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Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 03/2019, Volume 199, Issue 6, pp. 799 - 802
[...]mean PApwa was increased in all PAH subtypes, and this observation could not be explained by differences in hematologic parameters or aortic radiodensity... 
RESPIRATORY SYSTEM | CRITICAL CARE MEDICINE | Hypertension | Medical imaging | Transplants & implants | Pulmonary arteries | Mortality | Coronary vessels | Calcification | Scleroderma | Age
Journal Article
Circulation, ISSN 0009-7322, 11/2015, Volume 132, Issue Suppl_3 Suppl 3, pp. A14631 - A14631
BackgroundPulmonary arterial hypertension (PAH) is associated with imbalance between proliferation and apoptosis of pulmonary artery (PA) smooth muscle cells... 
Journal Article
Circulation, ISSN 0009-7322, 11/2015, Volume 132, Issue Suppl_3 Suppl 3, pp. A14331 - A14331
BackgroundPulmonary arterial smooth muscle cells (PASMCs) from pulmonary arterial hypertension (PAH) patients exhibit a pro-proliferative, pro-migratory and... 
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 04/2016, Volume 133, Issue 14, pp. 1371 - 1385
BACKGROUND—Mutations in the KCNK3 gene have been identified in some patients suffering from heritable pulmonary arterial hypertension (PAH). KCNK3 encodes an... 
hypertension, pulmonary | pulmonary artery | cell proliferation | electrophysiology | ion channels | SIGNALING PATHWAYS | CARDIAC & CARDIOVASCULAR SYSTEMS | TYROSINE KINASE | RATS | pulmonary | MONOCROTALINE | FUNCTIONAL-PROPERTIES | MATRIX METALLOPROTEINASES | TO-MESENCHYMAL TRANSITION | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | hypertension | EXPRESSION | ortho-Aminobenzoates - pharmacology | Rats, Wistar | Humans | Myocytes, Smooth Muscle - pathology | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Hypertension, Pulmonary - chemically induced | Cell Division | Nerve Tissue Proteins - biosynthesis | Nerve Tissue Proteins - antagonists & inhibitors | Genetic Predisposition to Disease | Nerve Tissue Proteins - physiology | Vascular Resistance | Bone Morphogenetic Protein Receptors, Type II - genetics | Potassium Channels, Tandem Pore Domain - genetics | Rats | Hypertension, Pulmonary - genetics | Inflammation | Monocrotaline - toxicity | Fibroblasts - pathology | Sulfonamides - pharmacology | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Patch-Clamp Techniques | Animals | Membrane Potentials | Potassium Channels, Tandem Pore Domain - physiology | Potassium Channels, Tandem Pore Domain - antagonists & inhibitors | Endothelium, Vascular - pathology | Adventitia - pathology | Potassium Channels, Tandem Pore Domain - biosynthesis | Hemodynamics | Mutation | Hypertension, Pulmonary - complications | Research | Gene mutations | Patients | Health aspects | Pulmonary hypertension
Journal Article
Circulation Research, ISSN 0009-7330, 08/2019, Volume 125, Issue Suppl_1
Journal Article
FRONTIERS IN PHYSIOLOGY, ISSN 1664-042X, 09/2019, Volume 10
Rationale: Pulmonary hypertension (PH) is a rare but fatal disease characterized by elevated pulmonary pressures and vascular remodeling, leading to right... 
ACTIVATION | PHYSIOLOGY | pulmonary hypertension | Monocrotaline | sympathetic nervous system | VEGF | DISEASE SEVERITY | PEPTIDE | RESPONSES | sympathetic activation | MESSENGER-RNA | neuroinflammation | MICROGLIA | spinal cord | DYSFUNCTION | EXPRESSION | oxidative stress | BRAIN
Journal Article