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Gut, ISSN 0017-5749, 10/2017, Volume 66, Issue 10, pp. 1748 - 1760
ObjectiveEpidemiological and clinical data indicate that patients suffering from IBD with long-standing colitis display a higher risk to develop colorectal... 
COLORECTAL CANCER | IBD | CELL MATRIX INTERACTION | INTEGRINS | INTESTINAL BARRIER FUNCTION | CELLS | ALPHA-6-BETA-4 INTEGRIN | INFLAMMATION | ABSENCE | EPIDERMOLYSIS-BULLOSA | INTESTINAL BARRIER | GASTROENTEROLOGY & HEPATOLOGY | TUMOR-GROWTH | EXPRESSION | PROMOTES | BETA | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Epithelial Cells - metabolism | Colitis - genetics | Colorectal Neoplasms - genetics | B-Lymphocytes | Hemidesmosomes - physiology | Caspase 1 - metabolism | Colitis - pathology | Hemidesmosomes - genetics | Adenocarcinoma - metabolism | Adenocarcinoma - physiopathology | Mucus - metabolism | Adenocarcinoma - genetics | Integrin alpha6beta4 - metabolism | Cytokines - genetics | Colorectal Neoplasms - metabolism | Adaptive Immunity | Severity of Illness Index | Cytokines - metabolism | Signal Transduction | T-Lymphocytes | Lymphocyte Activation | Myeloid Differentiation Factor 88 - genetics | Integrin alpha6 - genetics | Permeability | Colorectal Neoplasms - physiopathology | Basement Membrane - physiopathology | Animals | Colitis - physiopathology | Keratin-8 - metabolism | Keratin-18 - metabolism | Colitis - metabolism | Mice | Homeostasis - genetics | Intestinal Mucosa - pathology | Complications and side effects | Inflammatory bowel diseases | Antibiotics | Colorectal cancer | Dosage and administration | Research | Epidemiology | Risk factors | Adenocarcinoma | Transformation | Animal models | Epithelial cells | Hyperplasia | Colorectal carcinoma | Helper cells | Homeostasis | Genomes | Lymphocytes T | Small intestine | Defects | Metastases | Keratin | Cell activation | Laminin | Intestine | Hemidesmosomes | Tumorigenesis | Colon | Immune system | Dysplasia | Cytokines | Secretion | Inflammation | Patients | Interleukin 18 | Inflammatory bowel disease | Studies | Lymphocytes B | Colitis | Tumors | Life Sciences | Development Biology | Inflammatory Bowel Disease | 1506
Journal Article
BMC Genomics, ISSN 1471-2164, 10/2014, Volume 15, Issue 1, pp. 852 - 852
Journal Article
Frontiers in Genetics, ISSN 1664-8021, 2015, Volume 5, p. 470
Among a diversity of animal models of disease, the zebrafish is a promising model organism for enabling novel translational biomedical research. To fully... 
Orthology inference | zebrafish-to-human gene mapping | Genome annotation | Zebrafish | Translational research | DATABASE RESOURCES | CONNECTIVITY MAP | SEQUENCE | GENETICS & HEREDITY | NATIONAL CENTER | MODEL | BLAST | orthology inference | genome annotation
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2008, Volume 283, Issue 35, pp. 24212 - 24223
Journal Article
Scientific Reports, ISSN 2045-2322, 05/2016, Volume 6, Issue 1
The zebrafish has the capacity to regenerate its heart after severe injury. While the function of a few genes during this process has been studied, we are far... 
TARGET | MYOCARDIAL-INFARCTION | CRYOINJURY | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | INJURY | EPICARDIAL CELLS | CARDIOMYOCYTES | MODEL | DIFFERENTIATION | CARDIAC REGENERATION
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 08/2007, Volume 212, Issue 2, pp. 439 - 449
Mice lacking the α6 integrin chain die at birth with severe skin blistering. To further study the function of α6 integrin in skin, we generated conditionally... 
Journal Article
Scientific reports, 05/2016, Volume 6, p. 26822
The zebrafish has the capacity to regenerate its heart after severe injury. While the function of a few genes during this process has been studied, we are far... 
