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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2016, Volume 113, Issue 21, pp. 5999 - 6004
Cancer cells develop under immune surveillance, thus necessitating immune escape for successful growth. Loss of MHC class I expression provides a key immune... 
NLRC5 | Immune evasion | Mhc class I | CITA | Cancer | CELLS | HLA CLASS-I | MULTIDISCIPLINARY SCIENCES | MHC class I | NUCLEOTIDE-BINDING DOMAIN | TUMORS | MHC CLASS-I | BREAST-CANCER | RESPONSES | CARCINOGENESIS | immune evasion | GENE-EXPRESSION | cancer | ANTIGEN | ATP-Binding Cassette Sub-Family B Member 2 - immunology | Tumor Escape | CD8-Positive T-Lymphocytes - pathology | Humans | Neoplasm Proteins - immunology | Intracellular Signaling Peptides and Proteins - immunology | Male | Transcriptional Activation - immunology | beta 2-Microglobulin - immunology | Antigen Presentation | Neoplasms - genetics | Trans-Activators - genetics | Female | Neoplasm Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Proteasome Endopeptidase Complex - immunology | Trans-Activators - immunology | Histocompatibility Antigens Class I - immunology | Neoplasms - mortality | Gene Expression Regulation, Neoplastic - immunology | ATP-Binding Cassette Sub-Family B Member 2 - genetics | Histocompatibility Antigens Class I - genetics | Proteasome Endopeptidase Complex - genetics | Neoplasms - immunology | Cysteine Endopeptidases - genetics | Biomarkers, Tumor - genetics | CD8-Positive T-Lymphocytes - immunology | Neoplasms - pathology | Biomarkers, Tumor - immunology | Cysteine Endopeptidases - immunology | beta 2-Microglobulin - genetics | Biological Sciences
Journal Article
Frontiers in immunology, ISSN 1664-3224, 2018, Volume 9, p. 1591
Hypoxia is not only a prominent contributor to the heterogeneity of solid tumors but also a crucial stressor in the microenvironment to drive adaptations for... 
Hypoxia | Immunosuppression | Microenvironment | Metabolism | Immunotherapy | LACTIC-ACID | hypoxia | CARBONIC-ANHYDRASE-IX | HLA-G EXPRESSION | IMMUNE ESCAPE | SURFACE EXPRESSION | immunosuppression | IMMUNOLOGY | INTEGRIN-ASSOCIATED PROTEIN | microenvironment | INDUCIBLE FACTORS | immunotherapy | REGULATORY T-CELLS | metabolism | SUPPRESSOR-CELLS | EXTRACELLULAR PH
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 03/2018, Volume 9, p. 578
...Author(s): Yakir Guri, Thierry M. Nordmann, Jason Roszik Cancer is a complex disease and a leading cause of death worldwide. Immunity is critical for cancer... 
Signaling | Rapamycin | Tumorigenesis | Metabolism | Immunity | Immunotherapy | ACTIVATION | signaling | KINASE MTOR | AKT | immunity | OVARIAN-CANCER | IMMUNOLOGY | BREAST-CANCER CELLS | EPITHELIAL-CELLS | immunotherapy | metabolism | tumorigenesis | SUPPRESSOR-CELLS | COMPLEX 2 | MACROPHAGE POLARIZATION | T-CELLS | rapamycin | Cellular signal transduction | Immune response | Research | Cancer cells
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 09/2018, Volume 9, p. 2029
...Author(s): Jason Roszik, Keiji Tanese, Suhendan Ekmekcioglu Editorial: Targeting Metabolism in Cancer Immunotherapy To access, purchase, authenticate... 
Tumor microenvironment | Immune cells | Metabolism | Immunotherapy | Cancer | CELLS | immunotherapy | immune cells | cancer | metabolism | tumor microenvironment | IMMUNOLOGY | DORMANCY | Usage | Care and treatment | Research | Immune system
Journal Article
BMC Genomics, ISSN 1471-2164, 12/2014, Volume 15, Issue 1, pp. 1044 - 1044
...Author(s): Jason Roszik[sup.1,2] and Scott E Woodman[sup.1,2] Background Driver mutations provide a growth advantage for tumor cells and have been positively... 
Driver mutation | Visualization | Hotspots | Cancer | MELANOMA | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | CARCINOMA | Computational Biology - methods | Software | Mutation | Gene mutations | Amino acids
Journal Article
Journal Article
Frontiers in oncology, ISSN 2234-943X, 2018, Volume 8, p. 67
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 29, pp. 10660 - 10665
Clinical-grade T cells are genetically modified ex vivo to express chimeric antigen receptors (CARs) to redirect their specificity to target tumor-associated... 
