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21. Full Text Genetic associations at 53 loci highlight cell types and biological pathways relevant for kidney function
by Pattaro, Cristian and Teumer, Alexander and Gorski, Mathias and Chu, Auey Y and Li, Man and Mijatovic, Vladan and Garnaas, Maija and Tin, Aienne and Sorice, Rossella and Li, Yong and Taliun, Daniel and Olden, Matthias and Foster, Meredith and Yang, Qiong and Chen, Ming-Huei and Pers, Tune H and Johnson, Anew D and Ko, Yi-An and Fuchsberger, Christian and Tayo, Bamidele and Nalls, Michael and Feitosa, Mary F and Isaacs, Aaron and Dehghan, Abbas and d'Adamo, Pio and Adeyemo, Adebowale and Dieffenbach, Aida Karina and Zonderman, Alan B and Nolte, Ilja M and van der Most, Peter J and Wright, Alan F and Shuldiner, Alan R and Morrison, Alanna C and Hofman, Albert and Smith, Albert V and isbach, Albert W and Franke, Ane and Uitterlinden, Ane G and Metspalu, Anes and Tonjes, Anke and Lupo, Antonio and Robino, Antonietta and Johansson, Asa and Demirkan, Ayse and Kollerits, Barbara and Freedman, Barry I and Ponte, Belen and Oostra, Ben A and Paulweber, Bernhard and Kraemer, Bernhard K and Mitchell, Braxton D and Buckley, Brendan M and Peralta, Carmen A and Hayward, Caroline and Helmer, Catherine and Rotimi, Charles N and Shaffer, Christian M and Mueller, Christian and Sala, Cinzia and van Duijn, Cornelia M and Saint-Pierre, Aude and Ackermann, Daniel and Shriner, Daniel and Ruggiero, Daniela and Toniolo, Daniela and Lu, Yingchang and Cusi, Daniele and Czamara, Darina and Ellinghaus, David and Siscovick, David S and Ruderfer, Douglas and Gieger, Christian and Grallert, Harald and Rochtchina, Elena and Atkinson, Elizabeth J and Holliday, Elizabeth G and Boerwinkle, Eric and Salvi, Erika and Bottinger, Erwin P and Murgia, Federico and Rivadeneira, Fernando and Ernst, Florian and Kronenberg, Florian and Hu, Frank B and Navis, Gerjan J and Curhan, Gary C and Ehret, George B and Homuth, Georg and Coassin, Stefan and Thun, Gian-Ani and Pistis, Giorgio and Gambaro, Giovanni and Malerba, Giovanni and Montgomery, Grant W and Eiriksdottir, Gudny and Jacobs, Gunnar and Li, Guo and Wichmann, H-Erich and Campbell, Harry and Schmidt, Helena and ... and CARDIOGRAM and ICBP Consortium and AGEN Consortium and CHARGe-Heart Failure Grp and ECHOGen Consortium and CHARGe-Heart Failure Group and Medicinska fakulteten and Science for Life Laboratory, SciLifeLab and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för immunologi, genetik och patologi and Uppsala universitet and Medicinsk genetik och genomik
Nature Communications, ISSN 2041-1723, 01/2016, Volume 7, Issue 1, p. 10023
Reduced glomerular filtration rate defines chronic kidney disease and is associated with cardiovascular and all-cause mortality. We conducted a meta-analysis... 
