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Blood, ISSN 0006-4971, 12/2015, Volume 126, Issue 23, pp. 2939 - 2939
Abstract Introduction Targeted inhibitors of BTK, PI3K or BCL-2 all induce durable responses in patients with chemotherapy-refractory CLL. In the case of the... 
Journal Article
Cytokine, ISSN 1043-4666, 11/2014, Volume 70, Issue 1, p. 58
Here, we have characterized the first known auto-inflammatory disease caused by interleukin-18 (IL-18). This is due to a point mutation in the gene Wdr1, which... 
Journal Article
Lancet Oncology, The, ISSN 1470-2045, 2016, Volume 17, Issue 6, pp. 768 - 778
Journal Article
Blood, ISSN 0006-4971, 12/2014, Volume 124, Issue 21, pp. 1579 - 1579
Abstract Introduction The regulation of neutrophil lifespan is critical for a circumscribed immune response. Neutrophils are sensitive to Fas/CD95 death... 
Journal Article
Lancet Oncology, The, ISSN 1470-2045, 2017, Volume 18, Issue 2, pp. 230 - 240
Journal Article
BLOOD, ISSN 0006-4971, 06/2012, Volume 119, Issue 24, pp. 5807 - 5816
The BH3-mimetic ABT-737 and an orally bioavailable compound of the same class, navitoclax (ABT-263), have shown promising antitumor efficacy in preclinical and... 
MULTIPLE-MYELOMA | LUNG-CANCER | ANTAGONIST ABT-737 | FAMILY-MEMBERS | ACUTE LYMPHOBLASTIC-LEUKEMIA | APOPTOTIC RESPONSES | CHRONIC LYMPHOCYTIC-LEUKEMIA | BH3 MIMETIC ABT-737 | HEMATOLOGY | EXPRESSION | INHIBITOR ABT-737 | Leukemia - pathology | Humans | Molecular Targeted Therapy | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Biphenyl Compounds - therapeutic use | Proto-Oncogene Proteins c-bcl-2 - metabolism | Lymphoma - drug therapy | Bcl-2-Like Protein 11 | Biphenyl Compounds - pharmacology | Nitrophenols - pharmacology | bcl-X Protein - antagonists & inhibitors | Nitrophenols - therapeutic use | Protein Binding - drug effects | Cytoprotection - drug effects | Lymphoma - pathology | Membrane Proteins - metabolism | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Leukemia - genetics | Proto-Oncogene Proteins - metabolism | Aniline Compounds - pharmacology | bcl-2-Associated X Protein - metabolism | Etoposide - pharmacology | Leukemia - drug therapy | Lymphoma - genetics | Mutant Proteins - metabolism | Piperazines - therapeutic use | Sulfonamides - pharmacology | Piperazines - pharmacology | Apoptosis Regulatory Proteins - metabolism | Animals | Sulfonamides - therapeutic use | Myeloid Cell Leukemia Sequence 1 Protein | Apoptosis Regulatory Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Mice | Aniline Compounds - therapeutic use | bcl-X Protein - metabolism | Drug Resistance, Neoplasm - drug effects
Journal Article
Blood, ISSN 0006-4971, 11/2013, Volume 122, Issue 21, pp. 218 - 218
Abstract Introduction Cell death can be triggered by many stimuli leading to apoptosis, pyroptosis (Caspase-1-dependent cell death) or necroptosis... 
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 01/2016, Volume 374, Issue 4, pp. 311 - 322
An oral inhibitor of BCL2 produces substantial responses in the majority of patients with refractory chronic lymphocytic leukemia. The agent is so effective... 
