIKE RECEPTORS | SIGNALS | INDUCTION | ANTIMICROBIAL ACTIVITY | DERMATOLOGY | Immunity, Innate - immunology | Animals | Dermatitis - immunology | Infection - immunology | Humans | Immune System Diseases - immunology
Journal Article
Science, ISSN 0036-8075, 3/2013, Volume 339, Issue 6126, pp. 1448 - 1453
Type I interferons (IFN-α and IFN-β) are important for protection against many viral infections, whereas type II interferon (IFN-γ) is essential for host... 
Monocytes | Messenger RNA | Tuberculosis | Mycobacterium tuberculosis | Antimicrobials | REPORTS | Leprosy | Infections | Skin | Lesions | Gene expression | LISTERIA-MONOCYTOGENES | ACTIVATION | PROFILES | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | HUMAN TUBERCULOSIS | IL-1-BETA | INDUCTION | DEPENDENT ANTIMICROBIAL PATHWAY | SARCOIDOSIS | LEPROSY | Up-Regulation | Interferon-beta - immunology | Humans | Transcriptome | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - genetics | Monocytes - metabolism | Monocytes - immunology | Interferon-gamma - metabolism | Antimicrobial Cationic Peptides - metabolism | RNA, Messenger - metabolism | Receptors, Calcitriol - genetics | Mycobacterium leprae - immunology | Interferon-beta - genetics | Interleukin-10 - metabolism | beta-Defensins - metabolism | Interferon-gamma - genetics | Antimicrobial Cationic Peptides - genetics | Leprosy, Lepromatous - genetics | Mycobacterium leprae - physiology | Leprosy, Tuberculoid - immunology | beta-Defensins - genetics | Leprosy, Lepromatous - immunology | RNA, Messenger - genetics | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - metabolism | Microbial Viability | Receptors, Calcitriol - metabolism | Leprosy, Tuberculoid - genetics | Tuberculosis - immunology | Leprosy, Lepromatous - metabolism | Interferon-beta - metabolism | Tuberculosis - genetics | Leprosy, Tuberculoid - metabolism | Interferon-gamma - immunology | Interleukin-10 - genetics | Interleukins | Physiological aspects | Host-parasite relationships | Anti-infective agents | Interferon | Genetic aspects | Health aspects | Antimicrobial agents | Pathogenesis | Genes | Bacteria | Human behavior | Antiinfectives and antibacterials | Digital signatures
Journal Article
Journal of Bacteriology, ISSN 0021-9193, 02/2015, Volume 197, Issue 3, p. 615
  Langerhans cells participate in the immune response in leprosy by their ability to activate T cells that recognize the pathogen, Mycobacterium leprae, in a... 
Proteins | Bacteria | Glycoproteins | Glycosylation | T cell receptors | Mass spectrometry
Journal Article
Frontiers in Physiology, ISSN 1664-042X, 2014, Volume 5
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2015, Volume 112, Issue 50, pp. 15408 - 15413
Spontaneous CD8 T-cell responses occur in growing tumors but are usually poorly effective. Understanding the molecular and cellular mechanisms that drive these... 
Antitumor immunity | STING | Type I IFNs | Tumor endothelial cells | CD8 T cells | type I IFNs | DENDRITIC CELLS | RECOGNITION | MULTIDISCIPLINARY SCIENCES | SENSOR | antitumor immunity | I INTERFERON | 2ND-MESSENGER | tumor endothelial cells | RESPONSES | INTRACELLULAR DNA | MELANOMA | IMMUNOGENIC TUMORS | IPILIMUMAB | Dose-Response Relationship, Immunologic | Dendritic Cells - immunology | Immunity | Neoplasms - therapy | Melanoma, Experimental - immunology | Injections, Intralesional | Interferon Type I - metabolism | Dendritic Cells - drug effects | Membrane Proteins - metabolism | Disease Models, Animal | Receptor, Interferon alpha-beta - metabolism | Nucleotides, Cyclic - pharmacology | Antigens, Neoplasm - immunology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Melanoma, Experimental - pathology | Lymphocytes, Tumor-Infiltrating - drug effects | Melanoma - pathology | CTLA-4 Antigen - immunology | Animals | Signal Transduction - drug effects | Melanoma - immunology | Neoplasms - immunology | Cell Proliferation - drug effects | Nucleotides, Cyclic - administration & dosage | CD8-Positive T-Lymphocytes - immunology | Neoplasms - pathology | Endothelial Cells - drug effects | Lymphocytes, Tumor-Infiltrating - immunology | Growth | Cancer cells | Physiological aspects | Genetic aspects | T cells | Health aspects | Tumors | Endothelium | Biological Sciences
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2010, Volume 107, Issue 52, pp. 22593 - 22598
We investigated the mechanisms by which T-cell cytokines are able to influence the Toll-like receptor (TLR)-induced, vitamin D-dependent antimicrobial pathway... 
T lymphocytes | Up regulation | Monocytes | Messenger RNA | Cytokines | Vitamin D | Mycobacterium tuberculosis | Antimicrobials | Small interfering RNA | Physiological regulation | Interleukin-4 | Interferon-γ | Innate immune response | interleukin-4 | MYCOBACTERIUM-TUBERCULOSIS | ACTIVATION | CATHELICIDIN | IFN-GAMMA | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | AUTOPHAGY | CUTTING EDGE | TOLL-LIKE RECEPTOR-2 | innate immune response | MICROBIAL LIPOPROTEINS | interferon-gamma | Steroid Hydroxylases - metabolism | Gene Expression - drug effects | Monocytes - cytology | Humans | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - genetics | Monocytes - metabolism | Antimicrobial Cationic Peptides - metabolism | Th1 Cells - metabolism | Interleukin-4 - pharmacology | T-Lymphocytes - metabolism | Calcitriol - metabolism | beta-Defensins - metabolism | Antimicrobial Cationic Peptides - genetics | Steroid Hydroxylases - genetics | beta-Defensins - genetics | Toll-Like Receptor 1 - metabolism | Cells, Cultured | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - metabolism | Vitamin D3 24-Hydroxylase | Toll-Like Receptor 2 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Th2 Cells - metabolism | Blotting, Western | Monocytes - drug effects | Signal Transduction - drug effects | Vitamin D - analogs & derivatives | Vitamin D - metabolism | Cytokines - pharmacology | Interferon-gamma - pharmacology | Immune response | Calcifediol | Regulation | Alfacalcidol | Research | T cells | Properties | Health aspects | Biological Sciences | interferon-γ
Journal Article
Science, ISSN 0036-8075, 3/2006, Volume 311, Issue 5768, pp. 1770 - 1773
Journal Article