SERUM CREATININE | INDIVIDUALS | POPULATION | METAANALYSIS | VARIANTS | MULTIDISCIPLINARY SCIENCES | RISK | FALSE DISCOVERY RATES | HYPERTENSION | STAGE RENAL-DISEASE | GENOME-WIDE ASSOCIATION | Genotype | Genetic Predisposition to Disease | Genome-Wide Association Study | Humans | Gene Expression Regulation | Renal Insufficiency, Chronic - genetics | Genetik | Biological Sciences | Genetics | Naturvetenskap | Natural Sciences | Biologiska vetenskaper
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22. Full Text The genetics of blood pressure regulation and its target organs from association studies in 342,415 individuals
by Ehret, Georg B and Ferreira, Teresa and Chasman, Daniel I and Jackson, Anne U and Schmidt, Ellen M and Johnson, Toby and Thorleifsson, Gudmar and Luan, Jian'an and Donnelly, Louise A and Kanoni, Stavroula and Petersen, Ann-Kristin and Pihur, Vasyl and Strawbridge, Rona J and Shungin, Dmitry and Hughes, Maria F and Meirelles, Osorio and Kaakinen, Marika and Bouatia-Naji, Nabila and Kristiansson, Kati and Shah, Sonia and Kleber, Marcus E and Guo, Xiuqing and Lyytikäinen, Leo-Pekka and Fava, Cristiano and Eriksson, Niclas and Nolte, Ilja M and Magnusson, Patrik K and Salfati, Elias L and Rallidis, Loukianos S and Theusch, Elizabeth and Smith, Anew J. P and Folkersen, Lasse and Witkowska, Kate and Pers, Tune H and Joehanes, Roby and Kim, Stuart K and Lataniotis, Lazaros and Jansen, Rick and Johnson, Anew D and Warren, Helen and Kim, Young Jin and Zhao, Wei and Wu, Ying and Tayo, Bamidele O and Bochud, Murielle and Absher, Devin and Adair, Linda S and Amin, Najaf and Arking, Dan E and Axelsson, Tomas and Baldassarre, Damiano and Balkau, Beverley and Bandinelli, Stefania and Barnes, Michael R and Barroso, Inês and Bevan, Stephen and Bis, Joshua C and Bjornsdottir, Gyda and Boehnke, Michael and Boerwinkle, Eric and Bonnycastle, Lori L and Boomsma, Dorret I and Bornstein, Stefan R and Brown, Morris J and Burnier, Michel and Cabrera, Claudia P and Chambers, John C and Chang, I.-Shou and Cheng, Ching-Yu and Chines, Peter S and Chung, Ren-Hua and Collins, Francis S and Connell, John M and Döring, Angela and Dallongeville, Jean and Danesh, John and de Faire, Ulf and Delgado, Graciela and Dominiczak, Anna F and Doney, Alex S. F and nos, Fotios and Edkins, Sarah and Eicher, John D and Elosua, Roberto and Enroth, Stefan and Erdmann, Jeanette and Eriksson, Per and Esko, Tonu and Evangelou, Evangelos and Evans, Alun and Fall, Tove and Farrall, Martin and Felix, Janine F and Ferrières, Jean and Ferrucci, Luigi and Fornage, Myriam and Forrester, Terrence and Franceschini, Nora and Franco, Oscar H and Franco-Cereceda, Anders and ... and CHARGE-HF Consortium and Wellcome Trust Case Control Consor and CHARGE-EchoGen Consortium and Wellcome Trust Case Control Consortium and Medicinska fakulteten and Avdelningen för medicin and Umeå universitet and Medicin and Institutionen för odontologi and Institutionen för folkhälsa och klinisk medicin and Enheten för biobanksforskning and Näringsforskning
Nature genetics, ISSN 1061-4036, 2016, Volume 48, Issue 10, pp. 1171 - 1184
To dissect the genetic architecture of blood pressure and assess effects on target organ damage, we analyzed 128,272 SNPs from targeted and genome-wide arrays... 
COMMON VARIANTS | METAANALYSIS | SUSCEPTIBILITY | GENETICS & HEREDITY | LOCI | TRAITS | HYPERTENSION | GENOME-WIDE ASSOCIATION | Studies | Consortia | Hypotheses | Genealogy | Genetics | Blood pressure | Genomes | Meta-analysis | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
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23. Full Text Physical and neurobehavioral determinants of reproductive onset and success
by Day, Felix R and Helgason, Hannes and Chasman, Daniel I and Rose, Lynda M and Loh, Po-Ru and Scott, Robert A and Helgason, Agnar and Kong, Augustine and Masson, Gisli and Magnusson, Olafur Th and Gudbjartsson, Daniel and Thorsteinsdottir, Unnur and Buring, Julie E and Ridker, Paul M and Sulem, Patrick and Stefansson, Kari and Ong, Ken K and Perry, John R.B
Nature Genetics, ISSN 1061-4036, 06/2016, Volume 48, Issue 6, pp. 617 - 623
The ages of puberty, first sexual intercourse and first birth signify the onset of reproductive ability, behavior and success, respectively. In a genome-wide... 
