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PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, p. e9367
Background: Mitochondrial dysfunction and degradation takes a central role in current paradigms of neurodegeneration in Parkinson's disease (PD). Loss of DJ-1... 
PINK1 | FUSION | GENE | MULTIDISCIPLINARY SCIENCES | ENERGETIC DEPRESSION | CYSTEINE-SULFINIC ACID | MUSCLE | DEGRADATION | MUTATIONS | PATHOLOGY | FISSION | Oncogene Proteins - genetics | Phosphorylation | Reactive Oxygen Species - metabolism | Humans | Intracellular Signaling Peptides and Proteins - metabolism | Autophagy | Fibroblasts - ultrastructure | Lysosomes - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Fibroblasts - metabolism | Parkinson Disease - pathology | Oncogene Proteins - metabolism | Oxidative Phosphorylation | Mitochondria - metabolism | Parkinson Disease - genetics | Microscopy, Electron | Blotting, Western | Mice, Knockout | Peroxiredoxins | Lysosomes - ultrastructure | Protein Deglycase DJ-1 | Animals | Fibroblasts - cytology | Mice | Mutation | Mitogen-Activated Protein Kinase 1 - metabolism | Genes | Genetic aspects | Oxidative stress | Cell culture | Reactive oxygen species | Animal models | Parkinson's disease | Laboratories | Downstream effects | Parkinsons disease | Homeostasis | Lysosomes | Kinases | Accumulation | Degradation | Proteins | Mitochondria | Neurodegeneration | Penicillin | Fibroblasts | Aging | Physiology | Membrane potential | Movement disorders | Neurodegenerative diseases | Extracellular signal-regulated kinase | Organelles | PARK7 protein | Neurology | Brain research | Hypoxia | Electron transport | Respiration | Alzheimers disease | Phagocytosis | Apoptosis | Integrity
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