T lymphocytes | Aspergillus | Receptors | Cell walls | Hyphae | Fungal infections | Census agricultural regions | Immunity | Blood | Conidia | Fungus | β-1,3-glucan | Adoptive immunotherapy | T-cell therapy | beta-1,3-glucan | DEFENSE | ASPERGILLUS-FUMIGATUS | RECOGNITION | MULTIDISCIPLINARY SCIENCES | TRANSPLANT RECIPIENTS | DECTIN-1 | BETA-GLUCAN RECEPTOR | EFFECTOR | INVASIVE ASPERGILLOSIS | fungus | PULMONARY ASPERGILLOSIS | CHIMERIC-ANTIGEN-RECEPTOR | adoptive immunotherapy | Aspergillosis - therapy | Receptors, Antigen, T-Cell - metabolism | Humans | Immunophenotyping | Opportunistic Infections - therapy | Aspergillus - drug effects | Bioengineering - methods | Lectins, C-Type - metabolism | Animals | Aspergillosis - pathology | Dexamethasone - pharmacology | Carbohydrates - antagonists & inhibitors | Lymphocyte Activation - drug effects | T-Lymphocytes - drug effects | Aspergillosis - microbiology | Hyphae - drug effects | T-Lymphocytes - immunology | Antigens, CD19 - metabolism | Aspergillus - physiology | Mice | Opportunistic Infections - immunology | Opportunistic Infections - pathology | Aspergillosis - immunology | Hyphae - physiology | Bioengineering | Carbohydrates | Usage | Care and treatment | Immunotherapy | Mycoses | Physiological aspects | T cells | Health aspects | Methods | Immunological research | Biological Sciences
Journal Article
Cell Cycle, ISSN 1538-4101, 05/2017, Volume 16, Issue 9, pp. 813 - 814
Journal Article
by Farshidfar, Farshad and Zheng, Siyuan and Gingras, Marie-Claude and Newton, Yulia and Shih, Juliann and Robertson, A. Gordon and Hinoue, Toshinori and Hoadley, Katherine A and Gibb, Ewan A and Roszik, Jason and Covington, Kyle R and Wu, Chia-Chin and Shinbrot, Eve and Stransky, Nicolas and Hegde, Apurva and Yang, Ju Dong and Reznik, Ed and Sadeghi, Sara and Pedamallu, Chandra Sekhar and Ojesina, Akinyemi I and Hess, Julian M and Auman, J. Todd and Rhie, Suhn K and Bowlby, Reanne and Borad, Mitesh J and Zhu, Andrew X and Stuart, Josh M and Sander, Chris and Akbani, Rehan and Cherniack, Andrew D and Deshpande, Vikram and Mounajjed, Taofic and Foo, Wai Chin and Torbenson, Michael S and Kleiner, David E and Laird, Peter W and Wheeler, David A and McRee, Autumn J and Bathe, Oliver F and Andersen, Jesper B and Bardeesy, Nabeel and Roberts, Lewis R and Kwong, Lawrence N and Allotey, Loretta K and Ally, Adrian and Alvaro, Domenico and Appelbaum, Elizabeth L and Arora, Arshi and Balasundaram, Miruna and Balu, Saianand and Baylin, Stephen B and Beroukhim, Rameen and Berrios, Mario and Bodenheimer, Tom and Boice, Lori and Bootwalla, Moiz S and Bowen, Jay and Bragazzi, Maria Consiglia and Brooks, Denise and Cardinale, Vincenzo and Carlsen, Rebecca and Carpino, Guido and Carvalho, Andre L and Chaiteerakij, Roongruedee and Chandan, Vishal C and Chin, Lynda and Cho, Juok and Choe, Gina and Chuah, Eric and Chudamani, Sudha and Cibulskis, Carrie and Cordes, Matthew G and Crain, Daniel and Curley, Erin and De Rose, Agostino Maria and Defreitas, Timothy and Demchok, John A and Dhalla, Noreen and Ding, Li and Evason, Kimberley and Felau, Ina and Ferguson, Martin L and Franchitto, Antonio and Frazer, Scott and Fronick, Catrina C and Fulton, Lucinda A and Fulton, Robert S and Gabriel, Stacey B and Gardner, Johanna and Gastier-Foster, Julie M and Gaudio, Eugenio and Gehlenborg, Nils and Genovese, Giannicola and Gerken, Mark and Getz, Gad and Giama, Nasra H and Gibbs, Richard A and Giuliante, Felice and Grazi, Gian Luca and Hayes, D. Neil and ... and The Cancer Genome Atlas Network and Canc Genome Atlas Network and Cancer Genome Atlas Network
Cell reports (Cambridge), ISSN 2211-1247, 2017, Volume 18, Issue 11, pp. 2780 - 2794
Journal Article