EXECUTIVE FUNCTION | ESTROGENS | MENARCHE | BEHAVIOR | INTERCOURSE | GENETICS & HEREDITY | SCHIZOPHRENIA | HEALTH | PUBERTY | GENOME-WIDE ASSOCIATION | AGE | Puberty | RNA-Binding Proteins - genetics | Cell Adhesion Molecules - genetics | Genome-Wide Association Study | Sexual Behavior | Age Factors | Membrane Proteins - genetics | Humans | Male | Methionine Sulfoxide Reductases - genetics | Nerve Tissue Proteins - genetics | Young Adult | Estrogen Receptor alpha - genetics | Adolescent | Coitus | Adult | Female | Behavior | Quantitative trait loci | Genome-wide association studies | Human reproduction | Sex | Genetic research | Genetic aspects | Research | Identification and classification | Methods | Studies | Datasets | Mens health | Womens health | Genomes | Age | Success
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24. Full Text Meta-Analysis of Genome-Wide Association Studies in >80 000 Subjects Identifies Multiple Loci for C-Reactive Protein Levels
by Dehghan, A and Dupuis, J and Barbalic, M and Bis, J.C and Eiriksdottir, G and Lu, C and Pellikka, N and Wallaschofski, H and Kettunen, J and Henneman, P and Baumert, J and Strachan, D.P and Fuchsberger, C and Vitart, V and Wilson, J.F and Paré, G and Naitza, S and Rudock, M.E and Surakka, I and de Geus, E.J.C and Alizadeh, BZ and Guralnik, J and Shuldiner, A and Tanaka, T and Zee, R.Y.L and Schnabel, R.B and Nambi, V and Kavousi, M and Ripatti, S and Nauck, M and Smith, N.L and Smith, A.V and Sundvall, J and Scheet, P and Liu, Y and Ruokonen, A and Rose, L.M and Larson, M.G and Hoogeveen, R.C and Freimer, N.B and Teumer, A and Tracy, R.P and Launer, L.J and Buring, J.E and Yamamoto, J.F and Folsom, A.R and Sijbrands, E.J.G and Pankow, J and Elliott, P and Keaney, J.F and Sun, W and Sarin, A.-P and Fontes, J.D and Badola, S and Astor, B.C and Hofman, A and Pouta, A and Werdan, K and Greiser, K.H and Meyer zu Schwabedissen, H.E and Thiery, J and Jamshidi, Y and Nolte, I.M and Soranzo, N and Spector, T.D and Völzke, H and Aspelund, T and Young, L and Siscovick, D.S and Rotter, J.I and Schlessinger, D and Rudan, I and Hicks, A.A and Penninx, B.W.J.H and Gieger, C and Willemsen, G and Harris, T.B and Uitterlinden, A.G and Järvelin, M.J and Rice, K and Salomaa, V and Boerwinkle, E and Ferrucci, L and Snieder, H and Boomsma, D.I and Hayward, C and Pramstaller, P.P and van Duijn, C.M and Peltonen, L and Psaty, B.M and Gudnason, V and Ridker, P.M and Homuth, G and Koenig, W and Ballantyne, C.M and Witteman, J.C.M and Benjamin, E.J and Perola, M and Chasman, D.I
Circulation, ISSN 0009-7322, 2011, Volume 123, Issue 7, pp. 731 - 738
Background-C-reactive protein (CRP) is a heritable marker of chronic inflammation that is strongly associated with cardiovascular disease. We sought to... 