DOSE-ESCALATION | APOPTOSIS | MEDICINE, GENERAL & INTERNAL | CLL | INTERNATIONAL WORKSHOP | CYCLOPHOSPHAMIDE | RITUXIMAB | IBRUTINIB | INHIBITOR | PHASE-I | FLUDARABINE | Recurrence | Humans | Middle Aged | Male | Bridged Bicyclo Compounds, Heterocyclic - administration & dosage | Antineoplastic Agents - administration & dosage | Remission Induction | Sulfonamides - pharmacokinetics | Dose-Response Relationship, Drug | Disease-Free Survival | Bridged Bicyclo Compounds, Heterocyclic - adverse effects | Tumor Lysis Syndrome - etiology | Antineoplastic Agents - adverse effects | Aged, 80 and over | Sulfonamides - adverse effects | Adult | Female | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Aged | Antineoplastic Agents - pharmacokinetics | Leukemia, Lymphocytic, Chronic, B-Cell - drug therapy | Sulfonamides - administration & dosage | Bridged Bicyclo Compounds, Heterocyclic - pharmacokinetics | Antimitotic agents | Treatment outcome | Care and treatment | Usage | Safety and security measures | Lymphocytic leukemia | Analysis | Clinical trials | Dosage and administration | Antineoplastic agents | Pharmacokinetics | Risk factors | Flow cytometry | Chronic lymphatic leukemia | Cell survival | Leukemia | Minimal residual disease | Diarrhea | Chromosome deletion | Nausea | Lymphatic leukemia | Lymphoma | Patients | Proteins | Chemotherapy | Fludarabine | Clonal deletion | Lysis | Respiratory tract diseases | Lymphomas | Chromosome 17 | Neutropenia | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2016, Volume 538, Issue 7626, pp. 477 - 482
Avoidance of apoptosis is critical for the development and sustained growth of tumours. The pro-survival protein myeloid cell leukemia 1 (MCL1) is... 
MULTIPLE-MYELOMA | SURVIVAL | ANTI-APOPTOTIC MCL-1 | PROTEIN | DEPENDENCY | BCL-XL | MULTIDISCIPLINARY SCIENCES | HIGH-AFFINITY | TUMOR-CELLS | COMBINATION | NAVITOCLAX | Neoplasms - metabolism | Thiophenes - therapeutic use | Leukemia - pathology | Apoptosis - drug effects | Humans | Myeloid Cell Leukemia Sequence 1 Protein - metabolism | Male | Antineoplastic Agents - therapeutic use | Leukemia - metabolism | Thiophenes - administration & dosage | Antineoplastic Agents - administration & dosage | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Lymphoma - metabolism | Lymphoma - drug therapy | Multiple Myeloma - drug therapy | Myeloid Cell Leukemia Sequence 1 Protein - chemistry | Female | Lymphoma - pathology | Antineoplastic Agents - pharmacology | Myeloid Cell Leukemia Sequence 1 Protein - antagonists & inhibitors | Pyrimidines - administration & dosage | bcl-2-Associated X Protein - metabolism | Leukemia - drug therapy | Models, Molecular | Thiophenes - pharmacology | Pyrimidines - pharmacology | Multiple Myeloma - metabolism | Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Multiple Myeloma - pathology | Animals | Models, Biological | Pyrimidines - therapeutic use | Cell Line, Tumor | Mice | Neoplasms - pathology | Myelocytic leukemia | Physiological aspects | Nonlymphoid leukemia | Metastasis | Observations | Health aspects | Apoptosis | Proteins | Studies | Multiple myeloma | Cytotoxicity | Kinases | Drug dosages | Tumors | Cancer | Crystal structure
Journal Article
Nature Genetics, ISSN 1061-4036, 03/2013, Volume 45, Issue 3, pp. 242 - 252
The genetic basis of hypodiploid acute lymphoblastic leukemia (ALL), a subtype of ALL characterized by aneuploidy and poor outcome, is unknown. Genomic... 
P53 MUTATIONS | NEUROFIBROMATOSIS TYPE-1 GENE | TRANSMEMBRANE ADAPTER | IKAROS | GENETICS & HEREDITY | JUVENILE MYELOMONOCYTIC LEUKEMIA | REGULATORY T-CELLS | MUTANT P53 | HUMAN CANCERS | HEMATOLOGIC MALIGNANCIES | CHILDHOOD | Humans | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Aneuploidy | Transplantation, Heterologous | Phosphatidylinositol 3-Kinases - metabolism | Tumor Suppressor Protein p53 - genetics | Ikaros Transcription Factor - genetics | Haploidy | Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Base Sequence | Ikaros Transcription Factor - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Signal Transduction | Retinoblastoma Protein - metabolism | Tumor Suppressor Protein p53 - metabolism | Treatment Outcome | Phosphatidylinositol 3-Kinases - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Animals | Retinoblastoma Protein - genetics | Chromosome Aberrations | Cell Line, Tumor | Mice | Mutation | Care and treatment | Genomics | Physiological aspects | Genetic aspects | Research | Acute lymphocytic leukemia | Cancer | Medical research | Pediatrics | Cloning | Genetics | Genomes | Kinases | Chromosomes | Medicinsk genetik | Basic Medicine | Medical Genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article