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PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9313 - e9313
Background: Lewy body disease is a heterogeneous group of neurodegenerative disorders characterized by alpha-synuclein accumulation that includes dementia with... 
WILD-TYPE | CHAPERONE-MEDIATED AUTOPHAGY | ALZHEIMERS-DISEASE | MOUSE MODEL | FRONTOTEMPORAL DEMENTIA | BIOLOGY | AGGREGATE-PRONE PROTEINS | NEURONAL EXPRESSION | PARKINSONS-DISEASE | TRANSGENIC MICE | GAUCHER-DISEASE | Pyramidal Cells - metabolism | Humans | Nerve Degeneration - genetics | Male | Autophagy | Nerve Degeneration - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Lewy Body Disease - genetics | RNA Interference | Brain - ultrastructure | Aged, 80 and over | Female | alpha-Synuclein - genetics | Pyramidal Cells - ultrastructure | Disease Models, Animal | Cell Line | Ubiquitin-Activating Enzymes - genetics | Signal Transduction | Lewy Body Disease - pathology | Mice, Transgenic | Microscopy, Electron | Ubiquitin-Activating Enzymes - metabolism | Blotting, Western | Nerve Degeneration - pathology | Lewy Body Disease - metabolism | Microscopy, Confocal | Autophagy-Related Protein 7 | Animals | Alzheimer Disease - metabolism | Brain - pathology | Cell Line, Tumor | Pyramidal Cells - pathology | Aged | Mice | Alzheimer Disease - genetics | alpha-Synuclein - metabolism | TOR protein | Neurosciences | Animal models | Parkinson's disease | Pathogenesis | Toxicity | Parkinsons disease | Phagosomes | Nervous system | Synuclein | Neuromodulation | Accumulation | Modulators | Proteins | Infusion | Alterations | Neurodegeneration | Dementia disorders | Alzheimer's disease | Movement disorders | Enzymes | Neurodegenerative diseases | Neurons | Immunomodulation | Lewy body disease | Transgenic mice | Rapamycin | Patients | Lewy bodies | Studies | Pathology | Mutation | Alzheimers disease | Phagocytosis | Apoptosis | Dementia | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, pp. e19338 - e19338
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 2014, Volume 127, Issue 6, pp. 861 - 879
Immunotherapeutic approaches are currently in the spotlight for their potential as disease-modifying treatments for neurodegenerative disorders. The discovery... 
Parkinson's disease | AFFITOME | Immunotherapy | Vaccination | Alpha-synuclein | AFFITOPE | HUMAN ALPHA-SYNUCLEIN | ALZHEIMERS-DISEASE | PATHOLOGY | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | CLINICAL NEUROLOGY | EXPLORATORY ACTIVITY | AFFITOME (R) | A-BETA | MICE | MOTOR COORDINATION | LEWY-BODY-DISEASE | TRANSGENIC MOUSE MODEL | AFFITOPE (R) | Parkinson Disease - therapy | Memory Disorders - physiopathology | Motor Activity - physiology | T-Lymphocytes - physiology | Putamen - pathology | Humans | Caudate Nucleus - pathology | Nerve Degeneration - physiopathology | Axons - physiology | Synapses - pathology | Caudate Nucleus - physiopathology | Lewy Body Disease - therapy | Microglia - physiology | Vaccination - methods | Putamen - physiopathology | Microglia - pathology | alpha-Synuclein - genetics | T-Lymphocytes - pathology | Memory Disorders - pathology | Disease Models, Animal | Parkinson Disease - pathology | Synapses - physiology | Mice, Inbred C57BL | Lewy Body Disease - pathology | Antibodies - cerebrospinal fluid | Mice, Transgenic | Clinical Trials as Topic | Parkinson Disease - physiopathology | Nerve Degeneration - pathology | Animals | Axons - pathology | Memory Disorders - therapy | Antibodies - blood | alpha-Synuclein - metabolism | Lewy Body Disease - physiopathology | Nerve Degeneration - therapy | Oligomers | Platelet-derived growth factor | Peptides | Clinical trials | Genetic engineering | T cells | Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 10/2005, Volume 8, Issue 10, pp. 1343 - 1349
In Alzheimer disease, increased beta-secretase (BACE1) activity has been associated with neurodegeneration and accumulation of amyloid precursor protein (APP)... 
RNA INTERFERENCE | CLEAVING ENZYME | BETA-SECRETASE ACTIVITY | CARBOXYL-TERMINAL FRAGMENT | IN-VIVO | LENTIVIRAL VECTOR | MICE | AMYLOID PRECURSOR PROTEIN | EXPRESSION | NEUROSCIENCES | BRAIN | Microtubule-Associated Proteins - metabolism | Humans | Lentivirus - physiology | Brain - virology | Molecular Sequence Data | Glial Fibrillary Acidic Protein - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Endopeptidases - chemistry | Time Factors | Aspartic Acid Endopeptidases | Amyloid Precursor Protein Secretases | Amyloid beta-Protein Precursor - metabolism | Cloning, Molecular | Spatial Behavior - drug effects | Disease Models, Animal | Cell Line | Endopeptidases - metabolism | Maze Learning - physiology | Alzheimer Disease - drug therapy | Gene Expression Regulation - physiology | Mice, Transgenic | Maze Learning - drug effects | Brain - drug effects | Gene Expression Regulation - drug effects | Immunohistochemistry - methods | Amyloid beta-Protein Precursor - genetics | Animals | Endopeptidases - genetics | Analysis of Variance | RNA, Small Interfering - therapeutic use | Alzheimer Disease - metabolism | Brain - pathology | Genetic Vectors - physiology | Mice | Alzheimer Disease - genetics | Neurons | Physiological aspects | Genetic aspects | Diagnosis | Research | Gene expression | Alzheimer's disease | Risk factors | Index Medicus
Journal Article
Journal Article
Science, ISSN 0036-8075, 2/2005, Volume 307, Issue 5713, pp. 1282 - 1288
Journal Article
Journal Article