meta-analysis | genetics | inflammation | genome-wide association study | myocardial infarction | EPIDEMIOLOGIC APPLICATIONS | FRAMINGHAM | COMMON VARIANTS | CRP GENE | DESIGN | CARDIAC & CARDIOVASCULAR SYSTEMS | GENETICALLY ISOLATED POPULATION | NETHERLANDS TWIN REGISTER | PERIPHERAL VASCULAR DISEASE | CORONARY-HEART-DISEASE | HEMATOLOGY | ALPHA-GENE | epidemiology | coronary heart disease | C-reactive protein | genome-wide association
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25. Full Text Genomic analyses identify hundreds of variants associated with age at menarche and support a role for puberty timing in cancer risk
by Day, Felix R and Thompson, Deborah J and Helgason, Hannes and Chasman, Daniel I and Finucane, Hilary and Sulem, Patrick and Ruth, Katherine S and Whalen, Sean and Sarkar, Abhishek K and Albrecht, Eva and Altmaier, Elisabeth and Amini, Marzyeh and Barbieri, Caterina M and Boutin, Thibaud and Campbell, Archie and Demerath, Ellen and Giri, Ayush and He, Chunyan and Hottenga, Jouke J and Karlsson, Robert and Kolcic, Ivana and Loh, Po-Ru and Lunetta, Kathryn L and Mangino, Massimo and Marco, Brumat and McMahon, George and Medland, Sarah E and Nolte, Ilja M and Noordam, Raymond and Nutile, Teresa and Paternoster, Lavinia and Perjakova, Natalia and Porcu, Eleonora and Rose, Lynda M and Schraut, Katharina E and Segre, Ayellet V and Smith, Albert V and Stolk, Lisette and Teumer, Alexander and Anulis, Irene L and Bandinelli, Stefania and Beckmann, Matthias W and Benitez, Javier and Bergmann, Sven and Franke, Lude and Hartman, Catharina A and Oldehinkel, Albertine J and Wolffenbuttel, Bruce H. R and Alizadeh, Behrooz Z and Snieder, Harold and Endometrial Canc Accoc Consortium and InterAct Consortium and PRACTICAl Consortium and kConFab AOCS Investigators and LifeLines Cohort Study and Ovarian Canc Accoc Consortium and kConFab/AOCS Investigators and Ovarian Cancer Association Consortium and PRACTICAL consortium and Endometrial Cancer Association Consortium and The InterAct Consortium and The LifeLines Cohort Study
Nature Genetics, ISSN 1061-4036, 06/2017, Volume 49, Issue 6, pp. 834 - 841
The timing of puberty is a highly polygenic childhood trait that is epidemiologically associated with various adult diseases. Using 1000 Genomes... 
REGRESSION | METAANALYSIS | METABOLISM | GROWTH | BODY-MASS INDEX | LOCI | WIDE ASSOCIATION | MENDELIAN RANDOMIZATION | TRAITS | INHERITANCE | GENETICS & HEREDITY | Body Mass Index | Genomic Imprinting | Genetic Predisposition to Disease | Genome-Wide Association Study | Age Factors | Membrane Proteins - genetics | Menarche - genetics | Humans | Risk Factors | Intercellular Signaling Peptides and Proteins - genetics | Databases, Genetic | Male | Puberty - genetics | Neoplasms - genetics | Adolescent | Female | Ribonucleoproteins - genetics | Polymorphism, Single Nucleotide | Quantitative Trait Loci | Genetic variation | Menarche | Genetic aspects | Gene expression | Health aspects | Carcinogenesis | Risk factors | Enrichment | Transcription | Laboratories | Genomics | Genes | Risk | Genomes | Tissues | Preadipocyte factor 1 | Epidemiology | Variance | Complexity | Body mass index | Mens health | Randomization | Coding | Genetics | Trends | Children | Endometrium | Public health | Age | Medical research | Puberty | Internal medicine | Gynecology | Health risks | Time measurement | Obstetrics | Diseases | Medicine | Studies | Hospitals | Body mass | Womens health | Men | Breast | Links | Heritability | Polygenic inheritance | Prostate cancer | Prostate | Endocrinology | Cancer | InterAct Consortium | AOCS Investigators | PRACTICAL consortium | Endometrial Cancer Association Consortium | Ovarian Cancer Association Consortium | LifeLines Cohort Study | kConFab
