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NO paradox in asthma, 12/2005
Asthma results from allergen-driven intrapulmonary Th2 response, and is characterized by intermittent airway obstruction, airway hyperreactivity (AHR), and... 
asthma - nitric oxide - lipopolysaccharides
Journal
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 09/2019, Volume 106, Issue 3, pp. 653 - 664
Blomia tropicalis mite is highly prevalent in tropical and subtropical regions and it is associated with allergic diseases such as rhinitis and asthma. By... 
lungs | Blomia tropicalis | inflammation | bronchoalveolar lavage | CpG‐ODN | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, pp. e67115 - e67115
Experimental evidence and epidemiological studies indicate that exposure to endotoxin lipopolysaccharide (eLPS) or other TLR agonists prevent asthma. We have... 
CELL-WALL COMPONENTS | EXPERIMENTAL-MODEL | MULTIDISCIPLINARY SCIENCES | INDUCED EOSINOPHIL RECRUITMENT | ASTHMA | DERMATOPHAGOIDES-PTERONYSSINUS | RESPIRATORY ALLERGY | LUNG INFLAMMATION | MOUSE MODELS | T-CELLS | ENDOTHELIAL GROWTH-FACTOR | Toll-Like Receptor 2 - genetics | Pulmonary Eosinophilia - chemically induced | Pulmonary Eosinophilia - genetics | Th2 Cells - immunology | Eosinophils - immunology | Signal Transduction - immunology | Allergens - immunology | Asthma - immunology | Eosinophils - pathology | Allergens - toxicity | Th2 Cells - pathology | Disease Models, Animal | Neutrophils - pathology | Pulmonary Eosinophilia - immunology | Th17 Cells - pathology | Lipopolysaccharides - toxicity | Neutrophils - immunology | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Pulmonary Eosinophilia - pathology | Asthma - chemically induced | Asthma - genetics | Mice, Knockout | Animals | Signal Transduction - drug effects | Th17 Cells - immunology | Toll-Like Receptor 2 - immunology | Mice | Asthma - pathology | Pyroglyphidae - immunology | Allergens | Inflammation | Mitogens | Epidemiology | Analysis | Asthma | Lung | Helper cells | Aluminum sulfate | Critical care | Lymphocytes T | CD14 antigen | Immunity | Lipopolysaccharides | Respiratory tract | Cell activation | Immunology | Rodents | Toll-like receptors | Bacteria | Respiratory tract diseases | Gram-negative bacteria | Antigens | Immune response | Leukocytes (eosinophilic) | House dust | Interleukin 12 | Leukocytes (neutrophilic) | Exposure | TLR4 protein | Eosinophilia | Allergies | Medicine | Studies | Signaling | Dust | TLR2 protein | γ-Interferon | Neutrophilia | MyD88 protein | Interferon | Alum | Eosinophils | Index Medicus
Journal Article
Journal Article
PLoS pathogens, ISSN 1553-7374, 11/2017, Volume 13, Issue 11, pp. e1006730 - e1006730
[This corrects the article DOI: 10.1371/journal.ppat.1006054.]. 
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2016, Volume 11, Issue 5, pp. e0155454 - e0155454
Journal Article
Respiratory Research, ISSN 1465-9921, 05/2010, Volume 11, Issue 1, pp. 51 - 51
Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 03/2019, Volume 143, Issue 3, p. 1119
The transcriptional repressor B lymphocyte--induced maturation protein 1 (Blimp-1) has a key role in terminal differentiation in various T-cell subtypes.... 
Analysis | Inflammation | T cells | Transforming growth factors | Asthma
Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 03/2019, Volume 143, Issue 3, pp. 1119 - 1130.e3
The transcriptional repressor B lymphocyte–induced maturation protein 1 (Blimp-1) has a key role in terminal differentiation in various T-cell subtypes.... 
Allergy | IL-9 | TH9 differentiation | Blimp-1
Journal Article
Critical Care Medicine, ISSN 0090-3493, 02/2006, Volume 34, Issue 2, pp. 461 - 470
OBJECTIVE:We have documented an impaired neutrophil migration toward the infectious focus in severe sepsis. This phenomenon appears to be mediated by nitric... 
Lipopolysaccharide | Neutrophil migration | Sepsis | Nitric oxide | Toll-like receptor 4 | neutrophil migration | nitric oxide | POLYMORPHONUCLEAR LEUKOCYTES | GENETIC-CONTROL | ACUTE LUNG INJURY | FACTOR-ALPHA | TUMOR-NECROSIS-FACTOR | TOLL-LIKE RECEPTOR-4 | CECAL LIGATION | NITRIC-OXIDE | SEPTIC SHOCK