Journal Article
JOURNAL OF BIOLOGICAL CHEMISTRY, ISSN 0021-9258, 08/2008, Volume 283, Issue 35, pp. 24212 - 24223
Talin establishes a major link between integrins and actin filaments and contains two distinct integrin binding sites: one, IBS1, located in the talin head... 
VINCULIN-BINDING-SITE | ALPHA(IIB)BETA | DOMAIN | ACTIVATION | TYROSINE PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEINS | IDENTIFICATION | ACTININ BINDING | FOCAL ADHESIONS | BETA | Life Sciences | Biochemistry, Molecular Biology
Journal Article
Scientific Reports, ISSN 2045-2322, 2016, Volume 6, Issue 26822
The zebrafish has the capacity to regenerate its heart after severe injury. While the function of a few genes during this process has been studied, we are far... 
Journal Article
BMC Medical Genomics, ISSN 1755-8794, 12/2011, Volume 4, p. 83
Prediction of left ventricular (LV) remodeling after acute myocardial infarction (MI) is clinically important and would benefit from the discovery of new... 
Genetic markers | Usage | Care and treatment | Prognosis | Physiological aspects | Genetic aspects | Growth factor receptors | Research | Diagnosis | Heart attack | Proteins | Angiogenesis | Heart attacks | Angioplasty | Biomarkers | Data mining | Laboratory animals | Experiments | Manuscripts | Blood clots
Journal Article
Cardiogenetics, ISSN 2035-8253, 07/2011, Volume 1, Issue 1, pp. 5 - e5
Matrix metalloproteinase 9 (MMP9) is functionally implicated in the process of infarct healing. Several genetic variation of the MMP9 gene have been described,... 
matrix metalloproteinase 9, myocardial infarction, polymorphism, ventricular function
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2008, Volume 283, Issue 35, p. 24212
Talin establishes a major link between integrins and actin filaments and contains two distinct integrin binding sites: one, IBS1, located in the talin head... 
Journal Article
Journal of Cardiovascular Translational Research, ISSN 1937-5387, 12/2011, Volume 4, Issue 6, pp. 790 - 800
Recent evidence suggests that Toll-like receptor 4 (TLR4) is not only involved in innate immunity but is also an important mediator of adverse left ventricular... 
Biomedical Engineering | Human Genetics | Myocardial infarction | Adenosine | Medicine & Public Health | Toll-like receptor 4 | Inflammation | Cardiology | Left ventricular remodeling | Medicine/Public Health, general | Biomedicine general | MEDICINE, RESEARCH & EXPERIMENTAL | MATRIX-METALLOPROTEINASE-9 SECRETION | TOLL-LIKE-RECEPTOR-4 | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | A(2A) RECEPTORS | MYOCARDIAL-INFARCTION | HUMAN MACROPHAGES | NECROSIS-FACTOR-ALPHA | KAPPA-B | ISCHEMIA-REPERFUSION INJURY | TNF-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Myocardial Infarction - genetics | Humans | Middle Aged | Hyaluronic Acid - pharmacology | Male | RNA, Messenger - metabolism | Case-Control Studies | Myocardial Infarction - immunology | Dose-Response Relationship, Drug | Inflammation - metabolism | Adenosine A2 Receptor Agonists - pharmacology | Time Factors | Luxembourg | Receptor, Adenosine A2B - drug effects | Toll-Like Receptor 4 - agonists | Adult | Female | Registries | Cell Membrane - metabolism | Cell Membrane - drug effects | Macrophages - immunology | Heparitin Sulfate - pharmacology | Down-Regulation | Cells, Cultured | Toll-Like Receptor 4 - genetics | Inflammation - immunology | Myocardial Infarction - metabolism | Receptor, Adenosine A2A - metabolism | Toll-Like Receptor 4 - metabolism | Receptor, Adenosine A2B - metabolism | Receptor, Adenosine A2A - drug effects | Adenosine A2 Receptor Antagonists - pharmacology | Macrophages - metabolism | Lipopolysaccharides - pharmacology | Adenosine - metabolism | Inflammation - genetics | Ligands | Macrophages - drug effects | Aged | Cell Membrane - immunology
Journal Article
Journal of cellular physiology, ISSN 0021-9541, 08/2007, Volume 212, Issue 2, pp. 439 - 49
Journal Article