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26. Full Text Gene x Physical Activity Interactions in Obesity: Combined Analysis of 111,421 Individuals of European Ancestry
by Ahmad, S and Rukh, G and Varga, TV and Ali, A and Kurbasic, A and Shungin, D and Ericson, U and Koivula, RW and Chu, AY and Rose, LM and Ganna, A and Qi, QB and Stancakova, A and Sandholt, CH and Elks, CE and Curhan, G and Jensen, MK and Tamimi, RM and Allin, KH and Jorgensen, T and Brage, S and Langenberg, C and Aadahl, M and Grarup, N and Linneberg, A and Pare, G and Magnusson, PKE and Pedersen, NL and Boehnke, M and Hamsten, A and Mohlke, KL and Pasquale, LT and Pedersen, O and Scott, RA and Ridker, PM and Ingelsson, E and Laakso, M and Hansen, T and Qi, L and Wareham, NJ and Chasman, DI and Hallmans, G and Hu, FB and Renstrom, F and Orho-Melander, M and Franks, PW and DIRECT Consortium and InterAct Consortium and Medicinska fakulteten and Umeå universitet and Medicin and Institutionen för folkhälsa och klinisk medicin and Institutionen för odontologi and Enheten för biobanksforskning and Näringsforskning
PLOS GENETICS, ISSN 1553-7404, 07/2013, Volume 9, Issue 7, p. e1003607
Numerous obesity loci have been identified using genome-wide association studies. A UK study indicated that physical activity may attenuate the cumulative... 
FTO GENE | VARIANTS | GENETICS & HEREDITY | ADULTS | BODY-MASS INDEX | RISK | ENERGY-INTAKE | LOCI | TRAITS | WEIGHT | GENOME-WIDE ASSOCIATION | Obesity | Europeans | Physiological aspects | Genetic aspects | Research | Gene expression | Population genetics | Health aspects | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Hälsovetenskaper | Health Sciences
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27. Full Text FTO genotype is associated with phenotypic variability of body mass index
by Yang, J and Loos, R.J and Powell, J.E and Medland, S.E and Speliotes, E.K and Chasman, D.I and Rose, L.M and Thorleifsson, G and Steinthorsdottir, V and Magi, R and Waite, L and Smith, A.V and Yerges-Armstrong, L.M and Monda, K.L and Hadley, D and Mahajan, A and Li, G and Kapur, K and Vitart, V and Huffman, J.E and Wang, S.R and Palmer, C and Esko, T and Fischer, K and Zhao, J.H and Demirkan, A and Isaacs, A and Feitosa, M.F and Luan, J and Heard-Costa, N.L and White, C and Jackson, A.U and Preuss, M and Ziegler, A and Eriksson, J and Kutalik, Z and Frau, F and Nolte, I.M and Van Vliet-Ostaptchouk, J.V and Hottenga, J.J and Jacobs, K.B and Verweij, N and Goel, A and Medina-Gomez, C and Estrada, K and Bragg-Gresham, J.L and Sanna, S and Sidore, C and Tyrer, J and Teumer, A and Prokopenko, I and Mangino, M and Lindgren, C.M and Assimes, T.L and Shuldiner, A.R and Hui, J and Beilby, J.P and McArdle, W.L and Hall, P and Haritunians, T and Zgaga, L and Kolcic, I and Polasek, O and Zemunik, T and Oostra, B.A and Junttila, M.J and Gronberg, H and Schreiber, S and Peters, A and Hicks, A.A and Stephens, J and Foad, N.S and Laitinen, J and Pouta, A and Kaakinen, M and Willemsen, G and Vink, J and Wild, S.H and Navis, G and Asselbergs, F.W and Homuth, G and John, U and Iribarren, C and Harris, T and Launer, L and Gudnason, V and O'Connell, J.R and Boerwinkle, E and Cadby, G and Palmer, L.J and James, A.L and Musk, A.W and Ingelsson, E and Psaty, B.M and Beckmann, J.S and Waeber, G and Vollenweider, P and Hayward, C and Wright, A.F and Rudan, I and ...
Nature, ISSN 0028-0836, 2012, Volume 490, Issue 7419, pp. 267 - 272
There is evidence across several species for genetic control of phenotypic variation of complex traits, such that the variance among phenotypes is genotype... 
