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Free Radical Biology and Medicine, ISSN 0891-5849, 09/2017, Volume 110, p. 390
Protein kinase G (PKG) I[alpha] is the end-effector kinase that mediates nitric oxide (NO)-dependent and oxidant-dependent vasorelaxation to maintain blood... 
Obesity | Hydrogen peroxide | Nitric oxide | Oxidation-reduction reaction | Superoxide | Cyclic guanylic acid | Protein kinases | Hypertension | Heart | Leptin | Genetic engineering | Peroxides
Journal Article
Journal Article
JAMA - Journal of the American Medical Association, ISSN 0098-7484, 01/2019, Volume 321, Issue 1, pp. 69 - 79
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 09/2017, Volume 110, pp. 390 - 398
Protein kinase G (PKG) Iα is the end-effector kinase that mediates nitric oxide (NO)-dependent and oxidant-dependent vasorelaxation to maintain blood pressure... 
NOS uncoupling | PKG Iα | Oxidation | Diet-induced obesity | Disulfide | ACTIVATION | PROTEIN | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | RELAXATION | RISK | REDOX SENSOR | BLOOD-PRESSURE | ENDOCRINOLOGY & METABOLISM | TETRAHYDROBIOPTERIN | REDUCTASE | PKG I alpha | Methotrexate - pharmacology | Reactive Oxygen Species - metabolism | Resistin - genetics | Sugar Acids - pharmacology | Uncoupling Agents - pharmacology | Oxidative Stress | Leptin - metabolism | Glucagon - genetics | Myocytes, Smooth Muscle - pathology | Diet, High-Fat - adverse effects | Cyclic GMP-Dependent Protein Kinase Type I - genetics | Aorta - metabolism | Obesity - genetics | Hypertension - etiology | Cyclic GMP-Dependent Protein Kinase Type I - metabolism | Cattle | Obesity - etiology | Hypertension - genetics | Nitric Oxide Synthase Type III - metabolism | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Resistin - metabolism | Oxidation-Reduction | Signal Transduction | Endothelial Cells - metabolism | Aorta - drug effects | Gene Expression Regulation | Leptin - genetics | Carmustine - pharmacology | Nitric Oxide Synthase Type III - genetics | Hypertension - pathology | Hypertension - metabolism | Obesity - metabolism | Obesity - pathology | Aorta - pathology | Animals | Glucagon - metabolism | Mice | Endothelial Cells - pathology | Endothelial Cells - drug effects | Original
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2014, Volume 111, Issue 22, pp. 8167 - 8172
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2013, Volume 110, Issue 24, pp. 9909 - 9913
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2013, Volume 65, p. S82
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2013, Volume 65, pp. S82 - S82
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 06/2019, Volume 116, Issue 26, pp. 13016 - 13025
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2012, Volume 53, pp. S156 - S156
Journal Article
Circulation, ISSN 0009-7322, 07/2012, Volume 126, Issue 3, pp. 287 - 295
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2011, Volume 51, pp. S49 - S49
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2012, Volume 53, pp. S156 - S156
Journal Article
Proceedings of the National Academy of Sciences of the United States, ISSN 0027-8424, 06/2019, Volume 116, Issue 26, p. 13016
Chronic hypoxia causes pulmonary hypertension (PH), vascular remodeling, right ventricular (RV) hypertrophy, and cardiac failure. Protein kinase G I[alpha]... 
Oxidases | Biological products | Heart enlargement | Oxidation-reduction reaction | Peroxides | Superoxide | Protein kinases | Pulmonary hypertension | Protein binding
Journal Article
Public and Municipal Finance, ISSN 2222-1867, 08/2018, Volume 7, Issue 1, pp. 41 - 48
Journal Article