X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (88) 88
cardiac & cardiovascular systems (68) 68
index medicus (49) 49
male (46) 46
rats (40) 40
ischemia (38) 38
rats, sprague-dawley (34) 34
humans (33) 33
reperfusion injury (31) 31
mitochondria (27) 27
myocardial reperfusion injury - metabolism (26) 26
myocardial reperfusion injury - pathology (25) 25
myocardial reperfusion injury - prevention & control (25) 25
myocardium - metabolism (24) 24
physiology (24) 24
myocardium - pathology (23) 23
reperfusion (23) 23
myocytes, cardiac - metabolism (21) 21
cell death (19) 19
mice (19) 19
myocardial reperfusion injury - physiopathology (19) 19
necrosis (19) 19
peripheral vascular disease (19) 19
swine (19) 19
calcium (18) 18
cardioprotection (18) 18
in vitro techniques (18) 18
myocardial infarction - pathology (18) 18
calcium - metabolism (17) 17
hypercontracture (17) 17
ischemic preconditioning, myocardial (16) 16
mitochondrial permeability transition (16) 16
myocardial infarction (16) 16
time factors (16) 16
cardiology (15) 15
cell biology (15) 15
cells, cultured (15) 15
heart (15) 15
mitochondria, heart - metabolism (15) 15
physiological aspects (15) 15
injury (14) 14
mitochondrial dna (13) 13
myocardial reperfusion (13) 13
analysis (12) 12
cell-death (12) 12
female (12) 12
infarct size (12) 12
acute myocardial-infarction (11) 11
ischemia-reperfusion (11) 11
myocytes, cardiac - drug effects (11) 11
research (11) 11
biochemistry & molecular biology (10) 10
cardiovascular (10) 10
connexin 43 - metabolism (10) 10
hypoxia (10) 10
oxidative stress (10) 10
permeability (10) 10
rat-heart (10) 10
signal transduction (10) 10
activation (9) 9
coronary-artery occlusion (9) 9
disease models, animal (9) 9
inhibition (9) 9
l-lactate dehydrogenase - metabolism (9) 9
myocardial infarction - physiopathology (9) 9
myocytes, cardiac - pathology (9) 9
nitric-oxide (9) 9
rodents (9) 9
apoptosis (8) 8
cardiomyocytes (8) 8
hydrogen-ion concentration (8) 8
intercellular communication (8) 8
isolated rat-heart (8) 8
medicine & public health (8) 8
mitochondrial membrane transport proteins - metabolism (8) 8
myocardial contraction - drug effects (8) 8
myocardial infarction - metabolism (8) 8
myocardial infarction - prevention & control (8) 8
myocardial ischemia - metabolism (8) 8
percutaneous coronary intervention (8) 8
reduces infarct size (8) 8
adenosine triphosphate - metabolism (7) 7
calpain (7) 7
cells (7) 7
edema (7) 7
energy metabolism (7) 7
expression (7) 7
heart - drug effects (7) 7
infarction (7) 7
mice, inbred c57bl (7) 7
myocardial infarction - therapy (7) 7
myocardial-ischemia (7) 7
myocardium - cytology (7) 7
nitric oxide (7) 7
perfusion (7) 7
cardiac myocytes (6) 6
cardiomyocyte mitochondria (6) 6
cell death - drug effects (6) 6
gap junctions (6) 6
gap junctions - metabolism (6) 6
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 08/2012, Volume 16, Issue 8, pp. 1649 - 1655
Journal Article
Scientific reports, ISSN 2045-2322, 04/2019, Volume 9, Issue 1, pp. 6395 - 1
A correction to this article has been published and is linked from the HTML and PDF versions of this paper. The error has been fixed in the paper. 
Myocardium | Succinate dehydrogenase
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 9/2018, Volume 113, Issue 5, pp. 1 - 73
Journal Article
Journal Article
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 11/2017, Volume 112, pp. 131 - 131
Journal Article
Nature, ISSN 0028-0836, 10/2011, Volume 478, Issue 7367, pp. 114 - 118
Left ventricular mass (LVM) is a highly heritable trait(1) and an independent risk factor for all-cause mortality(2). So far, genome-wide association studies... 
PATHOGENESIS | OXIDATIVE STRESS | REPLICATION | BIOGENESIS | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | DISEASE | DEATH | MECHANISMS | LEFT-VENTRICULAR MASS | EXPRESSION | Receptors, Estrogen - metabolism | Hypertrophy, Left Ventricular - enzymology | Reactive Oxygen Species - metabolism | Chromosomes, Mammalian - genetics | Cardiomegaly - pathology | Male | Rats, Inbred Strains | Body Weight - genetics | Hypertrophy, Left Ventricular - genetics | Cell Respiration | Mitochondria - genetics | Hypertrophy, Left Ventricular - pathology | Organ Size - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Endodeoxyribonucleases - metabolism | Female | Endodeoxyribonucleases - deficiency | Gene Expression Regulation | Cardiomegaly - physiopathology | Rats | Lipid Metabolism | Mitochondria - metabolism | Cardiomegaly - enzymology | Mitochondria - pathology | Transcription Factors - metabolism | Genes, Mitochondrial - genetics | Endodeoxyribonucleases - genetics | Animals | Hypertrophy, Left Ventricular - physiopathology | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Apoptosis | Crosses, Genetic | Quantitative Trait Loci - genetics | Nucleases | Mitochondria | Heart enlargement | Physiological aspects | Genetic aspects | Research | Diagnosis | Risk factors | Studies | Rodents | Biomarkers | Genetics | Cardiomyocytes | Genomes | Blood pressure | Kinases
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2009, Volume 297, Issue 4, pp. 1281 - 1289
There is solid evidence that a sudden change in mitochondrial membrane permeability (mitochondrial permeability transition, MPT) plays a critical role in... 
Calcium | Mitochondria | Hypercontracture | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | calcium | mitochondria | CA2+ RELEASE | CARDIAC MYOCYTES | REOXYGENATION-INDUCED HYPERCONTRACTURE | MYOCARDIAL REPERFUSION | CELL-DEATH | INFARCT SIZE | CYCLOPHILIN-D | CYCLOSPORINE-A | hypercontracture | PERIPHERAL VASCULAR DISEASE | ENDOPLASMIC-RETICULUM | PROTECTS CARDIOMYOCYTES | Colchicine - pharmacology | Mitochondria, Heart - metabolism | Mitochondrial Membrane Transport Proteins - antagonists & inhibitors | Cyclosporine - pharmacology | Mitochondria, Heart - pathology | Sarcoplasmic Reticulum - drug effects | Tubulin Modulators - pharmacology | Fluoresceins - metabolism | Male | Mitochondria, Heart - drug effects | Microtubules - metabolism | Myocardial Reperfusion Injury - pathology | Time Factors | Cell Death | Membrane Potential, Mitochondrial | Sarcoplasmic Reticulum Calcium-Transporting ATPases - antagonists & inhibitors | Sarcoplasmic Reticulum - metabolism | Ryanodine - pharmacology | Ischemic Contracture - pathology | Mitochondrial Membrane Transport Proteins - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Calcium Signaling - drug effects | Myocytes, Cardiac - metabolism | Sarcoplasmic Reticulum - pathology | Thapsigargin - pharmacology | Ischemic Contracture - metabolism | Physiological aspects | Research | Cell death | Sarcoplasmic reticulum | Heart cells
Journal Article
Journal Article