GENETIC-VARIATION | VARIANTS | MULTIDISCIPLINARY SCIENCES | PHYSICAL-ACTIVITY | LOCI | SELECTION | TRAITS | ENVIRONMENTAL SENSITIVITY | ADULT OBESITY | CHILDHOOD | GENOME-WIDE ASSOCIATION | Body Mass Index | Genome-Wide Association Study | Humans | Repressor Proteins - genetics | Alpha-Ketoglutarate-Dependent Dioxygenase FTO | Male | Nerve Tissue Proteins - genetics | Body Height - genetics | Genetic Variation | Proteins - genetics | Phenotype | Female | Polymorphism, Single Nucleotide | Co-Repressor Proteins | Body mass index | Physiological aspects | Phenotypic plasticity | Obesity gene | Genetic aspects | Single nucleotide polymorphisms | Methods | Studies | Genotype & phenotype | Genetics | Gene loci | Medical and Health Sciences | Medicin och hälsovetenskap
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28. Full Text Meta-analysis of 65,734 Individuals Identifies TSPAN15 and SLC44A2 as Two Susceptibility Loci for Venous Thromboembolism
by Germain, Marine and Chasman, Daniel I and de Haan, Hugoline and Tang, Weihong and Lindström, Sara and Weng, Lu-Chen and de Andrade, Mariza and de Visser, Marieke C.H and Wiggins, Kerri L and Suchon, Pierre and Saut, Noémie and Smadja, David M and Le Gal, Grégoire and van Hylckama Vlieg, Astrid and Di Narzo, Antonio and Hao, Ke and Nelson, Christopher P and Rocanin-Arjo, Ares and Folkersen, Lasse and Monajemi, Ramin and Rose, Lynda M and Brody, Jennifer A and Slagboom, Eline and Aïssi, Dylan and Gagnon, France and Deleuze, Jean-Francois and Deloukas, Panos and Tzourio, Christophe and Dartigues, Jean-Francois and Berr, Claudine and Taylor, Kent D and Civelek, Mete and Eriksson, Per and Psaty, Bruce M and Houwing-Duitermaat, Jeanine and Goodall, Alison H and Cambien, François and Kraft, Peter and Amouyel, Philippe and Samani, Nilesh J and Basu, Saonli and Ridker, Paul M and Rosendaal, Frits R and Kabrhel, Christopher and Folsom, Aaron R and Heit, John and Reitsma, Pieter H and Trégouët, David-Alexandre and Smith, Nicholas L and Morange, Pierre-Emmanuel and Cardiogenics Consortium
The American Journal of Human Genetics, ISSN 0002-9297, 04/2015, Volume 96, Issue 4, pp. 532 - 542
Venous thromboembolism (VTE), the third leading cause of cardiovascular mortality, is a complex thrombotic disorder with environmental and genetic... 
BREAST-CANCER | PLASMA-LEVELS | ENDOTHELIAL-CELLS | GENETICS & HEREDITY | GENE-EXPRESSION | CARDIOVASCULAR-DISEASE | GENOTYPING ARRAYS | AGING RESEARCH | RISK VARIANTS | PROTEIN-C | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Venous Thromboembolism - genetics | Membrane Transport Proteins - genetics | Humans | Genotype | Odds Ratio | Tetraspanins - genetics | Membrane Glycoproteins - genetics | Quantitative trait loci | Genetic aspects | Disease susceptibility | Thromboembolism | Identification and classification | Life Sciences | Human health and pathology | Cardiology and cardiovascular system
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29. Full Text Loci associated with ischaemic stroke and its subtypes (SiGN): a genome-wide association study
by Pulit, Sara L and McArdle, Patrick F and Wong, Quenna and Malik, Rainer and Gwinn, Katrina and Achterberg, Sefanja and Algra, Ale and Amouyel, Philippe and Anderson, Christopher D and Arnett, Donna K Arnett and Arnett, Donna K and Arsava, Ethem Murat and Attia, John and Ay, Hakan and Bartz, Traci M and Battey, Thomas and Benavente, Oscar and Benavente, Oscar R and Bevan, Steve and Biffi, Alessandro and Bis, Joshua C and Blanton, Susan H and Boncoraglio, Giorgio B and Brown, Robert D and Burgess, Annette I and Carrera, Caty and Chapman Smith, Sherita N and Chasman, Daniel I and Chauhan, Ganesh and Chen, Wei-Min and Cheng, Yu-Ching and Chong, Michael and Cloonan, Lisa K and Cole, John W and Cotlarciuc, Ioana and Cruchaga, Carlos and Cuadrado-Godia, Elisa and Dave, Tushar and Dawson, Jesse and Debette, Stéphanie and Delavaran, Hossein and Dell, Cameron A and Dichgans, Martin and Doheny, Kimberly F and Dong, Chuanhui and Duggan, David J and Engström, Gunnar and Evans, Michele K and Pallejà, Xavier Estivill and Faul, Jessica D and Fernández-Cadenas, Israel and Fornage, Myriam and Frossard, Philippe M and Furie, Karen and Gamble, Dale M and Gieger, Christian and Giese, Anne-Katrin and Giralt-Steinhauer, Eva and González, Hector M and Goris, An and Gretarsdottir, Solveig and Grewal, Raji P and Grittner, Ulrike and Gustafsson, Stefan and Han, Buhm and Hankey, Graeme J and Heitsch, Laura and Higgins, Peter and Hochberg, Marc C and Holliday, Elizabeth and Hopewell, Jemma C and Horenstein, Richard B and Howard, George and Ikram, M Arfan and Ilinca, Andreea and Ingelsson, Erik and Irvin, Marguerite R and Jackson, Rebecca D and Jern, Christina and Conde, Jordi Jiménez and Johnson, Julie A and Jood, Katarina and Kahn, Muhammad S and Kaplan, Robert and Kappelle, L Jaap and Kardia, Sharon L R and Keene, Keith L and Kissela, Brett M and Kleindorfer, Dawn O and Koblar, Simon and Labovitz, Daniel and Launer, Lenore J and Laurie, Cathy C and Laurie, Cecelia A and Lee, Jin-Moo and Lee, Cue Hyunkyu and Lehm, Manuel and Lemmens, Robin and Levi, Christopher and Leys, Didier and ... and NINDS Stroke Genetics Network SiGN and ISGC and International Stroke Genetics Consortium (ISGC) and NINDS Stroke Genetics Network (SiGN) and Institutionen för neurovetenskap och fysiologi, sektionen för klinisk neurovetenskap och rehabilitering and Institute of Neuroscience and Physiology and Institutionen för biomedicin, avdelningen för medicinsk genetik och klinisk genetik and Göteborgs universitet and Gothenburg University and Institute of Biomedicine, Department of Pathology and Institute of Biomedicine, Department of Medical and Clinical Genetics and Sahlgrenska Academy and Institute of Neuroscience and Physiology, Department of Clinical Neuroscience and Rehabilitation and Institutionen för biomedicin, avdelningen för patologi and Sahlgrenska akademin and Institutionen för neurovetenskap och fysiologi
Lancet Neurology, The, ISSN 1474-4422, 2016, Volume 15, Issue 2, pp. 174 - 184
Summary Background The discovery of disease-associated loci through genome-wide association studies (GWAS) is the leading genetic approach to the... 
Neurology | Journal Article | Clinical Neurology | COMMON VARIANTS | IMPUTATION | METAANALYSIS | REPLICATION | TOAST | CAUSATIVE CLASSIFICATION | RISK | CLINICAL NEUROLOGY | ATRIAL-FIBRILLATION | Brain Ischemia - classification | Humans | Middle Aged | Brain Ischemia - genetics | Genetic Loci | Nerve Tissue Proteins - genetics | Case-Control Studies | Stroke - genetics | Young Adult | Stroke - classification | Adolescent | Aged, 80 and over | Adult | Aged | Genome-Wide Association Study - methods | Tetraspanins - genetics | Cohort Studies | Ischemia | Genetic research | Genomics | Disease susceptibility | Studies | Stroke | Genealogy | Medical imaging | Classification | Atherosclerosis | Genomes | Methods | Veins & arteries | Medicinska grundvetenskaper | Basic Medicine
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